日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Non-Glycosylated SARS-CoV-2 Omicron BA.5 Receptor Binding Domain (RBD) with a Native-like Conformation Induces a Robust Immune Response with Potent Neutralization in a Mouse Model

具有天然样构象的非糖基化SARS-CoV-2 Omicron BA.5受体结合域(RBD)在小鼠模型中诱导强烈的免疫反应和有效的中和作用

Wongnak, Rawiwan; Brindha, Subbaian; Oba, Mami; Yoshizue, Takahiro; Islam, Md Din; Islam, M Monirul; Takemae, Hitoshi; Mizutani, Tetsuya; Kuroda, Yutaka

Antisera Produced Using an E. coli-Expressed SARS-CoV-2 RBD and Complemented with a Minimal Dose of Mammalian-Cell-Expressed S1 Subunit of the Spike Protein Exhibits Improved Neutralization

使用大肠杆菌表达的 SARS-CoV-2 RBD 生产的抗血清,辅以最低剂量的哺乳动物细胞表达的刺突蛋白 S1 亚基,表现出更好的中和作用

Takahiro Yoshizue, Subbaian Brindha, Rawiwan Wongnak, Hitoshi Takemae, Mami Oba, Tetsuya Mizutani, Yutaka Kuroda

An Escherichia coli Expressed Multi-Disulfide Bonded SARS-CoV-2 RBD Shows Native-like Biophysical Properties and Elicits Neutralizing Antisera in a Mouse Model

在大肠杆菌中表达的多二硫键结合的SARS-CoV-2 RBD表现出类似天然的生物物理特性,并在小鼠模型中诱导产生中和抗血清。

Brindha, Subbaian; Yoshizue, Takahiro; Wongnak, Rawiwan; Takemae, Hitoshi; Oba, Mami; Mizutani, Tetsuya; Kuroda, Yutaka

A Multi-Disulfide Receptor-Binding Domain (RBD) of the SARS-CoV-2 Spike Protein Expressed in E. coli Using a SEP-Tag Produces Antisera Interacting with the Mammalian Cell Expressed Spike (S1) Protein

利用SEP标签在大肠杆菌中表达的SARS-CoV-2刺突蛋白的多二硫键受体结合域(RBD)可产生与哺乳动物细胞表达的刺突蛋白(S1)相互作用的抗血清。

Brindha, Subbaian; Kuroda, Yutaka

Thermodynamic Analysis of Point Mutations Inhibiting High-Temperature Reversible Oligomerization of PDZ3

抑制PDZ3高温可逆寡聚化的点突变的热力学分析

Saotome, Tomonori; Mezaki, Taichi; Brindha, Subbaian; Unzai, Satoru; Martinez, Jose C; Kidokoro, Shun-Ichi; Kuroda, Yutaka

An in-vivo comparative evaluation of two herbal extracts Emblica officinalis and Terminalia Chebula with chlorhexidine as an anticaries agent: A preliminary study

余甘子和诃子两种草药提取物与氯己定作为抗龋剂的体内比较评价:一项初步研究

Velmurugan, Arumugam; Madhubala, Manavalan Madhana; Bhavani, Sreekrishnapillai; Satheesh Kumar, Kasinathan Subbaian; Sathyanarayana, Srinivasan Sai; Gurucharan, Natanasikamani