日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Quantification of cerivastatin toxicity supports organismal performance assays as an effective tool during pharmaceutical safety assessment

对西立伐他汀毒性的量化分析支持将生物体性能测定作为药物安全性评估中的有效工具。

Gaukler, Shannon M; Ruff, James S; Galland, Tessa; Underwood, Tristan K; Kandaris, Kirstie A; Liu, Nicole M; Morrison, Linda C; Veranth, John M; Potts, Wayne K

Activation of Transient Receptor Potential Ankyrin-1 by Insoluble Particulate Material and Association with Asthma

不溶性颗粒物质激活瞬时受体电位锚蛋白-1及其与哮喘的关系

Deering-Rice, Cassandra E; Shapiro, Darien; Romero, Erin G; Stockmann, Chris; Bevans, Tatjana S; Phan, Quang M; Stone, Bryan L; Fassl, Bernhard; Nkoy, Flory; Uchida, Derek A; Ward, Robert M; Veranth, John M; Reilly, Christopher A

Activation of transient receptor potential ankyrin-1 (TRPA1) in lung cells by wood smoke particulate material

木材烟雾颗粒物激活肺细胞中的瞬时受体电位锚蛋白-1 (TRPA1)

Shapiro, Darien; Deering-Rice, Cassandra E; Romero, Erin G; Hughen, Ronald W; Light, Alan R; Veranth, John M; Reilly, Christopher A

Transient receptor potential vanilloid-1 (TRPV1) is a mediator of lung toxicity for coal fly ash particulate material

瞬时受体电位香草酸受体1 (TRPV1) 是煤粉灰颗粒物肺毒性的介质

Deering-Rice, Cassandra E; Johansen, Mark E; Roberts, Jessica K; Thomas, Karen C; Romero, Erin G; Lee, Jeewoo; Yost, Garold S; Veranth, John M; Reilly, Christopher A

Electrophilic components of diesel exhaust particles (DEP) activate transient receptor potential ankyrin-1 (TRPA1): a probable mechanism of acute pulmonary toxicity for DEP

柴油机尾气颗粒物(DEP)中的亲电成分激活瞬时受体电位锚蛋白-1(TRPA1):这可能是DEP引起急性肺毒性的机制之一。

Deering-Rice, Cassandra E; Romero, Erin G; Shapiro, Darien; Hughen, Ronald W; Light, Alan R; Yost, Garold S; Veranth, John M; Reilly, Christopher A

Correlation of in vitro cytokine responses with the chemical composition of soil-derived particulate matter.

体外细胞因子反应与土壤颗粒物化学成分的相关性

Veranth John M, Moss Tyler A, Chow Judith C, Labban Raed, Nichols William K, Walton John C, Watson John G, Yost Garold S

Inflammatory cytokines and cell death in BEAS-2B lung cells treated with soil dust, lipopolysaccharide, and surface-modified particles.

土壤粉尘、脂多糖和表面修饰颗粒处理 BEAS-2B 肺细胞后,炎症细胞因子和细胞死亡情况

Veranth John M, Reilly Christopher A, Veranth Martha M, Moss Tyler A, Langelier Charles R, Lanza Diane L, Yost Garold S