日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

In mouse and in vitro models, bowel preparation promotes pathogen colonization, translocation, and exacerbation of inflammation

在小鼠和体外模型中,肠道准备会促进病原体定植、移位和炎症加剧。

Clayton, Charlotte A; Porter, Imogen; Deng, Brian D; McCallum, Giselle; Srinivas, Apsara; Sie, Claire; He, Jerry Y; Pei, Alexander D; Tertigas, Dominique; Pepin, Deanna M; Fardeen, Touran; Ng, Katharine M; Sinha, Sidhartha R; Surette, Michael G; Vallance, Bruce A; Tropini, Carolina

The growth and pathogenesis of Citrobacter rodentium are compromised when multiple mucin sugar utilization pathways are disrupted, leading to accumulation of N-acetylglucosamine 6-phosphate

当多种粘蛋白糖利用途径受到干扰时,鼠柠檬酸杆菌的生长和致病性会受到影响,导致N-乙酰氨基葡萄糖-6-磷酸的积累。

Huang, Zhiquan Clarence; Mslati, Matthias Ahmad; Ma, Caixia; Yang, Hyungjun; Liang, Qiaochu; Crowley, Shauna; Gilliland, Ashley; Dyer, Roger; Ng, Irvin; Yu, Hongbing; Vallance, Bruce A

The gut-skin axis: a bi-directional, microbiota-driven relationship with therapeutic potential

肠-皮肤轴:一种双向的、由微生物群驱动的、具有治疗潜力的关系

Jimenez-Sanchez, Maira; Celiberto, Larissa S; Yang, Hyungjun; Sham, Ho Pan; Vallance, Bruce A

Defining enteric bacterial pathogenesis using organoids: Citrobacter rodentium uses EspC, an atypical mucinolytic protease, to penetrate mouse colonic mucus.

利用类器官定义肠道细菌致病性:鼠柠檬酸杆菌利用非典型粘蛋白溶解蛋白酶 EspC 穿透小鼠结肠粘液

Chen Yan, Gilliland Ashley, Liang Qiaochu, Han Xiao, Yang Hyungjun, Chan Jocelyn, Lévesque Dominique, Moon Kyung-Mee, Daneshgar Parandis, Boisvert François-Michel, Foster Leonard, Zandberg Wesley F, Bergstrom Kirk, Yu Hong B, Vallance Bruce A

Crosstalk with infant-derived Th17 cells, as well as exposure to IL-22 promotes maturation of intestinal epithelial cells in an enteroid model.

与婴儿来源的 Th17 细胞的相互作用以及暴露于 IL-22 可促进肠类器官模型中肠上皮细胞的成熟

Sharafian Zohreh, Littlejohn Paula T, Michalski Christina, Sousa James A, Cheung Janelle, Hill Mariana, Piper Hannah, Jacobson Kevan, Lavoie Pascal M, Allaire Joannie M, Vallance Bruce A

Fasting elicits gut microbiome signature changes that extend to type 1 diabetes patients

禁食会引起肠道微生物群特征的变化,这种变化也适用于 1 型糖尿病患者。

Graef, Franziska A; Berger, Bettina; Bahr, Lina S; Stange, Rainer; Michalsen, Andreas; Paul, Friedemann; Vallance, Bruce A; Jacobson, Kevan

Differential roles for Irgm1 in myeloid cells for immune resistance to pathogenic bacteria

Irgm1在髓系细胞中对病原菌免疫抵抗的不同作用

Fee, Brian E; Fee, Lanette R; Sprouse, Aemilia R; Vallance, Bruce A; Rai, Prashant; Fessler, Michael B; Xue, Yue; Huang, Hsin-I; Hammer, Gianna E; Taylor, Gregory A

Inflammasome activation links enteric Salmonella Typhimurium infection to a rapid, cytokine-dependent increase in intestinal mucin release

炎症小体激活将肠道鼠伤寒沙门氏菌感染与肠道黏蛋白释放的快速、细胞因子依赖性增加联系起来

Han, Xiao; Allaire, Joannie M; Crowley, Shauna M; Chan, Jocelyn J; Lau, Kelly; Zhang, Conghao; Hirota, Simon A; Bergstrom, Kirk; Knodler, Leigh A; Vallance, Bruce A

Pathogenic diversification of the gut commensal Providencia alcalifaciens via acquisition of a second type III secretion system.

肠道共生菌Providencia alcalifaciens通过获得第二种III型分泌系统而发生致病性多样化

Klein Jessica A, Predeus Alexander V, Greissl Aimee R, Clark-Herrera Mattie M, Cruz Eddy, Cundiff Jennifer A, Haeberle Amanda L, Howell Maya, Lele Aaditi, Robinson Donna J, Westerman Trina L, Wrande Marie, Wright Sarah J, Green Nicole M, Vallance Bruce A, McClelland Michael, Mejia Andres, Goodman Alan G, Elfenbein Johanna R, Knodler Leigh A

Pathogenic diversification of the gut commensal Providencia alcalifaciens via acquisition of a second type III secretion system

肠道共生菌Providencia alcalifaciens通过获得第二种III型分泌系统而发生致病性多样化

Klein, Jessica A; Predeus, Alexander V; Greissl, Aimee R; Clark-Herrera, Mattie M; Cruz, Eddy; Cundiff, Jennifer A; Haeberle, Amanda L; Howell, Maya; Lele, Aaditi; Robinson, Donna J; Westerman, Trina L; Wrande, Marie; Wright, Sarah J; Green, Nicole M; Vallance, Bruce A; McClelland, Michael; Mejia, Andres; Goodman, Alan G; Elfenbein, Johanna R; Knodler, Leigh A