Vilotti相关文献与解析，生知库 | 链接生命科学的每一步

Frasson, Emma; Simonetto, Marco; Bertolasi, Laura; Caneve, Giorgio; Vilotti, Cristina; Ruzza, Giampietro; Perelli, Anna; Piccinno, Maria Grazia; Monaco, Salvatore

重症肌无力

免疫/内分泌

发表日期：2021

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The ASIC3/P2X3 cognate receptor is a pain-relevant and ligand-gated cationic channel

ASIC3/P2X3 同源受体是一种与疼痛相关且受配体门控的阳离子通道

期刊:Nature Communications

影响因子:14.7

doi:10.1038/s41467-018-03728-5

Gabriele Stephan, Lumei Huang, Yong Tang, Sandra Vilotti, Elsa Fabbretti, Ye Yu, Wolfgang Nörenberg, Heike Franke, Flóra Gölöncsér, Beáta Sperlágh, Anke Dopychai, Ralf Hausmann, Günther Schmalzing, Patrizia Rubini, Peter Illes

信号转导

发表日期：2018

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Inefficient constitutive inhibition of P2X3 receptors by brain natriuretic peptide system contributes to sensitization of trigeminal sensory neurons in a genetic mouse model of familial hemiplegic migraine

脑钠肽系统对 P2X3 受体的无效组成性抑制导致家族性偏瘫性偏头痛遗传小鼠模型中三叉神经感觉神经元的敏化

期刊:Molecular Pain

影响因子:2.8

doi:10.1177/1744806916646110

Anna Marchenkova, Sandra Vilotti, Niels Ntamati, Arn Mjm van den Maagdenberg, Andrea Nistri

神经

发表日期：2016

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Brain natriuretic peptide constitutively downregulates P2X3 receptors by controlling their phosphorylation state and membrane localization

脑钠肽通过控制 P2X3 受体的磷酸化状态和膜定位来持续下调 P2X3 受体

期刊:Molecular Pain

影响因子:2.8

doi:10.1186/s12990-015-0074-6

Anna Marchenkova, Sandra Vilotti, Elsa Fabbretti, Andrea Nistri

表观遗传

发表日期：2015

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B-type natriuretic peptide-induced delayed modulation of TRPV1 and P2X3 receptors of mouse trigeminal sensory neurons

B型利钠肽诱导小鼠三叉神经感觉神经元TRPV1和P2X3受体的延迟调制

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0081138

Sandra Vilotti, Anna Marchenkova, Niels Ntamati, Andrea Nistri

神经

发表日期：2013

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TNFα levels and macrophages expression reflect an inflammatory potential of trigeminal ganglia in a mouse model of familial hemiplegic migraine

在家族性偏瘫性偏头痛小鼠模型中，TNFα水平和巨噬细胞表达反映了三叉神经节的炎症潜能。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0052394

Franceschini, Alessia; Vilotti, Sandra; Ferrari, Michel D; van den Maagdenberg, Arn M J M; Nistri, Andrea; Fabbretti, Elsa

The PML nuclear bodies-associated protein TTRAP regulates ribosome biogenesis in nucleolar cavities upon proteasome inhibition

PML 核体相关蛋白 TTRAP 在蛋白酶体抑制后调节核仁腔内核糖体的生物合成

期刊:Cell Death and Differentiation

影响因子:13.7

doi:10.1038/cdd.2011.118

S Vilotti, M Biagioli, R Foti, M Dal Ferro, Z Scotto Lavina, L Collavin, G Del Sal, S Zucchelli, S Gustincich

信号转导

发表日期：2012

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Parkinson's disease DJ-1 L166P alters rRNA biogenesis by exclusion of TTRAP from the nucleolus and sequestration into cytoplasmic aggregates via TRAF6

帕金森病 DJ-1 L166P 通过将 TTRAP 从核仁中排除并通过 TRAF6 隔离到细胞质聚集体中来改变 rRNA 的生物合成

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0035051

Sandra Vilotti, Marta Codrich, Marco Dal Ferro, Milena Pinto, Isidro Ferrer, Licio Collavin, Stefano Gustincich, Silvia Zucchelli

Tumor necrosis factor receptor-associated factor 6 (TRAF6) associates with huntingtin protein and promotes its atypical ubiquitination to enhance aggregate formation

肿瘤坏死因子受体相关因子 6 (TRAF6) 与亨廷顿蛋白相关并促进其非典型泛素化以增强聚集体形成

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1074/jbc.M110.187591

Silvia Zucchelli, Federica Marcuzzi, Marta Codrich, Elena Agostoni, Sandra Vilotti, Marta Biagioli, Milena Pinto, Alisia Carnemolla, Claudio Santoro, Stefano Gustincich, Francesca Persichetti

TRAF6 promotes atypical ubiquitination of mutant DJ-1 and alpha-synuclein and is localized to Lewy bodies in sporadic Parkinson's disease brains

TRAF6 促进突变型 DJ-1 和 α-突触核蛋白的非典型泛素化，并定位于散发性帕金森病患者大脑中的路易体

期刊:Human Molecular Genetics

影响因子:3.1

doi:10.1093/hmg/ddq290

Silvia Zucchelli, Marta Codrich, Federica Marcuzzi, Milena Pinto, Sandra Vilotti, Marta Biagioli, Isidro Ferrer, Stefano Gustincich

Statin-associated necrotizing autoimmune myopathy with concurrent myasthenia gravis

他汀类药物相关性坏死性自身免疫性肌病伴重症肌无力

The ASIC3/P2X3 cognate receptor is a pain-relevant and ligand-gated cationic channel

ASIC3/P2X3 同源受体是一种与疼痛相关且受配体门控的阳离子通道

Inefficient constitutive inhibition of P2X3 receptors by brain natriuretic peptide system contributes to sensitization of trigeminal sensory neurons in a genetic mouse model of familial hemiplegic migraine

脑钠肽系统对 P2X3 受体的无效组成性抑制导致家族性偏瘫性偏头痛遗传小鼠模型中三叉神经感觉神经元的敏化

Brain natriuretic peptide constitutively downregulates P2X3 receptors by controlling their phosphorylation state and membrane localization

脑钠肽通过控制 P2X3 受体的磷酸化状态和膜定位来持续下调 P2X3 受体

B-type natriuretic peptide-induced delayed modulation of TRPV1 and P2X3 receptors of mouse trigeminal sensory neurons

B型利钠肽诱导小鼠三叉神经感觉神经元TRPV1和P2X3受体的延迟调制

TNFα levels and macrophages expression reflect an inflammatory potential of trigeminal ganglia in a mouse model of familial hemiplegic migraine

在家族性偏瘫性偏头痛小鼠模型中，TNFα水平和巨噬细胞表达反映了三叉神经节的炎症潜能。

The PML nuclear bodies-associated protein TTRAP regulates ribosome biogenesis in nucleolar cavities upon proteasome inhibition

PML 核体相关蛋白 TTRAP 在蛋白酶体抑制后调节核仁腔内核糖体的生物合成

Parkinson's disease DJ-1 L166P alters rRNA biogenesis by exclusion of TTRAP from the nucleolus and sequestration into cytoplasmic aggregates via TRAF6

帕金森病 DJ-1 L166P 通过将 TTRAP 从核仁中排除并通过 TRAF6 隔离到细胞质聚集体中来改变 rRNA 的生物合成

Tumor necrosis factor receptor-associated factor 6 (TRAF6) associates with huntingtin protein and promotes its atypical ubiquitination to enhance aggregate formation

肿瘤坏死因子受体相关因子 6 (TRAF6) 与亨廷顿蛋白相关并促进其非典型泛素化以增强聚集体形成

TRAF6 promotes atypical ubiquitination of mutant DJ-1 and alpha-synuclein and is localized to Lewy bodies in sporadic Parkinson's disease brains

TRAF6 促进突变型 DJ-1 和 α-突触核蛋白的非典型泛素化，并定位于散发性帕金森病患者大脑中的路易体