日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Position statement: The need for EU legislation to require disclosure and labelling of the composition of medical devices

立场声明:欧盟立法需要要求披露和标注医疗器械的成分

Herman, A; Uter, W; Rustemeyer, T; Matura, M; Aalto-Korte, K; Duus Johansen, J; Gonçalo, M; White, I R; Balato, A; Giménez Arnau, A M; Brockow, K; Mortz, C G; Mahler, V; Goossens, A

A siRNA screen reveals the prosurvival effect of protein kinase A activation in conditions of unresolved endoplasmic reticulum stress

siRNA 筛选揭示了在未解决的内质网应激条件下蛋白激酶 A 激活的促生存作用

M A Aguileta, D Rojas-Rivera, V Goossens, Y Estornes, G Van Isterdael, P Vandenabeele, M J M Bertrand

Depletion of RIPK3 or MLKL blocks TNF-driven necroptosis and switches towards a delayed RIPK1 kinase-dependent apoptosis

RIPK3 或 MLKL 的消耗会阻止 TNF 驱动的坏死性凋亡,并转向延迟的 RIPK1 激酶依赖性凋亡

Q Remijsen, V Goossens, S Grootjans, C Van den Haute, N Vanlangenakker, Y Dondelinger, R Roelandt, I Bruggeman, A Goncalves, M J M Bertrand, V Baekelandt, N Takahashi, T V Berghe, P Vandenabeele

RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditions

在未解决的内质网应激条件下,RIPK1 促进死亡受体独立的 caspase-8 介导的细胞凋亡

Y Estornes, M A Aguileta, C Dubuisson, J De Keyser, V Goossens, K Kersse, A Samali, P Vandenabeele, M J M Bertrand

RIPK3 contributes to TNFR1-mediated RIPK1 kinase-dependent apoptosis in conditions of cIAP1/2 depletion or TAK1 kinase inhibition

在 cIAP1/2 耗竭或 TAK1 激酶抑制的情况下,RIPK3 有助于 TNFR1 介导的 RIPK1 激酶依赖性细胞凋亡

Y Dondelinger, M A Aguileta, V Goossens, C Dubuisson, S Grootjans, E Dejardin, P Vandenabeele, M J M Bertrand

Rapid detection of CFTR gene rearrangements impacts on genetic counselling in cystic fibrosis

快速检测CFTR基因重排对囊性纤维化遗传咨询有重要影响

Niel, F; Martin, J; Dastot-Le Moal, F; Costes, B; Boissier, B; Delattre, V; Goossens, M; Girodon, E