Trottier Yvon相关文献与解析，生知库 | 链接生命科学的每一步

Surdyka Magdalena, Kalinowska-PoÅka Å»aneta, Niewiadomska-Cimicka Anna, Jesion Ewelina, Fiszer Agnieszka, Singer-Mikosch Elisabeth, Fievet Lorraine, Przybyl Lukasz, Caron Nicholas S, Hayden Michael R, Nguyen Huu Phuc, Trottier Yvon, Figiel Maciej

其它

Human

Mouse

发表日期：2025

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Longitudinal MRI and 1H-MRS study of SCA7 mouse forebrain reveals progressive multiregional atrophy and early brain metabolite changes indicating early neuronal and glial dysfunction

对SCA7小鼠前脑的纵向MRI和1H-MRS研究揭示了进行性多区域萎缩和早期脑代谢物变化，表明早期神经元和胶质细胞功能障碍。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0296790

Pérot, Jean-Baptiste; Niewiadomska-Cimicka, Anna; Brouillet, Emmanuel; Trottier, Yvon; Flament, Julien

Selective transduction of cerebellar Purkinje and granule neurons using delivery of AAV-PHP.eB and AAVrh10 vectors at axonal terminal locations

利用AAV-PHP.eB和AAVrh10载体在轴突末端进行选择性转导，从而实现对小脑浦肯野神经元和颗粒神经元的靶向转导。

期刊:Frontiers in Molecular Neuroscience

影响因子:3.8

doi:10.3389/fnmol.2022.947490

Surdyka, Magdalena; Jesion, Ewelina; Niewiadomska-Cimicka, Anna; Trottier, Yvon; Kalinowska-Pośka, Żaneta; Figiel, Maciej

Gene Deregulation and Underlying Mechanisms in Spinocerebellar Ataxias With Polyglutamine Expansion

多聚谷氨酰胺扩增引起的脊髓小脑性共济失调的基因失调及其潜在机制

期刊:Frontiers in Neuroscience

影响因子:3.2

doi:10.3389/fnins.2020.00571

Niewiadomska-Cimicka, Anna; Hache, Antoine; Trottier, Yvon

神经科学

发表日期：2020

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Molecular Targets and Therapeutic Strategies in Spinocerebellar Ataxia Type 7

脊髓小脑性共济失调7型的分子靶点和治疗策略

期刊:Neurotherapeutics

影响因子:6.9

doi:10.1007/s13311-019-00778-5

Niewiadomska-Cimicka, Anna; Trottier, Yvon

神经科学

发表日期：2019

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Microbiological contamination in counterfeit and unapproved drugs

假冒和未经批准药物中的微生物污染

期刊:

影响因子:

doi:10.1186/2050-6511-15-34

Pullirsch, Dieter; Bellemare, Julie; Hackl, Andreas; Trottier, Yvon-Louis; Mayrhofer, Andreas; Schindl, Heidemarie; Taillon, Christine; Gartner, Christian; Hottowy, Brigitte; Beck, Gerhard; Gagnon, Jacques

微生物学

发表日期：2014

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Preventing polyglutamine-induced activation of c-Jun delays neuronal dysfunction in a mouse model of SCA7 retinopathy

抑制多聚谷氨酰胺诱导的c-Jun激活可延缓SCA7视网膜病变小鼠模型中的神经元功能障碍。

期刊:Neurobiology of Disease

影响因子:5.6

doi:10.1016/j.nbd.2006.11.002

Merienne, Karine; Friedman, James; Akimoto, Masayuki; Abou-Sleymane, Gretta; Weber, Chantal; Swaroop, Anand; Trottier, Yvon

CAG-targeted brain-permeable therapy tested in biallelic humanized polyQ mouse models.

在双等位基因人源化polyQ小鼠模型中测试了CAG靶向脑渗透性疗法

Longitudinal MRI and 1H-MRS study of SCA7 mouse forebrain reveals progressive multiregional atrophy and early brain metabolite changes indicating early neuronal and glial dysfunction

对SCA7小鼠前脑的纵向MRI和1H-MRS研究揭示了进行性多区域萎缩和早期脑代谢物变化，表明早期神经元和胶质细胞功能障碍。

Selective transduction of cerebellar Purkinje and granule neurons using delivery of AAV-PHP.eB and AAVrh10 vectors at axonal terminal locations

利用AAV-PHP.eB和AAVrh10载体在轴突末端进行选择性转导，从而实现对小脑浦肯野神经元和颗粒神经元的靶向转导。

Gene Deregulation and Underlying Mechanisms in Spinocerebellar Ataxias With Polyglutamine Expansion

多聚谷氨酰胺扩增引起的脊髓小脑性共济失调的基因失调及其潜在机制

Molecular Targets and Therapeutic Strategies in Spinocerebellar Ataxia Type 7

脊髓小脑性共济失调7型的分子靶点和治疗策略

Microbiological contamination in counterfeit and unapproved drugs

假冒和未经批准药物中的微生物污染

Preventing polyglutamine-induced activation of c-Jun delays neuronal dysfunction in a mouse model of SCA7 retinopathy

抑制多聚谷氨酰胺诱导的c-Jun激活可延缓SCA7视网膜病变小鼠模型中的神经元功能障碍。