Taya Yoichi相关文献与解析，生知库 | 链接生命科学的每一步

Otomo, Ryo; Otsubo, Chihiro; Matsushima-Hibiya, Yuko; Miyazaki, Makoto; Tashiro, Fumio; Ichikawa, Hitoshi; Kohno, Takashi; Ochiya, Takahiro; Yokota, Jun; Nakagama, Hitoshi; Taya, Yoichi; Enari, Masato

成纤维细胞

细胞生物学

肿瘤

肿瘤免疫

发表日期：2014

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Distinct and site-specific phosphorylation of the retinoblastoma protein at serine 612 in differentiated cells

分化细胞中视网膜母细胞瘤蛋白在丝氨酸612位点的特异性磷酸化

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0086709

Hattori, Takayuki; Uchida, Chiharu; Takahashi, Hirotaka; Yamamoto, Naoki; Naito, Mikihiko; Taya, Yoichi

Induction of ZEB proteins by inactivation of RB protein is key determinant of mesenchymal phenotype of breast cancer

RB蛋白失活诱导ZEB蛋白表达是乳腺癌间质表型的关键决定因素。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M111.313759

Arima, Yoshimi; Hayashi, Hidemi; Sasaki, Mikako; Hosonaga, Mari; Goto, Takaaki M; Chiyoda, Tatsuyuki; Kuninaka, Shinji; Shibata, Tatsuhiro; Ohata, Hirokazu; Nakagama, Hitoshi; Taya, Yoichi; Saya, Hideyuki

Cytoplasmic tethering is involved in synergistic inhibition of p53 by Mdmx and Mdm2

细胞质束缚参与了Mdmx和Mdm2对p53的协同抑制作用。

期刊:Cancer Science

影响因子:4.3

doi:10.1111/j.1349-7006.2009.01180.x

Ohtsubo, Chihiro; Shiokawa, Daisuke; Kodama, Masami; Gaiddon, Christian; Nakagama, Hitoshi; Jochemsen, Aart G; Taya, Yoichi; Okamoto, Koji

细胞生物学

发表日期：2009

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Hedgehog signaling overrides p53-mediated tumor suppression by activating Mdm2.

Hedgehog信号通路通过激活Mdm2来抑制p53介导的肿瘤生长

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.0712216105

Abe Yoshinori, Oda-Sato Eri, Tobiume Kei, Kawauchi Keiko, Taya Yoichi, Okamoto Koji, Oren Moshe, Tanaka Nobuyuki

Phosphorylation of pRB at Ser612 by Chk1/2 leads to a complex between pRB and E2F-1 after DNA damage

DNA损伤后，Chk1/2对pRB Ser612位点的磷酸化导致pRB与E2F-1形成复合物。

期刊:EMBO Journal

影响因子:8.3

doi:10.1038/sj.emboj.7601652

Inoue, Yasumichi; Kitagawa, Masatoshi; Taya, Yoichi

Dissecting functional roles of p53 N-terminal transactivation domains by microarray expression analysis

利用微阵列表达分析解析p53 N端转录激活结构域的功能作用

期刊:Cancer Science

影响因子:4.3

doi:10.1111/j.1349-7006.2006.00375.x

Ohki, Rieko; Kawase, Tatsuya; Ohta, Tsutomu; Ichikawa, Hitoshi; Taya, Yoichi

发表日期：2007

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Differential roles of ATM- and Chk2-mediated phosphorylations of Hdmx in response to DNA damage.

ATM 和 Chk2 介导的 Hdmx 磷酸化在 DNA 损伤反应中的不同作用

期刊:Molecular and Cellular Biology

影响因子:2.7

doi:10.1128/MCB.00562-06

Pereg Yaron, Lam Suzanne, Teunisse Amina, Biton Sharon, Meulmeester Erik, Mittelman Leonid, Buscemi Giacomo, Okamoto Koji, Taya Yoichi, Shiloh Yosef, Jochemsen Aart G

DNA damage-induced phosphorylation of MdmX at serine 367 activates p53 by targeting MdmX for Mdm2-dependent degradation

DNA损伤诱导的MdmX在丝氨酸367位点的磷酸化激活p53，其机制是通过靶向MdmX进行Mdm2依赖性降解。

期刊:Molecular and Cellular Biology

影响因子:2.7

doi:10.1128/MCB.25.21.9608-9620.2005

Okamoto, Koji; Kashima, Kenji; Pereg, Yaron; Ishida, Michiko; Yamazaki, Satomi; Nota, Ayumi; Teunisse, Amina; Migliorini, Domenico; Kitabayashi, Issay; Marine, Jean-Christophe; Prives, Carol; Shiloh, Yosef; Jochemsen, Aart G; Taya, Yoichi

Endoplasmic reticulum stress induces p53 cytoplasmic localization and prevents p53-dependent apoptosis by a pathway involving glycogen synthase kinase-3beta

内质网应激诱导p53胞质定位，并通过涉及糖原合成酶激酶-3β的通路抑制p53依赖性细胞凋亡。

期刊:Genes & Development

影响因子:7.7

doi:10.1101/gad.1165804

Qu, LiKe; Huang, Shirley; Baltzis, Dionissios; Rivas-Estilla, Ana-Maria; Pluquet, Olivier; Hatzoglou, Maria; Koumenis, Costas; Taya, Yoichi; Yoshimura, Akihiko; Koromilas, Antonis E

TSPAN12 is a critical factor for cancer-fibroblast cell contact-mediated cancer invasion

TSPAN12是癌细胞-成纤维细胞接触介导的癌细胞侵袭的关键因素。

Distinct and site-specific phosphorylation of the retinoblastoma protein at serine 612 in differentiated cells

分化细胞中视网膜母细胞瘤蛋白在丝氨酸612位点的特异性磷酸化

Induction of ZEB proteins by inactivation of RB protein is key determinant of mesenchymal phenotype of breast cancer

RB蛋白失活诱导ZEB蛋白表达是乳腺癌间质表型的关键决定因素。

Cytoplasmic tethering is involved in synergistic inhibition of p53 by Mdmx and Mdm2

细胞质束缚参与了Mdmx和Mdm2对p53的协同抑制作用。

Hedgehog signaling overrides p53-mediated tumor suppression by activating Mdm2.

Hedgehog信号通路通过激活Mdm2来抑制p53介导的肿瘤生长

Phosphorylation of pRB at Ser612 by Chk1/2 leads to a complex between pRB and E2F-1 after DNA damage

DNA损伤后，Chk1/2对pRB Ser612位点的磷酸化导致pRB与E2F-1形成复合物。

Dissecting functional roles of p53 N-terminal transactivation domains by microarray expression analysis

利用微阵列表达分析解析p53 N端转录激活结构域的功能作用

Differential roles of ATM- and Chk2-mediated phosphorylations of Hdmx in response to DNA damage.

ATM 和 Chk2 介导的 Hdmx 磷酸化在 DNA 损伤反应中的不同作用

DNA damage-induced phosphorylation of MdmX at serine 367 activates p53 by targeting MdmX for Mdm2-dependent degradation

DNA损伤诱导的MdmX在丝氨酸367位点的磷酸化激活p53，其机制是通过靶向MdmX进行Mdm2依赖性降解。

Endoplasmic reticulum stress induces p53 cytoplasmic localization and prevents p53-dependent apoptosis by a pathway involving glycogen synthase kinase-3beta

内质网应激诱导p53胞质定位，并通过涉及糖原合成酶激酶-3β的通路抑制p53依赖性细胞凋亡。