Vilsen相关文献与解析，生知库 | 链接生命科学的每一步

Na+,K+-ATPase with Disrupted Na+ Binding Sites I and III Binds Na+ with Increased Affinity at Site II and Undergoes Na+-Activated Phosphorylation with ATP

Na+,K+-ATPase 的 Na+ 结合位点 I 和 III 被破坏，与位点 II 处的 Na+ 结合亲和力增加，并与 ATP 发生 Na+ 活化的磷酸化

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom14010135

Hang N Nielsen, Rikke Holm, Ryan Sweazey, Jens Peter Andersen, Pablo Artigas, Bente Vilsen

表观遗传

发表日期：2024

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Dataset of lithium-ion battery degradation based on a forklift mission profile for state-of-health estimation and lifetime prediction

基于叉车作业场景的锂离子电池退化数据集，用于电池健康状态评估和寿命预测

期刊:Data in Brief

影响因子:1.4

doi:10.1016/j.dib.2023.109861

Vilsen, Søren B; Stroe, Daniel-Ioan

发表日期：2024

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Temperature instability of a mutation at a multidomain junction in Na,K-ATPase isoform ATP1A3 (p.Arg756His) produces a fever-induced neurological syndrome

Na,K-ATPase 同工型 ATP1A3 (p.Arg756His) 多域连接突变的温度不稳定性导致发烧引起的神经系统综合征

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1016/j.jbc.2022.102758

Elena Arystarkhova, Mads S Toustrup-Jensen, Rikke Holm, Jae-Kyun Ko, Kyung Eun Lee, Polina Feschenko, Laurie J Ozelius, Allison Brashear, Bente Vilsen, Kathleen J Sweadner

神经

发表日期：2023

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Role of a conserved ion-binding site tyrosine in ion selectivity of the Na+/K+ pump

保守离子结合位点酪氨酸在 Na+/K+ 泵离子选择性中的作用

期刊:Journal of General Physiology

影响因子:3.3

doi:10.1085/jgp.202113039

Kerri Spontarelli, Daniel T Infield, Hang N Nielsen, Rikke Holm, Victoria C Young, Jason D Galpin, Christopher A Ahern, Bente Vilsen, Pablo Artigas

信号转导

发表日期：2022

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Binding of cardiotonic steroids to Na(+),K(+)-ATPase in the E2P state

强心甾类激素与E2P状态下Na(+),K(+)-ATP酶的结合

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.2020438118

Kanai, Ryuta; Cornelius, Flemming; Ogawa, Haruo; Motoyama, Kanna; Vilsen, Bente; Toyoshima, Chikashi

免疫/内分泌

发表日期：2021

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The α2β2 isoform combination dominates the astrocytic Na+ /K+ -ATPase activity and is rendered nonfunctional by the α2.G301R familial hemiplegic migraine type 2-associated mutation

α2β2 同工型组合主导星形胶质细胞 Na+ /K+ -ATPase 活性，并因 α2.G301R 家族性偏瘫性偏头痛 2 型相关突变而失去功能

期刊:Glia

影响因子:5.4

doi:10.1002/glia.23194

Anca Stoica, Brian Roland Larsen, Mette Assentoft, Rikke Holm, Leanne Melissa Holt, Frederik Vilhardt, Bente Vilsen, Karin Lykke-Hartmann, Michelle Lynne Olsen, Nanna MacAulay

Glutamate transporter activity promotes enhanced Na+ /K+ -ATPase-mediated extracellular K+ management during neuronal activity

谷氨酸转运蛋白活性促进神经元活动期间 Na+ /K+ -ATPase 介导的细胞外 K+ 管理增强

期刊:Journal of Physiology-London

影响因子:2.1

doi:10.1113/JP272531

Brian Roland Larsen, Rikke Holm, Bente Vilsen, Nanna MacAulay

Rescue of Na+ affinity in aspartate 928 mutants of Na+,K+-ATPase by secondary mutation of glutamate 314

通过谷氨酸314的二次突变挽救Na+,K+-ATPase天冬氨酸928突变体的Na+亲和力

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M114.625509

Holm, Rikke; Einholm, Anja P; Andersen, Jens P; Vilsen, Bente

发表日期：2015

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Distinct neurological disorders with ATP1A3 mutations

ATP1A3基因突变引起的不同神经系统疾病

期刊:Lancet Neurology

影响因子:45.5

doi:10.1016/S1474-4422(14)70011-0

Heinzen, Erin L; Arzimanoglou, Alexis; Brashear, Allison; Clapcote, Steven J; Gurrieri, Fiorella; Goldstein, David B; Jóhannesson, Sigurður H; Mikati, Mohamad A; Neville, Brian; Nicole, Sophie; Ozelius, Laurie J; Poulsen, Hanne; Schyns, Tsveta; Sweadner, Kathleen J; van den Maagdenberg, Arn; Vilsen, Bente

发表日期：2014

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How palytoxin transforms the Na(+),K(+) pump into a cation channel

帕利毒素如何将钠钾泵转化为阳离子通道

Na+,K+-ATPase with Disrupted Na+ Binding Sites I and III Binds Na+ with Increased Affinity at Site II and Undergoes Na+-Activated Phosphorylation with ATP

Na+,K+-ATPase 的 Na+ 结合位点 I 和 III 被破坏，与位点 II 处的 Na+ 结合亲和力增加，并与 ATP 发生 Na+ 活化的磷酸化

Dataset of lithium-ion battery degradation based on a forklift mission profile for state-of-health estimation and lifetime prediction

基于叉车作业场景的锂离子电池退化数据集，用于电池健康状态评估和寿命预测

Temperature instability of a mutation at a multidomain junction in Na,K-ATPase isoform ATP1A3 (p.Arg756His) produces a fever-induced neurological syndrome

Na,K-ATPase 同工型 ATP1A3 (p.Arg756His) 多域连接突变的温度不稳定性导致发烧引起的神经系统综合征

Role of a conserved ion-binding site tyrosine in ion selectivity of the Na+/K+ pump

保守离子结合位点酪氨酸在 Na+/K+ 泵离子选择性中的作用

Binding of cardiotonic steroids to Na(+),K(+)-ATPase in the E2P state

强心甾类激素与E2P状态下Na(+),K(+)-ATP酶的结合

The α2β2 isoform combination dominates the astrocytic Na+ /K+ -ATPase activity and is rendered nonfunctional by the α2.G301R familial hemiplegic migraine type 2-associated mutation

α2β2 同工型组合主导星形胶质细胞 Na+ /K+ -ATPase 活性，并因 α2.G301R 家族性偏瘫性偏头痛 2 型相关突变而失去功能

Glutamate transporter activity promotes enhanced Na+ /K+ -ATPase-mediated extracellular K+ management during neuronal activity

谷氨酸转运蛋白活性促进神经元活动期间 Na+ /K+ -ATPase 介导的细胞外 K+ 管理增强

Rescue of Na+ affinity in aspartate 928 mutants of Na+,K+-ATPase by secondary mutation of glutamate 314

通过谷氨酸314的二次突变挽救Na+,K+-ATPase天冬氨酸928突变体的Na+亲和力

Distinct neurological disorders with ATP1A3 mutations

ATP1A3基因突变引起的不同神经系统疾病