日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Internalized SNCA/α-synuclein fibrils become truncated and resist degradation in neurons while glial cells rapidly degrade SNCA fibrils.

神经元内化的 SNCA/α-突触核蛋白原纤维会发生截断并抵抗降解,而神经胶质细胞则会迅速降解 SNCA 原纤维。

Karim Md Razaul, Tiegs Elizabeth, Gasparini Emilie, Schlichte Riley, Vermilyea Scott C, Lee Michael K

Epigenetic sperm quality testing for predicting fertility treatment success: a real-world and multi-site analysis

利用表观遗传学方法检测精子质量以预测生育治疗成功率:一项真实世界多中心分析

Brogaard, Kristin; Hudson, Susan; Bedient, Carrie; Eblen, Abby; Van Heertum, Kristin; Giuliani, Emma; Smith, Meghan; Kaye, Madeline; Schutt, Amy; Hansard, Lisa; Shavell, Valerie; Silverberg, Kaylen; Thakur, Mili; Olson, Andrew; Miller, Ryan; Schneider, Lorry; Petersen, Derek; Daines, Bryce; Robinson, Lakingya; Reynolds, Alexis; Singh, Nirvika; Kaskar, Khalied; Kassab, Jordan; Lipshultz, Larry; VerMilyea, Matthew

A diluted one-step warming protocol: survival of vitrified blastocyst

稀释的一步复温方案:玻璃化冷冻囊胚的存活率

Licata, Emanuele; VerMilyea, Matthew; Ducote, Brooke; Ferguson, Tonya; Bianco, Claudia; Gallo, Mariagrazia; Paciotti, Gemma; Passerini, Renata; Meneghini, Caterina; Fabiani, Cristina; Galanti, Francesco; Dal Lago, Alessandro; Rago, Rocco

Soluble and Insoluble Lysates from the Human A53T Mutant α-Synuclein Transgenic Mouse Model Induces α-Synucleinopathy Independent of Injection Site

来自人A53T突变α-突触核蛋白转基因小鼠模型的可溶性和不溶性裂解物可诱导α-突触核蛋白病,且与注射部位无关。

Barnes, Justin; Vermilyea, Scott C; Meints, Joyce; Martell-Martinez, Héctor; Lee, Michael K

Protein kinase CK2α' as a dual modulator of immune signaling and synaptic dysfunction in Tauopathy.

蛋白激酶 CK2α' 在 Tau 蛋白病中作为免疫信号传导和突触功能障碍的双重调节因子。

White Angel, Gavrilyuk Peter, Gu Persephone, Moya Rafael Falcon, Thurston Reid, Fickak Amal, Rozema Nicholas B, Keshavaram Prarthana, Vermilyea Scott, Schlichte Riley, Meints Joyce, Zhang Ying, Araque Alfonso, Lee Michael, Gomez-Pastor Rocio

Protein kinase CK2α' as a dual modulator of neuroimmune signaling and synaptic dysfunction in Tauopathy.

蛋白激酶 CK2α' 在 Tau 蛋白病中作为神经免疫信号传导和突触功能障碍的双重调节因子

White Angel, Gavrilyuk Peter, Moya Rafael Falcon, Thurston Reid, Fickak Amal, Rozema Nicholas B, Keshavaram Prarthana, Vermilyea Scott, Schlichte Riley, Meints Joyce, Zhang Ying, Araque Alfonso, Lee Michael, Gomez-Pastor Rocio

Landau-phonon polaritons in Dirac heterostructures

狄拉克异质结构中的朗道声子极化子

Wehmeier, Lukas; Xu, Suheng; Mayer, Rafael A; Vermilyea, Brian; Tsuneto, Makoto; Dapolito, Michael; Pu, Rui; Du, Zengyi; Chen, Xinzhong; Zheng, Wenjun; Jing, Ran; Zhou, Zijian; Watanabe, Kenji; Taniguchi, Takashi; Gozar, Adrian; Li, Qiang; Kuzmenko, Alexey B; Carr, G Lawrence; Du, Xu; Fogler, Michael M; Basov, D N; Liu, Mengkun

Internalized α-synuclein fibrils become truncated and resist degradation in neurons while glial cells rapidly degrade α-synuclein fibrils

内化的 α-突触核蛋白原纤维被截短并抵抗神经元中的降解,而神经胶质细胞则迅速降解 α-突触核蛋白原纤维

Md Razaul Karim, Emilie Gasparini, Elizabeth Tiegs, Riley Schlichte, Scott C Vermilyea, Michael K Lee

Soluble and insoluble lysates from the human A53T mutant α-synuclein transgenic mouse model induces α-synucleinopathy independent of injection site

来自人A53T突变α-突触核蛋白转基因小鼠模型的可溶性和不溶性裂解物均可诱导α-突触核蛋白病,且与注射部位无关。

Lee, Michael; Barnes, Justin; Vermilyea, Scott; Meints, Joyce; Martinez, Hector

Correction to: Dysregulation of astrocytic Ca(2+) signaling and gliotransmitter release in mouse models of α-synucleinopathies

更正:α-突触核蛋白病小鼠模型中星形胶质细胞Ca(2+)信号传导和胶质递质释放的失调

Nanclares, Carmen; Poynter, Jonah; Martell-Martinez, Hector A; Vermilyea, Scott; Araque, Alfonso; Kofuji, Paulo; Lee, Michael K; Covelo, Ana