LGALS3 (galectin 3) mediates an unconventional secretion of SNCA/α-synuclein in response to lysosomal membrane damage by the autophagic-lysosomal pathway in human midbrain dopamine neurons

LGALS3（半乳糖凝集素3）通过自噬-溶酶体途径介导人中脑多巴胺神经元溶酶体膜损伤后SNCA/α-突触核蛋白的非常规分泌。