Starnbach Michael N相关文献与解析，生知库

The PD-1/PD-L1 pathway is induced during Borrelia burgdorferi infection and inhibits T cell joint infiltration without compromising bacterial clearance

PD-1/PD-L1通路在伯氏疏螺旋体感染期间被激活，并抑制T细胞关节浸润，但不影响细菌清除。

期刊:PLoS Pathogens

影响因子:4.9

doi:10.1371/journal.ppat.1010903

Helble, Jennifer D; McCarthy, Julie E; Sawden, Machlan; Starnbach, Michael N; Hu, Linden T

T cell responses to Chlamydia

衣原体引起的T细胞反应

期刊:Pathogens and Disease

影响因子:2.7

doi:10.1093/femspd/ftab014

Helble, Jennifer D; Starnbach, Michael N

Antigen-specific memory and naïve CD4+ T cells following secondary Chlamydia trachomatis infection

继发性沙眼衣原体感染后的抗原特异性记忆和初始CD4+ T细胞

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0240670

Helble, Jennifer D; Mann, Alexander O; Starnbach, Michael N

A FACS-Based Genome-wide CRISPR Screen Reveals a Requirement for COPI in Chlamydia trachomatis Invasion

基于FACS的全基因组CRISPR筛选揭示了COPI在沙眼衣原体入侵中的必要性

期刊:iScience

影响因子:4.1

doi:10.1016/j.isci.2018.12.011

Park, Joseph S; Helble, Jennifer D; Lazarus, Jacob E; Yang, Guanhua; Blondel, Carlos J; Doench, John G; Starnbach, Michael N; Waldor, Matthew K

发表日期：2019

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Early Colonization of the Upper Genital Tract by Chlamydia muridarum Is Associated with Enhanced Inflammation Later in Infection

鼠衣原体早期定植于上生殖道与感染后期炎症加剧有关

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00405-19

Helble, Jennifer D; Reinhold-Larsson, Nicole V; Starnbach, Michael N

炎症/感染

Immunology/Inflammation

The Vacuolar Pathway in Macrophages Plays a Major Role in Antigen Cross-Presentation Induced by the Pore-Forming Protein Sticholysin II Encapsulated Into Liposomes

巨噬细胞中的液泡途径在由包裹在脂质体中的成孔蛋白Sticholysin II诱导的抗原交叉呈递中起着重要作用

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2018.02473

Cruz-Leal, Yoelys; Grubaugh, Daniel; Nogueira, Catarina V; Lopetegui-González, Isbel; Del Valle, Anaixis; Escalona, Felipe; Laborde, Rady J; Alvarez, Carlos; Fernández, Luis E; Starnbach, Michael N; Higgins, Darren E; Lanio, María E

Pathology after Chlamydia trachomatis infection is driven by nonprotective immune cells that are distinct from protective populations

沙眼衣原体感染后的病理变化是由非保护性免疫细胞驱动的，这些细胞与保护性免疫细胞群截然不同。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.1711356115

Lijek, Rebeccah S; Helble, Jennifer D; Olive, Andrew J; Seiger, Kyra W; Starnbach, Michael N

Chlamydia trachomatis impairs T cell priming by inducing dendritic cell death

沙眼衣原体通过诱导树突状细胞死亡来损害T细胞的启动。

Type I IFNs contribute to upregulation of PD-L1 during Chlamydia trachomatis infection

I型干扰素促进沙眼衣原体感染期间PD-L1的上调

Cytotoxic CD4+ T Cells Are Induced during Infection with Chlamydia trachomatis

沙眼衣原体感染期间会诱导细胞毒性 CD4+ T 细胞的产生

The PD-1/PD-L1 pathway is induced during Borrelia burgdorferi infection and inhibits T cell joint infiltration without compromising bacterial clearance

PD-1/PD-L1通路在伯氏疏螺旋体感染期间被激活，并抑制T细胞关节浸润，但不影响细菌清除。

T cell responses to Chlamydia

衣原体引起的T细胞反应

Antigen-specific memory and naïve CD4+ T cells following secondary Chlamydia trachomatis infection

继发性沙眼衣原体感染后的抗原特异性记忆和初始CD4+ T细胞

A FACS-Based Genome-wide CRISPR Screen Reveals a Requirement for COPI in Chlamydia trachomatis Invasion

基于FACS的全基因组CRISPR筛选揭示了COPI在沙眼衣原体入侵中的必要性

Early Colonization of the Upper Genital Tract by Chlamydia muridarum Is Associated with Enhanced Inflammation Later in Infection

鼠衣原体早期定植于上生殖道与感染后期炎症加剧有关

The Vacuolar Pathway in Macrophages Plays a Major Role in Antigen Cross-Presentation Induced by the Pore-Forming Protein Sticholysin II Encapsulated Into Liposomes

巨噬细胞中的液泡途径在由包裹在脂质体中的成孔蛋白Sticholysin II诱导的抗原交叉呈递中起着重要作用

Pathology after Chlamydia trachomatis infection is driven by nonprotective immune cells that are distinct from protective populations

沙眼衣原体感染后的病理变化是由非保护性免疫细胞驱动的，这些细胞与保护性免疫细胞群截然不同。