Torii Tomohiro相关文献与解析，生知库 | 链接生命科学的每一步

Graded regulation of microtubule-binding of Tau by the phosphorylation state of the proline-rich region in living neurons

活神经元中富含脯氨酸区域的磷酸化状态对Tau蛋白微管结合的梯度调控

期刊:

影响因子:

doi:10.1101/2025.11.11.687775

Nakata, Rinaho; Torii, Tomohiro; Miyasaka, Tomohiro; Misonou, Hiroaki

Abnormal expression of Tau in damaged oligodendrocytes of HLD1 mice

HLD1小鼠受损少突胶质细胞中Tau蛋白的异常表达

期刊:Neural Regeneration Research

影响因子:6.7

doi:10.4103/1673-5374.387983

Molecular Pathogenic Mechanisms of Hypomyelinating Leukodystrophies (HLDs)

髓鞘形成不足性脑白质营养不良症（HLDs）的分子致病机制

期刊:Neurology International

影响因子:3

doi:10.3390/neurolint15030072

Torii, Tomohiro; Yamauchi, Junji

神经科学

发表日期：2023

文献求助收藏

FTD/ALS Type 7-Associated Thr104Asn Mutation of CHMP2B Blunts Neuronal Process Elongation, and Is Recovered by Knockdown of Arf4, the Golgi Stress Regulator.

FTD/ALS 7 型相关 CHMP2B Thr104Asn 突变会抑制神经元突起的延伸，而敲低高尔基体应激调节因子 Arf4 可以恢复这种抑制作用

期刊:Neurology International

影响因子:3

doi:10.3390/neurolint15030063

Shirai Remina, Cho Mizuka, Isogai Mikinori, Fukatsu Shoya, Okabe Miyu, Okawa Maho, Miyamoto Yuki, Torii Tomohiro, Yamauchi Junji

The organization of microtubules and Tau in oligodendrocytes: Tau pathology in damaged oligodendrocytes

少突胶质细胞中微管和Tau蛋白的组织结构：受损少突胶质细胞中的Tau蛋白病理

期刊:Frontiers in Cell and Developmental Biology

影响因子:4.3

doi:10.3389/fcell.2022.950682

Torii, Tomohiro; Miyasaka, Tomohiro; Misonou, Hiroaki

Data on the effects of Charcot-Marie-Tooth disease type 2N-associated AARS missense mutation (Arg329-to-His) on the cell biological properties

关于夏科-马里-图斯病2N型相关AARS错义突变（Arg329-to-His）对细胞生物学特性的影响数据

期刊:Data in Brief

影响因子:1.4

doi:10.1016/j.dib.2019.104029

Imaizumi, Naoko; Takeuchi, Yu; Hirano, Haruka; Torii, Tomohiro; Seki, Yoichi; Morimoto, Takako; Miyamoto, Yuki; Sakagami, Hiroyuki; Yamauchi, Junji

细胞生物学

发表日期：2019

文献求助收藏

The promoter region of 46-kDa CNPase is sufficient for its expression in corpus callosum

46 kDa CNPase的启动子区域足以使其在胼胝体中表达。

期刊:Molecular Genetics and Metabolism Reports

影响因子:1.9

doi:10.1016/j.ymgmr.2018.03.003

Miyamoto, Yuki; Torii, Tomohiro; Tanoue, Akito; Yamamoto, Masahiro; Yamauchi, Junji

发表日期：2018

文献求助收藏

Data on the effect of knockout of neruregulin-1 type III on Remak bundle structure

敲除神经调节蛋白-1 III 型对雷马克束结构的影响数据

期刊:Data in Brief

影响因子:1.4

doi:10.1016/j.dib.2018.03.099

Miyamoto, Yuki; Torii, Tomohiro; Inoue, Masashi; Morimoto, Takako; Yamamoto, Masahiro; Yamauchi, Junji

发表日期：2018

文献求助收藏

Paradoxical gain-of-function mutant of the G-protein-coupled receptor PROKR2 promotes early puberty

G蛋白偶联受体PROKR2的矛盾性功能获得性突变促进青春期提前

期刊:Journal of Cellular and Molecular Medicine

影响因子:4.2

doi:10.1111/jcmm.13146

Fukami, Maki; Suzuki, Erina; Izumi, Yoko; Torii, Tomohiro; Narumi, Satoshi; Igarashi, Maki; Miyado, Mami; Katsumi, Momori; Fujisawa, Yasuko; Nakabayashi, Kazuhiko; Hata, Kenichiro; Umezawa, Akihiro; Matsubara, Yoichi; Yamauchi, Junji; Ogata, Tsutomu

信号转导

发表日期：2017

文献求助收藏

Septins in the nervous system: from cytoskeletal dynamics to neurological disorders

神经系统中的隔蛋白：从细胞骨架动力学到神经系统疾病

Graded regulation of microtubule-binding of Tau by the phosphorylation state of the proline-rich region in living neurons

活神经元中富含脯氨酸区域的磷酸化状态对Tau蛋白微管结合的梯度调控

Abnormal expression of Tau in damaged oligodendrocytes of HLD1 mice

HLD1小鼠受损少突胶质细胞中Tau蛋白的异常表达

Molecular Pathogenic Mechanisms of Hypomyelinating Leukodystrophies (HLDs)

髓鞘形成不足性脑白质营养不良症（HLDs）的分子致病机制

FTD/ALS Type 7-Associated Thr104Asn Mutation of CHMP2B Blunts Neuronal Process Elongation, and Is Recovered by Knockdown of Arf4, the Golgi Stress Regulator.

FTD/ALS 7 型相关 CHMP2B Thr104Asn 突变会抑制神经元突起的延伸，而敲低高尔基体应激调节因子 Arf4 可以恢复这种抑制作用

The organization of microtubules and Tau in oligodendrocytes: Tau pathology in damaged oligodendrocytes

少突胶质细胞中微管和Tau蛋白的组织结构：受损少突胶质细胞中的Tau蛋白病理

Data on the effects of Charcot-Marie-Tooth disease type 2N-associated AARS missense mutation (Arg329-to-His) on the cell biological properties

关于夏科-马里-图斯病2N型相关AARS错义突变（Arg329-to-His）对细胞生物学特性的影响数据

The promoter region of 46-kDa CNPase is sufficient for its expression in corpus callosum

46 kDa CNPase的启动子区域足以使其在胼胝体中表达。

Data on the effect of knockout of neruregulin-1 type III on Remak bundle structure

敲除神经调节蛋白-1 III 型对雷马克束结构的影响数据

Paradoxical gain-of-function mutant of the G-protein-coupled receptor PROKR2 promotes early puberty

G蛋白偶联受体PROKR2的矛盾性功能获得性突变促进青春期提前