Tykocki Nathan R相关文献与解析，生知库 | 链接生命科学的每一步

Quantifying whole bladder biomechanics using the novel pentaplanar reflected image macroscopy system

利用新型五平面反射图像宏观系统量化整个膀胱的生物力学特性

期刊:Biomechanics and Modeling in Mechanobiology

影响因子:2.7

doi:10.1007/s10237-023-01727-0

Hennig, Grant; Saxena, Pragya; Broemer, Eli; Herrera, Gerald M; Roccabianca, Sara; Tykocki, Nathan R

发表日期：2023

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The mast cell stimulator compound 48/80 causes urothelium-dependent increases in murine urinary bladder contractility

肥大细胞刺激剂化合物 48/80 可引起小鼠膀胱收缩力的尿路上皮依赖性增强。

期刊:

影响因子:

doi:10.1152/ajprenal.00116.2023

Jones, B Malique; Mingin, Gerald C; Tykocki, Nathan R

Remodeling of extracellular matrix in the urinary bladder of paraplegic rats results in increased compliance and delayed fiber recruitment 16 weeks after spinal cord injury

截瘫大鼠膀胱细胞外基质的重塑导致顺应性增加，并在脊髓损伤后16周出现纤维募集延迟。

期刊:Acta Biomaterialia

影响因子:9.6

doi:10.1016/j.actbio.2022.01.015

Tuttle, Tyler G; Lujan, Heidi L; Tykocki, Nathan R; DiCarlo, Stephen E; Roccabianca, Sara

Histamine receptors rapidly desensitize without altering nerve-evoked contractions in murine urinary bladder smooth muscle

组胺受体在不改变小鼠膀胱平滑肌神经诱发收缩的情况下迅速脱敏。

期刊:

影响因子:

doi:10.1152/ajprenal.00355.2021

Jones, B Malique; Mingin, Gerald C; Tykocki, Nathan R

信号转导

发表日期：2022

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Excitability and contractility in arterioles and venules from the urinary bladder

膀胱小动脉和小静脉的兴奋性和收缩性

期刊:

影响因子:

doi:10.1016/bs.ctm.2020.01.003

Tykocki, Nathan R; Monson, Frederick C

发表日期：2020

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Development of stress-induced bladder insufficiency requires functional TRPV1 channels

应激性膀胱功能不全的发生需要功能性TRPV1通道。

期刊:

影响因子:

doi:10.1152/ajprenal.00231.2018

Tykocki, Nathan R; Heppner, Thomas J; Erickson, Cuixia Shi; van Batavia, Jason; Vizzard, Margaret A; Nelson, Mark T; Mingin, Gerald C

TRPV1

发表日期：2018

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Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles

平滑肌离子通道与阻力动脉和小动脉血管张力的调节

期刊:Comprehensive Physiology

影响因子:5.2

doi:10.1002/cphy.c160011

Tykocki, Nathan R; Boerman, Erika M; Jackson, William F

发表日期：2017

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Inhibition of vascular smooth muscle inward-rectifier K(+) channels restores myogenic tone in mouse urinary bladder arterioles

抑制血管平滑肌内向整流钾离子通道可恢复小鼠膀胱小动脉的肌源性张力

期刊:

影响因子:

doi:10.1152/ajprenal.00682.2016

Tykocki, Nathan R; Bonev, Adrian D; Longden, Thomas A; Heppner, Thomas J; Nelson, Mark T

发表日期：2017

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Compound 48/80 increases bladder compliance by activating MMP-2 and inhibiting TIMP-2.

化合物 48/80 通过激活 MMP-2 和抑制 TIMP-2 来增加膀胱顺应性

Modeling bladder mechanics with 4D reconstruction of murine ex vivo bladder filling

利用小鼠离体膀胱充盈的4D重建模型模拟膀胱力学

Quantifying whole bladder biomechanics using the novel pentaplanar reflected image macroscopy system

利用新型五平面反射图像宏观系统量化整个膀胱的生物力学特性

The mast cell stimulator compound 48/80 causes urothelium-dependent increases in murine urinary bladder contractility

肥大细胞刺激剂化合物 48/80 可引起小鼠膀胱收缩力的尿路上皮依赖性增强。

Remodeling of extracellular matrix in the urinary bladder of paraplegic rats results in increased compliance and delayed fiber recruitment 16 weeks after spinal cord injury

截瘫大鼠膀胱细胞外基质的重塑导致顺应性增加，并在脊髓损伤后16周出现纤维募集延迟。

Histamine receptors rapidly desensitize without altering nerve-evoked contractions in murine urinary bladder smooth muscle

组胺受体在不改变小鼠膀胱平滑肌神经诱发收缩的情况下迅速脱敏。

Excitability and contractility in arterioles and venules from the urinary bladder

膀胱小动脉和小静脉的兴奋性和收缩性

Development of stress-induced bladder insufficiency requires functional TRPV1 channels

应激性膀胱功能不全的发生需要功能性TRPV1通道。

Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles

平滑肌离子通道与阻力动脉和小动脉血管张力的调节

Inhibition of vascular smooth muscle inward-rectifier K(+) channels restores myogenic tone in mouse urinary bladder arterioles

抑制血管平滑肌内向整流钾离子通道可恢复小鼠膀胱小动脉的肌源性张力