日期 影响因子
日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:
–

Restoration of Shal/K(V)4 proteostasis and motor function in a Drosophila model of spinocerebellar ataxia type 19/22.

在果蝇脊髓小脑性共济失调 19/22 型模型中恢复 Shal/K(V)4 蛋白稳态和运动功能

期刊:Cellular and Molecular Life Sciences
影响因子:6.2
doi:10.1007/s00018-025-05711-y
Hsiao Cheng-Tsung, Fu Ssu-Ju, Cheng Kai-Min, Lo Hsiang, Tang Chih-Yung, Chan Chih-Chiang, Jeng Chung-Jiuan
免疫/内分泌
Drosophila
发表日期：2025
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Regulation of testosterone synthesis in Leydig cells by ClC-2 chloride channel.

ClC-2氯离子通道对睾丸间质细胞中睾酮合成的调节

期刊:Reproduction
影响因子:3.7
doi:10.1530/REP-24-0432
Fu Ssu-Ju, Syu Min-Shan, Tang Chih-Yu, Huang Ching-Yuan, Jeng Chung-Jiuan, Tang Chih-Yung, Hu Meng-Chun
细胞生物学
发表日期：2025
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Novel KCND3 Variant Underlying Nonprogressive Congenital Ataxia or SCA19/22 Disrupt K(V)4.3 Protein Expression and K+ Currents with Variable Effects on Channel Properties

一种导致非进行性先天性共济失调或SCA19/22的新型KCND3变异体，会破坏K(V)4.3蛋白的表达和K+电流，并对通道特性产生不同的影响。

期刊:International Journal of Molecular Sciences
影响因子:4.9
doi:10.3390/ijms22094986
Zanni, Ginevra; Hsiao, Cheng-Tsung; Fu, Ssu-Ju; Tang, Chih-Yung; Capuano, Alessandro; Bosco, Luca; Graziola, Federica; Bellacchio, Emanuele; Servidei, Serenella; Primiano, Guido; Soong, Bing-Wen; Jeng, Chung-Jiuan
发表日期：2021
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Defective Gating and Proteostasis of Human ClC-1 Chloride Channel: Molecular Pathophysiology of Myotonia Congenita

人类ClC-1氯离子通道的门控和蛋白稳态缺陷：先天性肌强直的分子病理生理学

期刊:Frontiers in Neurology
影响因子:2.8
doi:10.3389/fneur.2020.00076
Jeng, Chung-Jiuan; Fu, Ssu-Ju; You, Chia-Ying; Peng, Yi-Jheng; Hsiao, Cheng-Tsung; Chen, Tsung-Yu; Tang, Chih-Yung
Human
发表日期：2020
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Calmodulin modulates the Ca(2+)-dependent inactivation and expression level of bovine Ca(V)2.2 expressed in HEK293T cells

钙调蛋白调节HEK293T细胞中表达的牛Ca(V)2.2的Ca(2+)依赖性失活和表达水平。

期刊:IBRO Reports
影响因子:
doi:10.1016/j.ibror.2017.03.002
Chi, Chih-Hung; Tang, Chih-Yung; Pan, Chien-Yuan
细胞生物学
T细胞
T细胞
免疫/内分泌
发表日期：2017
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The episodic ataxia type 1 mutation I262T alters voltage-dependent gating and disrupts protein biosynthesis of human Kv1.1 potassium channels

发作性共济失调1型突变I262T会改变电压依赖性门控并破坏人Kv1.1钾通道的蛋白质生物合成。

期刊:Scientific Reports
影响因子:3.9
doi:10.1038/srep19378
Chen, Szu-Han; Fu, Ssu-Ju; Huang, Jing-Jia; Tang, Chih-Yung
发表日期：2016
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Myotonia congenita mutation enhances the degradation of human CLC-1 chloride channels

先天性肌强直突变会增强人CLC-1氯离子通道的降解

期刊:PLoS One
影响因子:2.6
doi:10.1371/journal.pone.0055930
Lee, Ting-Ting; Zhang, Xiao-Dong; Chuang, Chao-Chin; Chen, Jing-Jer; Chen, Yi-An; Chen, Shu-Ching; Chen, Tsung-Yu; Tang, Chih-Yung
强直性脊柱炎
CLC
发表日期：2013
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Dominantly inherited myotonia congenita resulting from a mutation that increases open probability of the muscle chloride channel CLC-1

显性遗传性先天性肌强直是由一种突变引起的，该突变增加了肌肉氯离子通道CLC-1的开放概率。

期刊:Neuromolecular Medicine
影响因子:3.9
doi:10.1007/s12017-012-8190-1
Richman, David P; Yu, Yawei; Lee, Ting-Ting; Tseng, Pang-Yen; Yu, Wei-Ping; Maselli, Ricardo A; Tang, Chih-Yung; Chen, Tsung-Yu
强直性脊柱炎
CLC
发表日期：2012
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