日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Residual Breast Cancer Cells Co-opt SOX5-Driven Endochondral Ossification to Maintain Dormancy

残留乳腺癌细胞利用SOX5驱动的软骨内成骨作用维持休眠状态

Sreekumar, Amulya; Blankemeyer, Eric; Sterner, Christopher J; Pan, Tien-Chi; Pant, Dhruv K; Acolatse, Sarah; Turkistani, Hamza; Belka, George K; Nayak, Anupma; Carlin, Sean D; Assenmacher, Charles-Antoine; Sellmyer, Mark A; Mankoff, David A; Chodosh, Lewis A

Engineered immunological niche directs therapeutic development in models of progressive multiple sclerosis.

工程化免疫微环境指导进行性多发性硬化症模型中的治疗开发

Rad Laila M, Hughes Kevin R, Wheeler Sydney N, Decker Joseph T, Orbach Sophia M, Galvan Angelica, Thornhill Jasmine, Griffin Kate V, Turkistani Hamza, Urie Russell R, Irani David N, Shea Lonnie D, Morris Aaron H

Residual Breast Cancer Cells Co-opt SOX5-driven Endochondral Ossification to Maintain Dormancy.

残留的乳腺癌细胞利用 SOX5 驱动的软骨内成骨作用来维持休眠状态

Sreekumar Amulya, Blankemeyer Eric, Sterner Christopher J, Pan Tien-Chi, Pant Dhruv K, Acolatse Sarah, Turkistani Hamza, Belka George K, Carlin Sean D, Assenmacher Charles A, Sellmyer Mark A, Mankoff David A, Chodosh Lewis A