Suvendra N相关文献与解析，生知库 | 链接生命科学的每一步

The role of miRNAs in the development of Super-Tregs as a potential therapy for neurodegenerative diseases

miRNA在超级调节性T细胞发育中的作用及其作为神经退行性疾病潜在疗法的意义

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2026.1708031

Mukherjee, Kamalika; Bhattacharyya, Suvendra N

HuR-driven reversible mitochondrial shuttling buffers cytosolic miRNA levels in hepatic cells

HuR驱动的可逆线粒体穿梭调节肝细胞胞质miRNA水平

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1016/j.jbc.2026.111255

Banerjee, Saikat; Hom Choudhury, Sourav; Chatterjee, Susanta; Hu, Guoku; Mukherjee, Kamalika; Bhattacharyya, Suvendra N

Change of guards: ELAVL proteins switch miRNA export responsibility to regulate differentiation of PC12 cells

角色转换：ELAVL蛋白转变miRNA输出职责，调控PC12细胞分化

期刊:RNA Biology

影响因子:3.4

doi:10.1080/15476286.2026.2647903

Ray, Sritama; Bhattacharyya, Suvendra N; Mukherjee, Kamalika

Export of miRNAs from activated macrophages is cooperative and HuR-dependent

活化巨噬细胞中 miRNA 的输出是协同的，并且依赖于 HuR。

期刊:FEBS Letters

影响因子:3

doi:10.1002/1873-3468.70309

Ghosh, Syamantak; Mukherjee, Kamalika; Bhattacharyya, Suvendra N

Ras Homolog Enriched in Brain Protein Reverses Amyloid Beta-Induced Escape of Inflammatory Cytokine mRNAs From Immunoisolated RNA Processing Bodies of Glioblastoma Cells

脑蛋白中富集的Ras同源物逆转淀粉样β蛋白诱导的炎症细胞因子mRNA从胶质母细胞瘤细胞免疫分离的RNA加工体中逃逸

期刊:FASEB Journal

影响因子:4.2

doi:10.1096/fj.202502872R

Ray, Sritama; Mukherjee, Kamalika; Bhattacharyya, Suvendra N

Protocol to study internalization and localization dynamics of exogenously added proteins in detergent-permeabilized human cells.

研究外源添加蛋白质在去垢剂透化的人类细胞中的内化和定位动态的方案

期刊:STAR Protocols

影响因子:1.3

doi:10.1016/j.xpro.2025.103992

Ray Sritama, Mukherjee Kamalika, Bhattacharyya Suvendra N

Human antigen R transfers miRNA to Syntaxin 5 to synergize miRNA export from activated macrophages

人类抗原 R 将 miRNA 转移到突触融合蛋白 5，以协同激活巨噬细胞中的 miRNA 输出

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1016/j.jbc.2024.107170

Sourav Hom Choudhury, Shreya Bhattacharjee, Kamalika Mukherjee, Suvendra N Bhattacharyya

HuR-miRNA complex activates RAS GTPase RalA to facilitate endosome targeting and extracellular export of miRNAs

HuR-miRNA复合物激活RAS GTP酶RalA，促进miRNA的内体靶向和细胞外输出。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1016/j.jbc.2024.105750

Ghosh, Syamantak; Hom Choudhury, Sourav; Mukherjee, Kamalika; Bhattacharyya, Suvendra N

Accelerated export of Dicer1 from lipid-challenged hepatocytes buffers cellular miRNA-122 levels and prevents cell death

脂质刺激下肝细胞中Dicer1的加速输出可缓冲细胞内miRNA-122水平并防止细胞死亡

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1016/j.jbc.2023.104999

Bandyopadhyay, Diptankar; Basu, Sudarshana; Mukherjee, Ishita; Chakrabarti, Saikat; Chakrabarti, Partha; Mukherjee, Kamalika; Bhattacharyya, Suvendra N

Rescue of mitochondrial power outages with γPNA-based miR-122 inhibitor

利用基于γPNA的miR-122抑制剂挽救线粒体功能障碍

The role of miRNAs in the development of Super-Tregs as a potential therapy for neurodegenerative diseases

miRNA在超级调节性T细胞发育中的作用及其作为神经退行性疾病潜在疗法的意义

HuR-driven reversible mitochondrial shuttling buffers cytosolic miRNA levels in hepatic cells

HuR驱动的可逆线粒体穿梭调节肝细胞胞质miRNA水平

Change of guards: ELAVL proteins switch miRNA export responsibility to regulate differentiation of PC12 cells

角色转换：ELAVL蛋白转变miRNA输出职责，调控PC12细胞分化

Export of miRNAs from activated macrophages is cooperative and HuR-dependent

活化巨噬细胞中 miRNA 的输出是协同的，并且依赖于 HuR。

Ras Homolog Enriched in Brain Protein Reverses Amyloid Beta-Induced Escape of Inflammatory Cytokine mRNAs From Immunoisolated RNA Processing Bodies of Glioblastoma Cells

脑蛋白中富集的Ras同源物逆转淀粉样β蛋白诱导的炎症细胞因子mRNA从胶质母细胞瘤细胞免疫分离的RNA加工体中逃逸

Protocol to study internalization and localization dynamics of exogenously added proteins in detergent-permeabilized human cells.

研究外源添加蛋白质在去垢剂透化的人类细胞中的内化和定位动态的方案

Human antigen R transfers miRNA to Syntaxin 5 to synergize miRNA export from activated macrophages

人类抗原 R 将 miRNA 转移到突触融合蛋白 5，以协同激活巨噬细胞中的 miRNA 输出

HuR-miRNA complex activates RAS GTPase RalA to facilitate endosome targeting and extracellular export of miRNAs

HuR-miRNA复合物激活RAS GTP酶RalA，促进miRNA的内体靶向和细胞外输出。

Accelerated export of Dicer1 from lipid-challenged hepatocytes buffers cellular miRNA-122 levels and prevents cell death

脂质刺激下肝细胞中Dicer1的加速输出可缓冲细胞内miRNA-122水平并防止细胞死亡