Solaimuthu相关文献与解析，生知库 | 链接生命科学的每一步

Solaimuthu, Balakrishnan; Khatib, Anees; Tanna, Mayur; Karmi, Abdelrahman; Hayashi, Arata; Abu Rmaileh, Areej; Lichtenstein, Michal; Takoe, Suranjana; Jolly, Mohit Kumar; Shaul, Yoav David

肿瘤

肿瘤免疫

JAK/STAT

DPYSL2 interacts with JAK1 to mediate breast cancer cell migration

DPYSL2 与 JAK1 相互作用介导乳腺癌细胞迁移

期刊:Journal of Cell Biology

影响因子:7.4

doi:10.1083/jcb.202106078

Areej Abu Rmaileh, Balakrishnan Solaimuthu, Anees Khatib, Shirel Lavi, Mayur Tanna, Arata Hayashi, Michal Ben Yosef, Michal Lichtenstein, Nir Pillar, Yoav D Shaul

Overexpression of UBA5 in Cells Mimics the Phenotype of Cells Lacking UBA5

UBA5 在细胞中的过表达模仿了缺乏 UBA5 的细胞的表型

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms23137445

Sujata Kumari, Sayanika Banerjee, Manoj Kumar, Arata Hayashi, Balakrishnan Solaimuthu, Einav Cohen-Kfir, Yoav D Shaul, Alexander Rouvinski, Reuven Wiener

Depletion of Fumarate Hydratase, an Essential TCA Cycle Enzyme, Drives Proliferation in a Two-Step Model

延胡索酸水合酶（TCA 循环必需酶）的消耗可促进两步模型中的增殖

期刊:Cancers

影响因子:

doi:10.3390/cancers14225508

Balakrishnan Solaimuthu, Michal Lichtenstein, Arata Hayashi, Anees Khatib, Inbar Plaschkes, Yuval Nevo, Mayur Tanna, Ophry Pines, Yoav D Shaul

Large-Scale Differential Gene Expression Transcriptomic Analysis Identifies a Metabolic Signature Shared by All Cancer Cells

大规模差异基因表达转录组分析揭示所有癌细胞共有的代谢特征

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom10050701

Rmaileh, Areej Abu; Solaimuthu, Balakrishnan; Tanna, Mayur; Khatib, Anees; Yosef, Michal Ben; Hayashi, Arata; Lichtenstein, Michal; Shaul, Yoav D

The glutathione peroxidase 8 (GPX8)/IL-6/STAT3 axis is essential in maintaining an aggressive breast cancer phenotype

谷胱甘肽过氧化物酶 8 (GPX8)/IL-6/STAT3 轴对于维持侵袭性乳腺癌表型至关重要

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.2010275117

Anees Khatib, Balakrishnan Solaimuthu, Michal Ben Yosef, Areej Abu Rmaileh, Mayur Tanna, Gidi Oren, Michal Schlesinger Frisch, Jonathan H Axelrod, Michal Lichtenstein, Yoav D Shaul

The exostosin glycosyltransferase 1/STAT3 axis is a driver of breast cancer aggressiveness

外骨糖基转移酶1/STAT3轴是乳腺癌侵袭性的驱动因素

DPYSL2 interacts with JAK1 to mediate breast cancer cell migration

DPYSL2 与 JAK1 相互作用介导乳腺癌细胞迁移

Overexpression of UBA5 in Cells Mimics the Phenotype of Cells Lacking UBA5

UBA5 在细胞中的过表达模仿了缺乏 UBA5 的细胞的表型

Depletion of Fumarate Hydratase, an Essential TCA Cycle Enzyme, Drives Proliferation in a Two-Step Model

延胡索酸水合酶（TCA 循环必需酶）的消耗可促进两步模型中的增殖

Large-Scale Differential Gene Expression Transcriptomic Analysis Identifies a Metabolic Signature Shared by All Cancer Cells

大规模差异基因表达转录组分析揭示所有癌细胞共有的代谢特征

The glutathione peroxidase 8 (GPX8)/IL-6/STAT3 axis is essential in maintaining an aggressive breast cancer phenotype

谷胱甘肽过氧化物酶 8 (GPX8)/IL-6/STAT3 轴对于维持侵袭性乳腺癌表型至关重要