日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Life course longitudinal growth and risk of knee osteoarthritis at age 53 years: evidence from the 1946 British birth cohort study

生命历程纵向生长与53岁时膝骨关节炎风险:来自1946年英国出生队列研究的证据

Staines, K A; Hardy, R; Samvelyan, H J; Ward, K A; Cooper, R

Endochondral Growth Defect and Deployment of Transient Chondrocyte Behaviors Underlie Osteoarthritis Onset in a Natural Murine Model

软骨内生长缺陷和瞬时软骨细胞行为的出现是自然小鼠模型中骨关节炎发病的基础

Staines, K A; Madi, K; Mirczuk, S M; Parker, S; Burleigh, A; Poulet, B; Hopkinson, M; Bodey, A J; Fowkes, R C; Farquharson, C; Lee, P D; Pitsillides, A A

Phospho1 deficiency transiently modifies bone architecture yet produces consistent modification in osteocyte differentiation and vascular porosity with ageing.

Phospho1 缺乏症会暂时改变骨骼结构,但随着年龄增长,会对骨细胞分化和血管孔隙度产生持续性改变

Javaheri B, Carriero A, Staines K A, Chang Y-M, Houston D A, Oldknow K J, Millan J L, Kazeruni Bassir N, Salmon P, Shefelbine S, Farquharson C, Pitsillides A A

miRNA-221 and miRNA-222 synergistically function to promote vascular calcification

miRNA-221 和 miRNA-222 协同作用促进血管钙化

Mackenzie, N C W; Staines, K A; Zhu, D; Genever, P; Macrae, V E

Cartilage to bone transitions in health and disease

健康和疾病状态下软骨向骨骼的过渡

Staines, K A; Pollard, A S; McGonnell, I M; Farquharson, C; Pitsillides, A A

Chondrogenic ATDC5 cells: an optimised model for rapid and physiological matrix mineralisation

软骨生成ATDC5细胞:一种用于快速生理性基质矿化的优化模型

Newton, P T; Staines, K A; Spevak, L; Boskey, A L; Teixeira, C C; Macrae, V E; Canfield, A E; Farquharson, C