日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

S100A8/9-NLRP3-mediated chronic unresolved inflammation drives cardiac pathologies following invasive pneumococcal disease

S100A8/9-NLRP3介导的慢性未消退炎症驱动侵袭性肺炎球菌疾病后的心脏病变

Tousif, Sultan; Minassian, Daniel; He, Chao; Singh, Baldeep; Umbarkar, Prachi; Bhati, Arvind Singh; Mohasin, Mohammed; Erdmann, Nathan; Xie, Min; Sethu, Palaniappan; Orihuela, Carlos J; Lal, Hind

Fibroblast-specific MyD88-dependent signaling aggravates inflammation and cardiac dysfunction in the MI heart.

成纤维细胞特异性 MyD88 依赖性信号传导会加剧心肌梗死心脏的炎症和心脏功能障碍

Umbarkar Prachi, Tousif Sultan, Jaiswal Ashish, Bhati Arvind Singh, Toro Cora Angelica, Sethi Rohan, Zhang Qinkun, Lal Hind

Fibroblast-Specific Depletion of Human Antigen R Alleviates Myocardial Fibrosis Induced by Cardiac Stress

成纤维细胞特异性地消除人类抗原 R 可减轻心脏应激引起的心肌纤维化

Mallikarjun Patil, Sarojini Singh, Praveen Kumar Dubey, Sultan Tousif, Prachi Umbarkar, Qinkun Zhang, Hind Lal, Mary Kathryn Sewell-Loftin, Channakeshava Sokke Umeshappa, Yohannes T Ghebre, Steven Pogwizd, Jianyi Zhang, Prasanna Krishnamurthy

The Shared Airway: Tracheal Mass Excision

共享气道:气管肿块切除术

Shringarpure, Amruta; Saba, Surendhar; Umbarkar, Sanjeeta; Sarkar, Manjula

Diagnosis and management of residual atrial septal defect after surgical failure: A case series

手术失败后残余房间隔缺损的诊断和处理:病例系列研究

Sharma, Satyavan; Shah, Akash Bhupesh; Machigar, Shankar Y; Umbarkar, Rajendra

Animal models of haploinsufficiency revealed the isoform-specific role of GSK-3 in HFD-induced obesity and glucose intolerance

单倍体功能不全动物模型揭示了 GSK-3 在高脂饮食诱导的肥胖和葡萄糖耐受不良中的亚型特异性作用

Gupte, Manisha; Umbarkar, Prachi; Lemon, Jacob; Tousif, Sultan; Lal, Hind

Ponatinib Drives Cardiotoxicity by S100A8/A9-NLRP3-IL-1β Mediated Inflammation

帕纳替尼通过S100A8/A9-NLRP3-IL-1β介导的炎症途径驱动心脏毒性

Tousif, Sultan; Singh, Anand P; Umbarkar, Prachi; Galindo, Cristi; Wheeler, Nicholas; Toro Cora, Angelica; Zhang, Qinkun; Prabhu, Sumanth D; Lal, Hind

Cardiac fibroblast GSK-3α aggravates ischemic cardiac injury by promoting fibrosis, inflammation, and impairing angiogenesis

心脏成纤维细胞 GSK-3α 通过促进纤维化、炎症和损害血管生成来加重缺血性心脏损伤

Prachi Umbarkar, Suma Ejantkar, Sulivette Y Ruiz Ramirez, Angelica Toro Cora, Qinkun Zhang, Sultan Tousif, Hind Lal

Cardiac fibroblast GSK-3α aggravates ischemic cardiac injury by promoting fibrosis, inflammation, and impairing angiogenesis

心脏成纤维细胞 GSK-3α 通过促进纤维化、炎症和抑制血管生成,加重缺血性心脏损伤。

Prachi Umbarkar,Suma Ejantkar,Sulivette Y Ruiz Ramirez,Angelica Toro Cora,Qinkun Zhang,Sultan Tousif,Hind Lal

Sustained Increases in Cardiomyocyte Protein O-Linked β-N-Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment

心肌细胞蛋白 O-连接 β-N-乙酰葡萄糖胺水平持续升高导致心脏肥大和线粒体功能下降,但不会出现收缩功能障碍

Chae-Myeong Ha, Sayan Bakshi, Manoja K Brahma, Luke A Potter, Samuel F Chang, Zhihuan Sun, Gloria A Benavides, Lihao He, Prachi Umbarkar, Luyun Zou, Samuel Curfman, Sini Sunny, Andrew J Paterson, Namakkal-Soorappan Rajasekaran, Jarrod W Barnes, Jianhua Zhang, Hind Lal, Min Xie, Victor M Darley-Usmar