Thomas Dechat相关文献与解析，生知库 | 链接生命科学的每一步

Nana Naetar #, Konstantina Georgiou #, Christian Knapp, Irena Bronshtein, Elisabeth Zier, Petra Fichtinger, Thomas Dechat, Yuval Garini, Roland Foisner

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IHC-P

发表日期：2021

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Nucleoplasmic lamins define growth-regulating functions of lamina-associated polypeptide 2α in progeria cells

核质层蛋白决定层蛋白相关多肽 2α 在早衰细胞中的生长调节功能

期刊:Journal of Cell Science

影响因子:3.3

doi:10.1242/jcs.208462

Sandra Vidak, Konstantina Georgiou, Petra Fichtinger, Nana Naetar, Thomas Dechat, Roland Foisner

A-type lamins bind both hetero- and euchromatin, the latter being regulated by lamina-associated polypeptide 2 alpha

型层蛋白可与异染色质和真染色质结合，后者受层蛋白相关多肽 2α 调控

期刊:Genome Research

影响因子:6.2

doi:10.1101/gr.196220.115

Kevin Gesson, Philipp Rescheneder, Michael P Skoruppa, Arndt von Haeseler, Thomas Dechat, Roland Foisner

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发表日期：2016

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Proliferation of progeria cells is enhanced by lamina-associated polypeptide 2α (LAP2α) through expression of extracellular matrix proteins

层相关多肽 2α (LAP2α) 通过表达细胞外基质蛋白来增强早衰细胞的增殖

期刊:Genes & Development

影响因子:1.9

doi:10.1101/gad.263939.115

Sandra Vidak, Nard Kubben, Thomas Dechat, Roland Foisner

The muscle dystrophy-causing ΔK32 lamin A/C mutant does not impair the functions of the nucleoplasmic lamin-A/C-LAP2α complex in mice

引起肌肉营养不良的 ΔK32 层蛋白 A/C 突变体不会损害小鼠核质层蛋白-A/C-LAP2α 复合物的功能

期刊:Journal of Cell Science

影响因子:3.3

doi:10.1242/jcs.115246

Ursula Pilat, Thomas Dechat, Anne T Bertrand, Nikola Woisetschläger, Ivana Gotic, Rita Spilka, Katarzyna Biadasiewicz, Gisèle Bonne, Roland Foisner

Alterations in mitosis and cell cycle progression caused by a mutant lamin A known to accelerate human aging

已知会加速人类衰老的突变层蛋白 A 导致有丝分裂和细胞周期进程发生改变

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.0700854104

Thomas Dechat, Takeshi Shimi, Stephen A Adam, Antonio E Rusinol, Douglas A Andres, H Peter Spielmann, Michael S Sinensky, Robert D Goldman

Lamin A/C binding protein LAP2alpha is required for nuclear anchorage of retinoblastoma protein

层蛋白 A/C 结合蛋白 LAP2alpha 是视网膜母细胞瘤蛋白核锚定所必需的

期刊:Molecular Biology of the Cell

影响因子:3.1

doi:10.1091/mbc.e02-07-0450

Ewa Markiewicz, Thomas Dechat, Roland Foisner, Roy A Quinlan, Christopher J Hutchison

Twisted-plywood-like tissue formation in vitro. Does curvature do the twist?

体外扭曲胶合板状组织形成。曲率会导致扭曲吗？

LAP2alpha maintains a mobile and low assembly state of A-type lamins in the nuclear interior

LAP2alpha 维持核内部 A 型层蛋白的移动和低组装状态

Nucleoplasmic lamins define growth-regulating functions of lamina-associated polypeptide 2α in progeria cells

核质层蛋白决定层蛋白相关多肽 2α 在早衰细胞中的生长调节功能

A-type lamins bind both hetero- and euchromatin, the latter being regulated by lamina-associated polypeptide 2 alpha

型层蛋白可与异染色质和真染色质结合，后者受层蛋白相关多肽 2α 调控

Proliferation of progeria cells is enhanced by lamina-associated polypeptide 2α (LAP2α) through expression of extracellular matrix proteins

层相关多肽 2α (LAP2α) 通过表达细胞外基质蛋白来增强早衰细胞的增殖

The muscle dystrophy-causing ΔK32 lamin A/C mutant does not impair the functions of the nucleoplasmic lamin-A/C-LAP2α complex in mice

引起肌肉营养不良的 ΔK32 层蛋白 A/C 突变体不会损害小鼠核质层蛋白-A/C-LAP2α 复合物的功能

Alterations in mitosis and cell cycle progression caused by a mutant lamin A known to accelerate human aging

已知会加速人类衰老的突变层蛋白 A 导致有丝分裂和细胞周期进程发生改变

Lamin A/C binding protein LAP2alpha is required for nuclear anchorage of retinoblastoma protein

层蛋白 A/C 结合蛋白 LAP2alpha 是视网膜母细胞瘤蛋白核锚定所必需的