Deletion of the murine ortholog of human 9p21.3 locus promotes atherosclerosis by increasing macrophage proinflammatory activity
小鼠9p21.3位点同源基因的缺失可通过增强巨噬细胞的促炎活性促进动脉粥样硬化。
期刊:Frontiers in Cardiovascular Medicine
影响因子:2.9
doi:10.3389/fcvm.2023.1113890
Kettunen, Sanna; Ruotsalainen, Anna-Kaisa; Örd, Tiit; Suoranta, Tuisku; Heikkilä, Janne; Kaikkonen, Minna U; Laham-Karam, Nihay; Ylä-Herttuala, Seppo
巨噬细胞
巨噬细胞
心血管
细胞生物学
动脉粥样硬化
免疫/内分泌