FancJ (Brip1) loss-of-function allele results in spermatogonial cell depletion during embryogenesis and altered processing of crossover sites during meiotic prophase I in mice

FancJ（Brip1）功能丧失等位基因导致小鼠胚胎发生过程中精原细胞耗竭，并在减数分裂前期 I 期间改变交叉位点的处理