日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Ion Channel Partnerships: Odd and Not-So-Odd Couples Controlling Neuronal Ion Channel Function

离子通道的相互作用:控制神经元离子通道功能的奇特组合和不那么奇特的组合

Vierra, Nicholas C; Trimmer, James S

Regulation of neuronal excitation-transcription coupling by Kv2.1-induced clustering of somatic L-type Ca(2+) channels at ER-PM junctions

Kv2.1诱导胞体L型Ca(2+)通道在内质网-质膜连接处聚集,从而调节神经元兴奋-转录偶联

Vierra, Nicholas C; O'Dwyer, Samantha C; Matsumoto, Collin; Santana, L Fernando; Trimmer, James S

Glucocorticoids Reprogram β-Cell Signaling to Preserve Insulin Secretion

糖皮质激素重编程β细胞信号传导以维持胰岛素分泌

Fine, Nicholas H F; Doig, Craig L; Elhassan, Yasir S; Vierra, Nicholas C; Marchetti, Piero; Bugliani, Marco; Nano, Rita; Piemonti, Lorenzo; Rutter, Guy A; Jacobson, David A; Lavery, Gareth G; Hodson, David J

TASK-1 Potassium Channels Limit Pancreatic α-Cell Calcium Influx and Glucagon Secretion

任务一:钾通道限制胰岛α细胞钙离子内流和胰高血糖素分泌

Dadi, Prasanna K; Luo, Brooke; Vierra, Nicholas C; Jacobson, David A

Inhibition of pancreatic β-cell Ca2+/calmodulin-dependent protein kinase II reduces glucose-stimulated calcium influx and insulin secretion, impairing glucose tolerance.

抑制胰岛β细胞Ca2+/钙调蛋白依赖性蛋白激酶II可减少葡萄糖刺激的钙离子内流和胰岛素分泌,从而损害葡萄糖耐量

Dadi Prasanna K, Vierra Nicholas C, Ustione Alessandro, Piston David W, Colbran Roger J, Jacobson David A