Vanida A Serna相关文献与解析，生知库 | 链接生命科学的每一步

Transient inhibition of p53 homologs protects ovarian function from two distinct apoptotic pathways triggered by anticancer therapies

暂时抑制 p53 同源物可保护卵巢功能免受抗癌疗法引发的两种不同凋亡途径的影响

期刊:Cell Death and Differentiation

影响因子:13.7

doi:10.1038/s41418-018-0151-2

So-Youn Kim, Devi M Nair, Megan Romero, Vanida A Serna, Anthony J Koleske, Teresa K Woodruff, Takeshi Kurita

Cellular kinetics of MED12-mutant uterine leiomyoma growth and regression in vivo

MED12 突变子宫平滑肌瘤体内生长和消退的细胞动力学

期刊:Endocrine-Related Cancer

影响因子:4.1

doi:10.1530/ERC-18-0184

Vanida A Serna, Xin Wu, Wenan Qiang, Justin Thomas, Michael L Blumenfeld, Takeshi Kurita

Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma

亚型特异性肿瘤相关成纤维细胞促进子宫平滑肌瘤的发病机制

期刊:Cancer Research

影响因子:12.5

doi:10.1158/0008-5472.CAN-17-1744

Xin Wu, Vanida A Serna, Justin Thomas, Wenan Qiang, Michael L Blumenfeld, Takeshi Kurita

Novel SOX17 frameshift mutations in endometrial cancer are functionally distinct from recurrent missense mutations

子宫内膜癌中新的 SOX17 移码突变在功能上与复发性错义突变不同

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.20213

Christopher J Walker, Matthew J O'Hern, Vanida A Serna, Takeshi Kurita, Mario A Miranda, Caroline E Sapp, David G Mutch, David E Cohn, Paul J Goodfellow

FGFR2IIIb-MAPK Activity Is Required for Epithelial Cell Fate Decision in the Lower Müllerian Duct

FGFR2IIIb-MAPK 活性是下苗勒氏管上皮细胞命运决定所必需的

期刊:Molecular Endocrinology

影响因子:

doi:10.1210/me.2016-1027

Jumpei Terakawa, Altea Rocchi, Vanida A Serna, Erwin P Bottinger, Jonathan M Graff, Takeshi Kurita

Diethylstilbestrol induces vaginal adenosis by disrupting SMAD/RUNX1-mediated cell fate decision in the Müllerian duct epithelium

己烯雌酚通过破坏苗勒氏管上皮中的 SMAD/RUNX1 介导的细胞命运决定来诱发阴道腺病

期刊:Developmental Biology

影响因子:2.5

doi:10.1016/j.ydbio.2013.06.024

Monica M Laronda, Kenji Unno, Kazutomo Ishi, Vanida A Serna, Lindsey M Butler, Alea A Mills, Grant D Orvis, Richard R Behringer, Chuxia Deng, Satrajit Sinha, Takeshi Kurita

SIX1 cooperates with RUNX1 and SMAD4 in cell fate commitment of Müllerian duct epithelium

SIX1 与 RUNX1 和 SMAD4 协同决定苗勒氏管上皮细胞的命运

Transient inhibition of p53 homologs protects ovarian function from two distinct apoptotic pathways triggered by anticancer therapies

暂时抑制 p53 同源物可保护卵巢功能免受抗癌疗法引发的两种不同凋亡途径的影响

Cellular kinetics of MED12-mutant uterine leiomyoma growth and regression in vivo

MED12 突变子宫平滑肌瘤体内生长和消退的细胞动力学

Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma

亚型特异性肿瘤相关成纤维细胞促进子宫平滑肌瘤的发病机制

Novel SOX17 frameshift mutations in endometrial cancer are functionally distinct from recurrent missense mutations

子宫内膜癌中新的 SOX17 移码突变在功能上与复发性错义突变不同

FGFR2IIIb-MAPK Activity Is Required for Epithelial Cell Fate Decision in the Lower Müllerian Duct

FGFR2IIIb-MAPK 活性是下苗勒氏管上皮细胞命运决定所必需的

Diethylstilbestrol induces vaginal adenosis by disrupting SMAD/RUNX1-mediated cell fate decision in the Müllerian duct epithelium

己烯雌酚通过破坏苗勒氏管上皮中的 SMAD/RUNX1 介导的细胞命运决定来诱发阴道腺病