Expression of human A53T alpha-synuclein without endogenous rat alpha-synuclein fails to elicit Parkinson's disease-related phenotypes in a novel humanized rat model.
在新型人源化大鼠模型中，表达人类 A53T α-突触核蛋白而没有内源性大鼠 α-突触核蛋白，无法引起帕金森病相关表型
期刊:PLoS One
影响因子:2.6
doi:10.1371/journal.pone.0329823
Polinski Nicole K, Puoliväli Jukka, Rauhala Leena, Stenius Taina-Kaisa, Bragge Timo, Parkkari Teija, Penttinen Anna-Maija, Chen Yi, Mabrouk Omar S, Glajch Kelly E, Hirst Warren D, Perkinton Michael, Martinez Terina N, Frasier Mark A
