Valery Chavez相关文献与解析，生知库 | 链接生命科学的每一步

Achraf Shamseddine #, Suchit H Patel #, Valery Chavez, Zachary R Moore, Mutayyaba Adnan, Melody Di Bona, Jun Li, Chau T Dang, Lakshmi V Ramanathan, Kevin C Oeffinger, Jennifer E Liu, Richard M Steingart, Alessandra Piersigilli, Nicholas D Socci, Angel T Chan, Anthony F Yu, Samuel F Bakhoum, Adam M S

信号转导

肿瘤

IHC

CII

发表日期：2023

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AI-Assisted chemical probe discovery for the understudied Calcium-Calmodulin Dependent Kinase, PNCK

人工智能辅助化学探针发现，用于研究不足的钙钙调蛋白依赖性激酶 PNCK

期刊:PLoS Computational Biology

影响因子:3.8

doi:10.1371/journal.pcbi.1010263

Derek J Essegian, Valery Chavez, Rabia Khurshid, Jaime R Merchan, Stephan C Schürer

信号转导

发表日期：2023

文献求助收藏

Cellular and molecular effects of PNCK, a non-canonical kinase target in renal cell carcinoma

肾细胞癌中非经典激酶靶点 PNCK 的细胞和分子效应

期刊:iScience

影响因子:4.6

doi:10.1016/j.isci.2022.105621

Derek J Essegian, Valery Chavez, Floritza Bustamante, Stephan C Schürer, Jaime R Merchan

The C-terminus of Gain-of-Function Mutant p53 R273H Is Required for Association with PARP1 and Poly-ADP-Ribose

获得功能突变 p53 R273H 的 C 端是与 PARP1 和聚 ADP 核糖结合所必需的

期刊:Molecular Cancer Research

影响因子:4.1

doi:10.1158/1541-7786.MCR-22-0133

Devon Lundine #, George K Annor #, Valery Chavez, Styliana Maimos, Zafar Syed, Shuhong Jiang, Viola Ellison, Jill Bargonetti

In vivo antitumor activity by dual stromal and tumor-targeted oncolytic measles viruses

双基质和肿瘤靶向溶瘤麻疹病毒的体内抗肿瘤活性

期刊:Cancer Gene Therapy

影响因子:4.8

doi:10.1038/s41417-020-0171-1

Yuqi Jing, Valery Chavez, Natasha Khatwani, Yuguang Ban, Andrea P Espejo, Xi Chen, Jaime R Merchan

Molecular Effects of Stromal-Selective Targeting by uPAR-Retargeted Oncolytic Virus in Breast Cancer

uPAR 重靶向溶瘤病毒对乳腺癌基质选择性靶向的分子效应

期刊:Molecular Cancer Research

影响因子:4.1

doi:10.1158/1541-7786.MCR-17-0016

Yuqi Jing, Valery Chavez, Yuguang Ban, Nicolas Acquavella, Doraya El-Ashry, Alexey Pronin, Xi Chen, Jaime R Merchan

Hypoxic resistance of KRAS mutant tumor cells to 3-Bromopyruvate is counteracted by Prima-1 and reversed by N-acetylcysteine

Prima-1 可抵消 KRAS 突变肿瘤细胞对 3-溴丙酮酸的缺氧抗性，而 N-乙酰半胱氨酸可逆转该现象

期刊:BMC Cancer

影响因子:3.4

doi:10.1186/s12885-016-2930-9

Andrea Orue, Valery Chavez, Mary Strasberg-Rieber, Manuel Rieber

Innate immune signaling drives late cardiac toxicity following DNA-damaging cancer therapies

先天免疫信号驱动 DNA 损伤癌症治疗后的晚期心脏毒性

AI-Assisted chemical probe discovery for the understudied Calcium-Calmodulin Dependent Kinase, PNCK

人工智能辅助化学探针发现，用于研究不足的钙钙调蛋白依赖性激酶 PNCK

Cellular and molecular effects of PNCK, a non-canonical kinase target in renal cell carcinoma

肾细胞癌中非经典激酶靶点 PNCK 的细胞和分子效应

The C-terminus of Gain-of-Function Mutant p53 R273H Is Required for Association with PARP1 and Poly-ADP-Ribose

获得功能突变 p53 R273H 的 C 端是与 PARP1 和聚 ADP 核糖结合所必需的

In vivo antitumor activity by dual stromal and tumor-targeted oncolytic measles viruses

双基质和肿瘤靶向溶瘤麻疹病毒的体内抗肿瘤活性

Molecular Effects of Stromal-Selective Targeting by uPAR-Retargeted Oncolytic Virus in Breast Cancer

uPAR 重靶向溶瘤病毒对乳腺癌基质选择性靶向的分子效应

Hypoxic resistance of KRAS mutant tumor cells to 3-Bromopyruvate is counteracted by Prima-1 and reversed by N-acetylcysteine

Prima-1 可抵消 KRAS 突变肿瘤细胞对 3-溴丙酮酸的缺氧抗性，而 N-乙酰半胱氨酸可逆转该现象