日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Mapping cellular vulnerability in Parkinson's disease using retro-AAVs and preformed α-synuclein fibrils.

利用逆转录AAV和预先形成的α-突触核蛋白原纤维绘制帕金森病细胞脆弱性图谱。

Geibl Fanni F, Musa Ahmed A S, Dietrich Leo, Wolter Helena, Wokosin David L, Khudayberdiev Sharof, Rust Marco B, Chen Rong, Dawson Valina L, Dawson Ted M, Oertel Wolfgang H, Surmeier D James, Henrich Martin T

N-acetyl-l-leucine lowers α-synuclein levels and improves synaptic function in Parkinson's disease models.

N-乙酰-L-亮氨酸可降低α-突触核蛋白水平,并改善帕金森病模型中的突触功能。

Song Pingping, Chen Chuyu, Franchini Rossella, Duong Bryan, Wang Yi-Zhi, Coukos Robert, Xie Zhong, Savas Jeffrey N, Zhou Yueqin, Bertoldi Mariarita, Surmeier D James, Parisiadou Loukia, Krainc Dimitri

Activity-Dependent Regulation of Basal Forebrain Cholinergic Neurons by Striatal Spiny Projection Neurons

纹状体棘状投射神经元对基底前脑胆碱能神经元的活动依赖性调节

Sun, Linqing; Zampese, Enrico; Pancani, Tristano; Wokosin, David; Tkatch, Tatiana; Surmeier, D James

Alpha-synuclein seed amplification assays: Data sharing, standardization needed for clinical use

α-突触核蛋白种子扩增检测:临床应用需要数据共享和标准化。

Lashuel, Hilal A; Surmeier, D James; Simuni, Tanya; Merchant, Kalpana; Caughey, Byron; Soto, Claudio; Fares, Mohamed-Bilal; Heym, Roland G; Melki, Ronald

State-dependent modulation of spiny projection neurons controls levodopa-induced dyskinesia in a mouse model of Parkinson's disease

棘状投射神经元的状态依赖性调节控制着帕金森病小鼠模型中左旋多巴诱导的运动障碍

Zhai, Shenyu; Cui, Qiaoling; Wokosin, David; Sun, Linqing; Tkatch, Tatiana; Crittenden, Jill R; Graybiel, Ann M; Surmeier, D James

Supramolecular nanostructure mimics GDNF trophic effects in vitro on human dopaminergic neurons.

超分子纳米结构在体外模拟 GDNF 对人类多巴胺能神经元的营养作用

Carballo-Molina Oscar A, Kolberg-Edelbrock Alexandra N, Alvarez-Saavedra Matías, Álvarez Zaida, Fyrner Timmy, Perez-Rosello Tamara, Syrgiannis Zois, Chin Stacey M, Takata Nozomu, Strong Madison, Palmer Liam C, James Surmeier D, Stupp Samuel I

Genetic modulation of mitochondrial NAD+ regeneration does not prevent dopaminergic neuron dysfunction caused by mitochondrial complex I impairment

基因调控线粒体NAD+再生并不能阻止由线粒体复合物I损伤引起的多巴胺能神经元功能障碍。

D'Alessandro, Karis B; Zampese, Enrico; Blum, Jenna L E; Kuusik, Britta; Palmiotti, Alec; Davidson, Shawn M; Reczek, Colleen R; Surmeier, D James; Chandel, Navdeep S

Ethical disclosure of biomarkers for Alzheimer risk in Latin American participants

在拉丁美洲参与者中,阿尔茨海默病风险生物标志物的伦理披露

Oviedo, Diana C; Haughbrook, Rasheda; Culjat, Carli; Ramirez Surmeier, Ladanya; Tratner, Adam E; Carreira, María B; Villarreal, Alcibiades E; Harmon, Sherelle L; Batista, Oriana I; Meng, Zhuo; Millender, Eugenia; Xavier Hall, Casey D; Britton, Gabrielle B

Genetic modulation of mitochondrial NAD+ regeneration does not prevent dopaminergic neuron dysfunction caused by mitochondrial complex I impairment.

基因调控线粒体 NAD+ 再生并不能阻止由线粒体复合物 I 损伤引起的多巴胺能神经元功能障碍

D'Alessandro Karis B, Zampese Enrico, Blum Jenna L E, Kuusik Britta, Palmiotti Alec, Davidson Shawn M, Reczek Colleen R, Surmeier D James, Chandel Navdeep S

State-dependent modulation of spiny projection neurons controls levodopa-induced dyskinesia in a mouse model of Parkinson's disease.

在帕金森病小鼠模型中,棘状投射神经元的状态依赖性调节控制左旋多巴诱发的运动障碍

Zhai Shenyu, Cui Qiaoling, Wokosin David, Sun Linqing, Tkatch Tatiana, Crittenden Jill R, Graybiel Ann M, Surmeier D James