Terrand相关文献与解析，生知库 | 链接生命科学的每一步

Awan, Sara; Lambert, Magalie; Imtiaz, Ali; Alpy, Fabien; Tomasetto, Catherine; Oulad-Abdelghani, Mustapha; Schaeffer, Christine; Moritz, Chloé; Julien-David, Diane; Najib, Souad; Martinez, Laurent O; Matz, Rachel L; Collet, Xavier; Silva-Rojas, Roberto; Böhm, Johann; Herz, Joachim; Terrand, Jérôme; Boucher, Philippe

Wnt/β-Catenin

心血管

动脉粥样硬化

代谢

发表日期：2022

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ShcA promotes chondrocyte hypertrophic commitment and osteoarthritis in mice through RunX2 nuclear translocation and YAP1 inactivation

ShcA 通过 RunX2 核转位和 YAP1 失活促进小鼠软骨细胞肥大和骨关节炎

期刊:Osteoarthritis and Cartilage

影响因子:7.2

doi:10.1016/j.joca.2022.07.001

A Abou-Jaoude, M Courtes, L Badique, D Elhaj Mahmoud, C Abboud, M Mlih, H Justiniano, M Milbach, M Lambert, A Lemle, S Awan, J Terrand, A Niemeier, A Barbero, X Houard, P Boucher, R L Matz

Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation

内皮细胞中衔接蛋白ShcA的缺失可防止单核细胞与巨噬细胞黏附、低密度脂蛋白氧化以及动脉粥样硬化病变的形成。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-018-22819-3

Abou-Jaoude, Antoine; Badiqué, Lise; Mlih, Mohamed; Awan, Sara; Guo, Sunning; Lemle, Alexandre; Abboud, Clauda; Foppolo, Sophie; Host, Lionel; Terrand, Jérôme; Justiniano, Hélène; Herz, Joachim; Matz, Rachel L; Boucher, Philippe

Author Correction: Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation

作者更正：内皮细胞中衔接蛋白ShcA的缺失可防止单核细胞与巨噬细胞黏附、低密度脂蛋白氧化以及动脉粥样硬化病变的形成。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/s41598-018-27564-1

Convergent Signaling Pathways Controlled by LRP1 (Receptor-related Protein 1) Cytoplasmic and Extracellular Domains Limit Cellular Cholesterol Accumulation.

由 LRP1（受体相关蛋白 1）胞质和胞外结构域控制的汇聚信号通路限制细胞胆固醇的积累

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M116.714485

El Asmar Zeina, Terrand JÃ©rome, Jenty Marion, Host Lionel, Mlih Mohamed, Zerr AurÃ©lie, Justiniano HÃ©lÃ¨ne, Matz Rachel L, Boudier Christian, Scholler Estelle, Garnier Jean-Marie, Bertaccini Diego, ThiersÃ© DaniÃ¨le, Schaeffer Christine, Van Dorsselaer Alain, Herz Joachim, Bruban VÃ©ronique, Boucher Philippe

The Src homology and collagen A (ShcA) adaptor protein is required for the spatial organization of the costamere/Z-disk network during heart development

Src 同源性和胶原蛋白 A (ShcA) 衔接蛋白是心脏发育过程中肋节/Z 盘网络空间组织所必需的

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1074/jbc.M114.597377

Mohamed Mlih, Lionel Host, Sophie Martin, Nathalie Niederhoffer, Laurent Monassier, Jérôme Terrand, Nadia Messaddeq, Michael Radke, Michael Gotthardt, Véronique Bruban, Frank Kober, Monique Bernard, Emmanuelle Canet-Soulas, Francisco Abt-Jijon, Philippe Boucher, Rachel L Matz

心血管

发表日期：2015

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The nuclear hormone receptor PPARγ counteracts vascular calcification by inhibiting Wnt5a signalling in vascular smooth muscle cells

核激素受体PPARγ通过抑制血管平滑肌细胞中的Wnt5a信号通路来对抗血管钙化。

期刊:Nature Communications

影响因子:15.7

doi:10.1038/ncomms2087

Woldt, Estelle; Terrand, Jérome; Mlih, Mohamed; Matz, Rachel L; Bruban, Véronique; Coudane, Fanny; Foppolo, Sophie; El Asmar, Zeina; Chollet, Maria Eugenia; Ninio, Ewa; Bednarczyk, Audrey; Thiersé, Danièle; Schaeffer, Christine; Van Dorsselaer, Alain; Boudier, Christian; Wahli, Walter; Chambon, Pierre; Metzger, Daniel; Herz, Joachim; Boucher, Philippe

Differential signaling by adaptor molecules LRP1 and ShcA regulates adipogenesis by the insulin-like growth factor-1 receptor

衔接分子LRP1和ShcA的差异性信号传导通过胰岛素样生长因子-1受体调节脂肪生成

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M110.212878

Woldt, Estelle; Matz, Rachel L; Terrand, Jérome; Mlih, Mohamed; Gracia, Céline; Foppolo, Sophie; Martin, Sophie; Bruban, Véronique; Ji, Julie; Velot, Emilie; Herz, Joachim; Boucher, Philippe

p21(WAF1/Cip1/Sdi1) knockout mice respond to doxorubicin with reduced cardiotoxicity

p21(WAF1/Cip1/Sdi1)基因敲除小鼠对阿霉素的反应表现为心脏毒性降低

期刊:Toxicology and Applied Pharmacology

影响因子:3.3

doi:10.1016/j.taap.2011.08.024

Jerome Terrand,Beibei Xu, Steve Morrissy, Thai Nho Dinh, Stuart Williams, Qin M Chen

毒理研究

发表日期：2011

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Cystatin C increases in cardiac injury: a role in extracellular matrix protein modulation

胱抑素 C 在心脏损伤中增加：在细胞外基质蛋白调节中的作用

期刊:Cardiovascular Research

影响因子:10.2

doi:10.1093/cvr/cvq138

Lifang Xie, Jerome Terrand, Beibei Xu, George Tsaprailis, Jean Boyer, Qin M Chen

Wnt5a Promotes Lysosomal Cholesterol Egress and Protects Against Atherosclerosis

Wnt5a促进溶酶体胆固醇外流并预防动脉粥样硬化

ShcA promotes chondrocyte hypertrophic commitment and osteoarthritis in mice through RunX2 nuclear translocation and YAP1 inactivation

ShcA 通过 RunX2 核转位和 YAP1 失活促进小鼠软骨细胞肥大和骨关节炎

Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation

内皮细胞中衔接蛋白ShcA的缺失可防止单核细胞与巨噬细胞黏附、低密度脂蛋白氧化以及动脉粥样硬化病变的形成。

Author Correction: Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation

作者更正：内皮细胞中衔接蛋白ShcA的缺失可防止单核细胞与巨噬细胞黏附、低密度脂蛋白氧化以及动脉粥样硬化病变的形成。

Convergent Signaling Pathways Controlled by LRP1 (Receptor-related Protein 1) Cytoplasmic and Extracellular Domains Limit Cellular Cholesterol Accumulation.

由 LRP1（受体相关蛋白 1）胞质和胞外结构域控制的汇聚信号通路限制细胞胆固醇的积累

The Src homology and collagen A (ShcA) adaptor protein is required for the spatial organization of the costamere/Z-disk network during heart development

Src 同源性和胶原蛋白 A (ShcA) 衔接蛋白是心脏发育过程中肋节/Z 盘网络空间组织所必需的

The nuclear hormone receptor PPARγ counteracts vascular calcification by inhibiting Wnt5a signalling in vascular smooth muscle cells

核激素受体PPARγ通过抑制血管平滑肌细胞中的Wnt5a信号通路来对抗血管钙化。

Differential signaling by adaptor molecules LRP1 and ShcA regulates adipogenesis by the insulin-like growth factor-1 receptor

衔接分子LRP1和ShcA的差异性信号传导通过胰岛素样生长因子-1受体调节脂肪生成

p21(WAF1/Cip1/Sdi1) knockout mice respond to doxorubicin with reduced cardiotoxicity

p21(WAF1/Cip1/Sdi1)基因敲除小鼠对阿霉素的反应表现为心脏毒性降低

Cystatin C increases in cardiac injury: a role in extracellular matrix protein modulation

胱抑素 C 在心脏损伤中增加：在细胞外基质蛋白调节中的作用