Than Tin Aung相关文献与解析，生知库 | 链接生命科学的每一步

New insights into the role and mechanism of c-Jun-N-terminal kinase signaling in the pathobiology of liver diseases

c-Jun N端激酶信号通路在肝脏疾病病理生物学中的作用和机制的新见解

期刊:Hepatology

影响因子:15.8

doi:10.1002/hep.29689

Win, Sanda; Than, Tin Aung; Zhang, Jun; Oo, Christina; Min, Robert Win Maw; Kaplowitz, Neil

The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects

JNK信号通路通过与线粒体SAB的相互作用以及上游翻译后效应调控细胞死亡

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms19113657

Win, Sanda; Than, Tin Aung; Kaplowitz, Neil

Antcin H Protects Against Acute Liver Injury Through Disruption of the Interaction of c-Jun-N-Terminal Kinase with Mitochondria

安替辛H通过破坏c-Jun N端激酶与线粒体的相互作用来保护肝脏免受急性损伤。

期刊:Antioxidants & Redox Signaling

影响因子:6.1

doi:10.1089/ars.2016.6833

Huo, Yazhen; Win, Sanda; Than, Tin Aung; Yin, Shutao; Ye, Min; Hu, Hongbo; Kaplowitz, Neil

c-Jun N-terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)-dependent pathway leading to inactivation of intramitochondrial Src

c-Jun N 端激酶通过一种新型的 Sab (SH3BP5) 依赖性通路介导小鼠肝损伤，导致线粒体内 Src 失活。

期刊:Hepatology

影响因子:15.8

doi:10.1002/hep.28486

Win, Sanda; Than, Tin Aung; Min, Robert Win Maw; Aghajan, Mariam; Kaplowitz, Neil

Silencing glycogen synthase kinase-3beta inhibits acetaminophen hepatotoxicity and attenuates JNK activation and loss of glutamate cysteine ligase and myeloid cell leukemia sequence 1.

抑制糖原合成酶激酶-3β可抑制对乙酰氨基酚的肝毒性，并减弱JNK激活和谷氨酸半胱氨酸连接酶的丢失以及髓系细胞白血病序列1

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M109.054999

Shinohara Mie, Ybanez Maria D, Win Sanda, Than Tin Aung, Jain Shilpa, Gaarde William A, Han Derick, Kaplowitz Neil

Effect of transgenic extrahepatic expression of betaine-homocysteine methyltransferase on alcohol or homocysteine-induced fatty liver.

转基因肝外表达甜菜碱-同型半胱氨酸甲基转移酶对酒精或同型半胱氨酸诱导的脂肪肝的影响

期刊:Alcoholism-Clinical and Experimental Research

影响因子:2.7

doi:10.1111/j.1530-0277.2008.00666.x

Ji Cheng, Shinohara Masao, Vance Dennis, Than Tin Aung, Ookhtens Murad, Chan Christine, Kaplowitz Neil

免疫/内分泌

发表日期：2008

文献求助收藏

Contrasting genome-wide distribution of 8-hydroxyguanine and acrolein-modified adenine during oxidative stress-induced renal carcinogenesis

氧化应激诱导的肾脏致癌过程中 8-羟基鸟嘌呤和丙烯醛修饰腺嘌呤的全基因组分布对比

期刊:American Journal of Pathology

影响因子:4.7

doi:10.2353/ajpath.2006.051280

Shinya Akatsuka, Than Tin Aung, Khokon Kumar Dutta, Li Jiang, Wen-Hua Lee, Yu-Ting Liu, Janice Onuki, Tomoyuki Shirase, Kyoko Yamasaki, Hirotomo Ochi, Yuji Naito, Toshikazu Yoshikawa, Hiroshi Kasai, Yohei Tominaga, Kunihiko Sakumi, Yusaku Nakabeppu, Yoshichika Kawai, Koji Uchida, Aiichi Yamasaki, Ta

Mitochondrial P-JNK target, SAB (SH3BP5), in regulation of cell death

线粒体P-JNK靶点SAB（SH3BP5）在细胞死亡调控中的作用

New insights into the role and mechanism of c-Jun-N-terminal kinase signaling in the pathobiology of liver diseases

c-Jun N端激酶信号通路在肝脏疾病病理生物学中的作用和机制的新见解

The Regulation of JNK Signaling Pathways in Cell Death through the Interplay with Mitochondrial SAB and Upstream Post-Translational Effects

JNK信号通路通过与线粒体SAB的相互作用以及上游翻译后效应调控细胞死亡

Antcin H Protects Against Acute Liver Injury Through Disruption of the Interaction of c-Jun-N-Terminal Kinase with Mitochondria

安替辛H通过破坏c-Jun N端激酶与线粒体的相互作用来保护肝脏免受急性损伤。

c-Jun N-terminal kinase mediates mouse liver injury through a novel Sab (SH3BP5)-dependent pathway leading to inactivation of intramitochondrial Src

c-Jun N 端激酶通过一种新型的 Sab (SH3BP5) 依赖性通路介导小鼠肝损伤，导致线粒体内 Src 失活。

Silencing glycogen synthase kinase-3beta inhibits acetaminophen hepatotoxicity and attenuates JNK activation and loss of glutamate cysteine ligase and myeloid cell leukemia sequence 1.

抑制糖原合成酶激酶-3β可抑制对乙酰氨基酚的肝毒性，并减弱JNK激活和谷氨酸半胱氨酸连接酶的丢失以及髓系细胞白血病序列1

Effect of transgenic extrahepatic expression of betaine-homocysteine methyltransferase on alcohol or homocysteine-induced fatty liver.

转基因肝外表达甜菜碱-同型半胱氨酸甲基转移酶对酒精或同型半胱氨酸诱导的脂肪肝的影响

Contrasting genome-wide distribution of 8-hydroxyguanine and acrolein-modified adenine during oxidative stress-induced renal carcinogenesis

氧化应激诱导的肾脏致癌过程中 8-羟基鸟嘌呤和丙烯醛修饰腺嘌呤的全基因组分布对比