Valianou相关文献与解析，生知库 | 链接生命科学的每一步

Matthildi Valianou, Natalia Filippidou, Daniel L Johnson, Peter Vogel, Erik Y Zhang, Xiaolei Liu, Yiyang Lu, Jane J Yu, John J Bissler, Aristotelis Astrinidis

免疫

细胞生物学

其它细胞

mTOR

发表日期：2019

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Sample Preparation Strategies for the Effective Quantitation of Hydrophilic Metabolites in Serum by Multi-Targeted HILIC-MS/MS

利用多靶点HILIC-MS/MS有效定量血清中亲水性代谢物的样品制备策略

期刊:Metabolites

影响因子:3.7

doi:10.3390/metabo7020013

Tsakelidou, Elisavet; Virgiliou, Christina; Valianou, Lemonia; Gika, Helen G; Raikos, Nikolaos; Theodoridis, Georgios

Pharmacological inhibition of Polo-like kinase 1 (PLK1) by BI-2536 decreases the viability and survival of hamartin and tuberin deficient cells via induction of apoptosis and attenuation of autophagy

BI-2536 通过药理学抑制 Polo 样激酶 1 (PLK1)，通过诱导细胞凋亡和减弱自噬，降低了 hamartin 和 tuberin 缺乏细胞的活力和存活率

期刊:Cell Cycle

影响因子:3.4

doi:10.4161/15384101.2014.986394

Matthildi Valianou, Andrew M Cox, Benjamin Pichette, Shannon Hartley, Unmesha Roy Paladhi, Aristotelis Astrinidis

Identification of inhibitory scFv antibodies targeting fibroblast activation protein utilizing phage display functional screens

利用噬菌体展示功能筛选鉴定针对成纤维细胞活化蛋白的抑制性 scFv 抗体

期刊:FASEB Journal

影响因子:4.4

doi:10.1096/fj.12-210377

Jiping Zhang, Matthildi Valianou, Heidi Simmons, Matthew K Robinson, Hyung-Ok Lee, Stefanie R Mullins, Wayne A Marasco, Gregory P Adams, Louis M Weiner, Jonathan D Cheng

FAP-overexpressing fibroblasts produce an extracellular matrix that enhances invasive velocity and directionality of pancreatic cancer cells

FAP过表达的成纤维细胞产生的细胞外基质可增强胰腺癌细胞的侵袭速度和方向性。

期刊:BMC Cancer

影响因子:3.4

doi:10.1186/1471-2407-11-245

Lee, Hyung-Ok; Mullins, Stefanie R; Franco-Barraza, Janusz; Valianou, Matthildi; Cukierman, Edna; Cheng, Jonathan D

Rapalog resistance is associated with mesenchymal-type changes in Tsc2-null cells

雷帕霉素抗性与 Tsc2 缺陷细胞的间充质型变化有关

Sample Preparation Strategies for the Effective Quantitation of Hydrophilic Metabolites in Serum by Multi-Targeted HILIC-MS/MS

利用多靶点HILIC-MS/MS有效定量血清中亲水性代谢物的样品制备策略

Pharmacological inhibition of Polo-like kinase 1 (PLK1) by BI-2536 decreases the viability and survival of hamartin and tuberin deficient cells via induction of apoptosis and attenuation of autophagy

BI-2536 通过药理学抑制 Polo 样激酶 1 (PLK1)，通过诱导细胞凋亡和减弱自噬，降低了 hamartin 和 tuberin 缺乏细胞的活力和存活率

Identification of inhibitory scFv antibodies targeting fibroblast activation protein utilizing phage display functional screens

利用噬菌体展示功能筛选鉴定针对成纤维细胞活化蛋白的抑制性 scFv 抗体

FAP-overexpressing fibroblasts produce an extracellular matrix that enhances invasive velocity and directionality of pancreatic cancer cells

FAP过表达的成纤维细胞产生的细胞外基质可增强胰腺癌细胞的侵袭速度和方向性。