日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Dual targeting of BRAF(V600E) and ferroptosis results in synergistic anticancer activity via iron overload and enhanced oxidative stress.

BRAF(V600E) 和铁死亡的双重靶向作用通过铁过载和增强氧化应激产生协同抗癌活性。

Hu Jiangnan, Ghosh Chandrayee, Khaket Tejinder P, Yang Zhongyue, Tabdili Yasmine, Alamaw Eden D, Boufraqech Myriem, Dixon Scott J, Kebebew Electron

Corrigendum to "Combination nitazoxanide and auranofin treatment has synergistic anticancer activity in anaplastic thyroid cancer through enhanced activation of oxidative stress that leads to apoptosis" [Cancer Letters 633 (2025) 217990]

对“尼他唑尼和金诺芬联合治疗通过增强氧化应激的激活导致细胞凋亡,对间变性甲状腺癌具有协同抗癌活性”的更正[Cancer Letters 633 (2025) 217990]

Ghosh, Chandrayee; Khaket, Tejinder Pal; Gunda, Viswanath; Yang, Zhongyue; Hu, Jiangnan; Alamaw, Eden Demissie; Zhang, Lisa; Zhang, Ya-Qin; Shen, Min; Tabdili, Yasmine; Boufraqech, Myriem; Kassu, Rodas; Kebebew, Electron

Combination nitazoxanide and auranofin treatment has synergistic anticancer activity in anaplastic thyroid cancer through enhanced activation of oxidative stress that leads to apoptosis.

硝唑尼特和金诺芬联合治疗对间变性甲状腺癌具有协同抗癌活性,其机制是通过增强氧化应激的激活,从而导致细胞凋亡。

Ghosh Chandrayee, Khaket Tejinder Pal, Gunda Viswanath, Yang Zhongyue, Hu Jiangnan, Alamaw Eden Demissie, Zhang Lisa, Zhang Ya-Qin, Shen Min, Tabdili Yasmine, Boufraqech Myriem, Kassu Rodas, Kebebew Electron