Ulmann Lauriane相关文献与解析，生知库 | 链接生命科学的每一步

Er-Lukowiak, Marco; Duan, Yinghui; Rassendren, Francois; Ulmann, Lauriane; Nicke, Annette; Ufer, Friederike; Friese, Manuel A; Koch-Nolte, Friedrich; Magnus, Tim; Rissiek, Björn

细胞生物学

T细胞

免疫/内分泌

发表日期：2020

文献求助收藏

Blocking Î±(2)Î´-1 Subunit Reduces Bladder Hypersensitivity and Inflammation in a Cystitis Mouse Model by Decreasing NF-kB Pathway Activation.

阻断 α(2)α´-1 亚基可降低膀胱炎小鼠模型中的膀胱过敏和炎症，其机制是通过降低 NF-kB 通路的激活

期刊:Frontiers in Pharmacology

影响因子:4.8

doi:10.3389/fphar.2019.00133

Boudieu Ludivine, Mountadem Sarah, Lashermes Amandine, Meleine Mathieu, Ulmann Lauriane, Rassendren FranÃ§ois, Aissouni Youssef, Sion Benoit, Carvalho FrÃ©dÃ©ric Antonio, Ardid Denis

Inflammatory-induced spinal dorsal horn neurons hyperexcitability is mediated by P2X4 receptors

炎症诱导的脊髓背角神经元过度兴奋是由P2X4受体介导的。

期刊:Pain Reports

影响因子:3.1

doi:10.1097/PR9.0000000000000660

Aby, Franck; Whitestone, Sara; Landry, Marc; Ulmann, Lauriane; Fossat, Pascal

Status epilepticus induces a particular microglial activation state characterized by enhanced purinergic signaling

癫痫持续状态会诱发一种特殊的微胶质细胞活化状态，其特征是嘌呤能信号增强。

期刊:Journal of Neuroscience

影响因子:4

doi:10.1523/JNEUROSCI.1820-08.2008

Avignone, Elena; Ulmann, Lauriane; Levavasseur, Françoise; Rassendren, François; Audinat, Etienne

Limited contribution of the of P2X4 receptor to LPS-induced microglial reaction in mice

P2X4受体对小鼠LPS诱导的小胶质细胞反应的贡献有限

Bone phenotype of P2X4 receptor knockout mice: implication of a P2X7 receptor mutation?

P2X4受体敲除小鼠的骨骼表型：是否暗示存在P2X7受体突变？

A P2rx7 Passenger Mutation Affects the Vitality and Function of T cells in Congenic Mice

P2rx7乘客突变影响同源小鼠T细胞的活力和功能

Blocking Î±(2)Î´-1 Subunit Reduces Bladder Hypersensitivity and Inflammation in a Cystitis Mouse Model by Decreasing NF-kB Pathway Activation.

阻断 α(2)α´-1 亚基可降低膀胱炎小鼠模型中的膀胱过敏和炎症，其机制是通过降低 NF-kB 通路的激活

Inflammatory-induced spinal dorsal horn neurons hyperexcitability is mediated by P2X4 receptors

炎症诱导的脊髓背角神经元过度兴奋是由P2X4受体介导的。

Status epilepticus induces a particular microglial activation state characterized by enhanced purinergic signaling

癫痫持续状态会诱发一种特殊的微胶质细胞活化状态，其特征是嘌呤能信号增强。