日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Steatosis, inflammasome upregulation, and fibrosis are attenuated in miR-155 deficient mice in a high fat-cholesterol-sugar diet-induced model of NASH

在高脂肪-胆固醇-糖饮食诱发的 NASH 模型中,缺乏 miR-155 的小鼠的脂肪变性、炎症小体上调和纤维化有所减弱

Shashi Bala #, Michal Ganz #, Mrigya Babuta, Yuan Zhuang, Timea Csak, Charles D Calenda, Gyongyi Szabo

Alcohol-induced miR-155 and HDAC11 inhibit negative regulators of the TLR4 pathway and lead to increased LPS responsiveness of Kupffer cells in alcoholic liver disease

酒精诱导的 miR-155 和 HDAC11 抑制 TLR4 通路的负调节因子,导致酒精性肝病中库普弗细胞的 LPS 反应性增强

Shashi Bala, Timea Csak, Karen Kodys, Donna Catalano, Aditya Ambade, Istvan Furi, Patrick Lowe, Yeonhee Cho, Arvin Iracheta-Vellve, Gyongyi Szabo

Progression of non-alcoholic steatosis to steatohepatitis and fibrosis parallels cumulative accumulation of danger signals that promote inflammation and liver tumors in a high fat-cholesterol-sugar diet model in mice

在小鼠高脂肪-胆固醇-糖饮食模型中,非酒精性脂肪变性进展为脂肪性肝炎和纤维化,与促进炎症和肝脏肿瘤的危险信号的累积同时发生

Michal Ganz, Terence N Bukong, Timea Csak, Banishree Saha, Jin-Kyu Park, Aditya Ambade, Karen Kodys, Gyongyi Szabo

Biodistribution and function of extracellular miRNA-155 in mice

小鼠细胞外 miRNA-155 的生物分布和功能

Shashi Bala, Timea Csak, Fatemeh Momen-Heravi, Dora Lippai, Karen Kodys, Donna Catalano, Abhishek Satishchandran, Victor Ambros, Gyongyi Szabo

MicroRNA-155 Deficiency Attenuates Liver Steatosis and Fibrosis without Reducing Inflammation in a Mouse Model of Steatohepatitis

MicroRNA-155 缺乏可减轻脂肪肝小鼠模型中的肝脏脂肪变性和纤维化,但不会减轻炎症

Timea Csak, Shashi Bala, Dora Lippai, Karen Kodys, Donna Catalano, Arvin Iracheta-Vellve, Gyongyi Szabo

Mitochondrial antiviral signaling protein defect links impaired antiviral response and liver injury in steatohepatitis in mice

线粒体抗病毒信号蛋白缺陷与小鼠脂肪性肝炎抗病毒反应受损和肝损伤有关

Timea Csak, Angela Dolganiuc, Karen Kodys, Bharath Nath, Jan Petrasek, Shashi Bala, Dora Lippai, Gyongyi Szabo

Up-regulation of microRNA-155 in macrophages contributes to increased tumor necrosis factor {alpha} (TNF{alpha}) production via increased mRNA half-life in alcoholic liver disease

巨噬细胞中 microRNA-155 的上调通过增加酒精性肝病中的 mRNA 半衰期导致肿瘤坏死因子 α (TNF α) 的产生增加

Shashi Bala, Miguel Marcos, Karen Kodys, Timea Csak, Donna Catalano, Pranoti Mandrekar, Gyongyi Szabo