日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Wastewater-based surveillance for community exposome surveillance, Louisville, Kentucky

肯塔基州路易斯维尔市基于废水的社区暴露组监测

Cai, Lu; Holm, Rochelle H; Biddle, Donald J; Zhang, Charlie H; Talley, Daymond; Smith, Ted; States, J Christopher

Chronic arsenic exposure and hsa-miR-186 overexpression causes transcriptome-wide differential alternative splicing contributing to skin carcinogenesis in human HaCaT cell line

慢性砷暴露和hsa-miR-186过表达导致转录组范围内的差异性可变剪接,从而促进人HaCaT细胞系的皮肤癌发生。

Banerjee, Mayukh; Scott, Jared L; Lykoudi, Angeliki; Hwang, Jae Y; Park, Juw W; States, J Christopher

Dysregulation of mRNA expression by hsa-miR-186 overexpression in arsenic-induced skin carcinogenesis

砷诱导的皮肤癌变过程中,hsa-miR-186 过表达导致 mRNA 表达失调

Banerjee, Mayukh; Lykoudi, Angeliki; Hwang, Jae Y; Pan, Jianmin; Rai, Shesh N; Park, Juw W; States, J Christopher

DNA damage response inhibition is an early event in cadmium-induced breast carcinogenesis.

DNA损伤反应抑制是镉诱导乳腺癌发生的早期事件

Nail Alexandra N, Chavez Ashley V, Bailey Aliyah N, Banerjee Mayukh, Scott Jared L, Thomas Shelia D, States J Christopher

Chronic arsenic exposure suppresses proteasomal and autophagic protein degradation

长期砷暴露会抑制蛋白酶体和自噬途径的蛋白质降解。

Augenstein, Isabell I; Nail, Alexandra N; Ferragut Cardoso, Ana P; States, J Christopher; Banerjee, Mayukh

Chronic arsenic exposure induces malignant transformation of human HaCaT cells through both deterministic and stochastic changes in transcriptome expression

慢性砷暴露通过转录组表达的确定性和随机性变化诱导人HaCaT细胞发生恶性转化

Banerjee, Mayukh; Srivastava, Sudhir; Rai, Shesh N; States, J Christopher

Heavy-metal associated breast cancer and colorectal cancer hot spots and their demographic and socioeconomic characteristics

重金属相关乳腺癌和结直肠癌高发区及其人口统计学和社会经济特征

Tomlinson, Madeline M; Pugh, Felicia; Nail, Alexandra N; Newton, Johnnie D; Udoh, Karen; Abraham, Stephie; Kavalukas, Sandy; Guinn, Brian; Tamimi, Rulla M; Laden, Francine; Iyer, Hari S; States, J Christopher; Ruther, Matthew; Ellis, C Tyler; DuPré, Natalie C

Invited Perspective: Humanized Mice for Arsenic Metabolism-A Better Model for Investigating Arsenic-Induced Diseases?

特邀观点:人源化小鼠砷代谢模型——研究砷诱发疾病的更佳模型?

States, J Christopher; Barchowsky, Aaron

Community-level exposomics: a population-centered approach to address public health concerns

社区层面的暴露组学:以人群为中心的公共卫生问题应对方法

Stingone, Jeanette A; Geller, Andrew M; Hood, Darryl B; Makris, Konstantinos C; Mouton, Charles P; States, J Christopher; Sumner, Susan J; Wu, K Lily; Rajasekar, Arcot K

miRNAs and arsenic-induced carcinogenesis

miRNA与砷诱导的致癌作用

Nail, Alexandra N; Ferragut Cardoso, Ana P; Montero, Lakyn K; States, J Christopher