日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Machine Learning Achieves Pathologist-Level Coeliac Disease Diagnosis

机器学习实现病理学家级别的乳糜泻诊断

Jaeckle, F; Denholm, J; Schreiber, B; Evans, S C; Wicks, M N; Chan, J Y H; Bateman, A C; Natu, S; Arends, M J; Soilleux, E

Stain normalization gives greater generalizability than stain jittering in neural network training for the classification of coeliac disease in duodenal biopsy whole slide images

在对十二指肠活检全切片图像进行乳糜泻分类的神经网络训练中,染色归一化比染色抖动具有更高的泛化能力。

Schreiber, B A; Denholm, J; Gilbey, J D; Schönlieb, C-B; Soilleux, E J

Multiple-instance-learning-based detection of coeliac disease in histological whole-slide images

基于多示例学习的组织学全切片图像乳糜泻检测

Denholm, J; Schreiber, B A; Evans, S C; Crook, O M; Sharma, A; Watson, J L; Bancroft, H; Langman, G; Gilbey, J D; Schönlieb, C-B; Arends, M J; Soilleux, E J

Correction: S1PR1 drives a feedforward signalling loop to regulate BATF3 and the transcriptional programme of Hodgkin lymphoma cells

更正:S1PR1驱动前馈信号环路,从而调控BATF3和霍奇金淋巴瘤细胞的转录程序。

Vrzalikova, K; Ibrahim, M; Vockerodt, M; Perry, T; Margielewska, S; Lupino, L; Nagy, E; Soilleux, E; Liebelt, D; Hollows, R; Last, A; Reynolds, G; Abdullah, M; Curley, H; Care, M; Krappmann, D; Tooze, R; Allegood, J; Spiegel, S; Wei, W; Woodman, C B J; Murray, P G

S1PR1 drives a feedforward signalling loop to regulate BATF3 and the transcriptional programme of Hodgkin lymphoma cells

S1PR1驱动前馈信号环路,从而调控BATF3和霍奇金淋巴瘤细胞的转录程序。

Vrzalikova, K; Ibrahim, M; Vockerodt, M; Perry, T; Margielewska, S; Lupino, L; Nagy, E; Soilleux, E; Liebelt, D; Hollows, R; Last, A; Reynolds, G; Abdullah, M; Curley, H; Care, M; Krappmann, D; Tooze, R; Allegood, J; Spiegel, S; Wei, W; Woodman, C B J; Murray, P G

Dendritic and mast cell involvement in the inflammatory response to primary malignant bone tumours

树突状细胞和肥大细胞参与原发性恶性骨肿瘤的炎症反应

Inagaki, Y; Hookway, E; Williams, K A; Hassan, A B; Oppermann, U; Tanaka, Y; Soilleux, E; Athanasou, N A

DC-SIGNR, a DC-SIGN homologue expressed in endothelial cells, binds to human and simian immunodeficiency viruses and activates infection in trans.

DC-SIGNR 是内皮细胞中表达的 DC-SIGN 同源物,它能与人类和猴免疫缺陷病毒结合,并激活反式感染

Pöhlmann S, Soilleux E J, Baribaud F, Leslie G J, Morris L S, Trowsdale J, Lee B, Coleman N, Doms R W

cis Expression of DC-SIGN allows for more efficient entry of human and simian immunodeficiency viruses via CD4 and a coreceptor.

DC-SIGN 的顺式表达使得人类和猴免疫缺陷病毒能够通过 CD4 和辅助受体更有效地进入细胞

Lee B, Leslie G, Soilleux E, O'Doherty U, Baik S, Levroney E, Flummerfelt K, Swiggard W, Coleman N, Malim M, Doms R W