Taye Nandaraj相关文献与解析，生知库 | 链接生命科学的每一步

Taye Nandaraj, Karoulias Stylianos Z, Balic Zerina, Wang Lauren W, Willard Belinda B, Martin Daniel, Richard Daniel, Okamoto Alexander S, Capellini Terence D, Apte Suneel S, Hubmacher Dirk

ADA

发表日期：2025

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En route towards a personalized medicine approach: Innovative therapeutic modalities for connective tissue disorders

迈向个性化医疗之路：结缔组织疾病的创新疗法

期刊:Matrix Biology

影响因子:4.8

doi:10.1016/j.matbio.2023.08.005

Redhead, Charlene; Taye, Nandaraj; Hubmacher, Dirk

发表日期：2023

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Stable Knockdown of Genes Encoding Extracellular Matrix Proteins in the C2C12 Myoblast Cell Line Using Small-Hairpin (sh)RNA.

利用小发夹（sh）RNA稳定敲低C2C12成肌细胞系中编码细胞外基质蛋白的基因

期刊:Jove-Journal of Visualized Experiments

影响因子:1

doi:10.3791/60824

Taye Nandaraj, Stanley Sarah, Hubmacher Dirk

细胞生物学

发表日期：2020

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SMAR1 favors immunosurveillance of cancer cells by modulating calnexin and MHC I expression.

SMAR1 通过调节钙网蛋白和 MHC I 的表达来促进对癌细胞的免疫监视

期刊:Neoplasia

影响因子:7.7

doi:10.1016/j.neo.2019.07.002

Alam Aftab, Taye Nandaraj, Patel Sonal, Thube Milind, Mullick Jayati, Shah Vibhuti Kumar, Pant Richa, Roychowdhury Tanaya, Banerjee Nilanjan, Chatterjee Subhrangsu, Bhattacharya Rittwika, Roy Rini, Mukhopadhyay Ashis, Mogare Devraj, Chattopadhyay Samit

Limb- and tendon-specific Adamtsl2 deletion identifies a role for ADAMTSL2 in tendon growth in a mouse model for geleophysic dysplasia

肢体和肌腱特异性 Adamtsl2 缺失揭示了 ADAMTSL2 在胶状骨发育不良小鼠模型中肌腱生长中的作用

期刊:Matrix Biology

影响因子:4.8

doi:10.1016/j.matbio.2019.02.001

Hubmacher, Dirk; Taye, Nandaraj; Balic, Zerina; Thacker, Stetson; Adams, Sheila M; Birk, David E; Schweitzer, Ronen; Apte, Suneel S

Late-phase synthesis of IκBα insulates the TLR4-activated canonical NF-κB pathway from noncanonical NF-κB signaling in macrophages

巨噬细胞中 IκBα 的后期合成将 TLR4 激活的经典 NF-κB 通路与非经典 NF-κB 信号通路隔离开来。

期刊:Science Signaling

影响因子:6.6

doi:10.1126/scisignal.aaf1129

Chatterjee, Budhaditya; Banoth, Balaji; Mukherjee, Tapas; Taye, Nandaraj; Vijayaragavan, Bharath; Chattopadhyay, Samit; Gomes, James; Basak, Soumen

Combined ADAMTS10 and ADAMTS17 inactivation exacerbates bone shortening and skin phenotypes.

ADAMTS10 和 ADAMTS17 的联合失活会加剧骨骼缩短和皮肤表型。

En route towards a personalized medicine approach: Innovative therapeutic modalities for connective tissue disorders

迈向个性化医疗之路：结缔组织疾病的创新疗法

Stable Knockdown of Genes Encoding Extracellular Matrix Proteins in the C2C12 Myoblast Cell Line Using Small-Hairpin (sh)RNA.

利用小发夹（sh）RNA稳定敲低C2C12成肌细胞系中编码细胞外基质蛋白的基因

SMAR1 favors immunosurveillance of cancer cells by modulating calnexin and MHC I expression.

SMAR1 通过调节钙网蛋白和 MHC I 的表达来促进对癌细胞的免疫监视

Limb- and tendon-specific Adamtsl2 deletion identifies a role for ADAMTSL2 in tendon growth in a mouse model for geleophysic dysplasia

肢体和肌腱特异性 Adamtsl2 缺失揭示了 ADAMTSL2 在胶状骨发育不良小鼠模型中肌腱生长中的作用

Late-phase synthesis of IκBα insulates the TLR4-activated canonical NF-κB pathway from noncanonical NF-κB signaling in macrophages

巨噬细胞中 IκBα 的后期合成将 TLR4 激活的经典 NF-κB 通路与非经典 NF-κB 信号通路隔离开来。