日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Development of peribronchiolar fibrosis is associated with local immune and MAPK pathway activation in a murine model of deployment-related constrictive bronchiolitis

在小鼠部署相关性缩窄性细支气管炎模型中,支气管周围纤维化的发展与局部免疫和MAPK通路激活有关。

Teitz-Tennenbaum, Seagal; Marinetti, Kayla N; Ganguly, Anutosh; Song, Brian; Hicks, Helen; Osterholzer, John J

Sulfur dioxide exposure of mice induces peribronchiolar fibrosis-A defining feature of deployment-related constrictive bronchiolitis

小鼠暴露于二氧化硫可诱发支气管周围纤维化——这是部署相关性缩窄性支气管炎的一个典型特征。

Teitz-Tennenbaum, Seagal; Marinetti, Kayla N; Lahiri, Shayanki; Siddiqui, Khadijah; Flory, Celia; Tennenbaum, Karinne; Hicks, Helen G; Song, Brian; Ganguly, Anutosh; Osterholzer, John J

CD36/Lyn kinase interactions within macrophages promotes pulmonary fibrosis in response to oxidized phospholipid

巨噬细胞内的 CD36/Lyn 激酶相互作用促进氧化磷脂引起的肺纤维化

Doyun Kwak, Patrick B Bradley, Natalia Subbotina, Song Ling, Seagal Teitz-Tennenbaum, John J Osterholzer, Thomas H Sisson #, Kevin K Kim #

Sustained Club Cell Injury in Mice Induces Histopathologic Features of Deployment-Related Constrictive Bronchiolitis

小鼠持续性支气管肺泡细胞损伤可诱发与部署相关性闭塞性细支气管炎的组织病理学特征

Teitz-Tennenbaum, Seagal; Viglianti, Steven P; Jomma, Ahmad; Palone, Quentin; Andrews, Halia; Selbmann, Kayla N; Lahiri, Shayanki; Subbotina, Natalia; Walker, Natalie; Perl, Anne-Karina T; Lama, Vibha N; Sisson, Thomas H; Osterholzer, John J

Diverse Injury Pathways Induce Alveolar Epithelial Cell CCL2/12, Which Promotes Lung Fibrosis

多种损伤途径诱导肺泡上皮细胞CCL2/12促进肺纤维化

Jibing Yang, Manisha Agarwal, Song Ling, Seagal Teitz-Tennenbaum, Rachel L Zemans, John J Osterholzer, Thomas H Sisson, Kevin K Kim

Urokinase Plasminogen Activator Overexpression Reverses Established Lung Fibrosis

尿激酶型纤溶酶原激活剂过表达可逆转已形成的肺纤维化

Horowitz, Jeffrey C; Tschumperlin, Daniel J; Kim, Kevin K; Osterholzer, John J; Subbotina, Natalya; Ajayi, Iyabode O; Teitz-Tennenbaum, Seagal; Virk, Ammara; Dotson, Megan; Liu, Fei; Sicard, Delphine; Jia, Shijing; Sisson, Thomas H

Autocrine IL-10 Signaling Promotes Dendritic Cell Type-2 Activation and Persistence of Murine Cryptococcal Lung Infection

自分泌IL-10信号促进2型树突状细胞活化和小鼠隐球菌肺部感染的持续存在

Teitz-Tennenbaum, Seagal; Viglianti, Steven P; Roussey, Jonathan A; Levitz, Stuart M; Olszewski, Michal A; Osterholzer, John J

Anti-PD-1 Antibody Treatment Promotes Clearance of Persistent Cryptococcal Lung Infection in Mice

抗 PD-1 抗体治疗促进小鼠持续性隐球菌肺部感染的清除

Jonathan A Roussey, Steven P Viglianti, Seagal Teitz-Tennenbaum, Michal A Olszewski, John J Osterholzer

Local GM-CSF-Dependent Differentiation and Activation of Pulmonary Dendritic Cells and Macrophages Protect against Progressive Cryptococcal Lung Infection in Mice

局部 GM-CSF 依赖的肺树突状细胞和巨噬细胞的分化和激活可预防小鼠进行性隐球菌肺部感染

Gwo-Hsiao Chen, Seagal Teitz-Tennenbaum, Lori M Neal, Benjamin J Murdock, Antoni N Malachowski, Anthony J Dils, Michal A Olszewski, John J Osterholzer

Early or late IL-10 blockade enhances Th1 and Th17 effector responses and promotes fungal clearance in mice with cryptococcal lung infection

早期或晚期 IL-10 阻断可增强 Th1 和 Th17 效应反应并促进隐球菌肺部感染小鼠的真菌清除

Benjamin J Murdock, Seagal Teitz-Tennenbaum, Gwo-Hsiao Chen, Anthony J Dils, Antoni N Malachowski, Jeffrey L Curtis, Michal A Olszewski, John J Osterholzer