日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

A CD40-targeting peptide, OPT501, modulates inflammation in canine diabetes mellitus improving clinical outcomes.

CD40靶向肽OPT501可调节犬糖尿病的炎症,从而改善临床结果。

Vaitaitis Gisela M, Waid Dan M, Sharkey Christina, Sharkey Steve, Webb Tracy L, Webb Craig, Wagner David H Jr

Canine diabetes mellitus demonstrates multiple markers of chronic inflammation including Th40 cell increases and elevated systemic-immune inflammation index, consistent with autoimmune dysregulation.

犬糖尿病表现出多种慢性炎症标志,包括 Th40 细胞增加和全身免疫炎症指数升高,这与自身免疫失调一致

Vaitaitis Gisela, Webb Tracy, Webb Craig, Sharkey Christina, Sharkey Steve, Waid Dan, Wagner David H

Ocrevus reduces TH40 cells, a biomarker of systemic inflammation, in relapsing multiple sclerosis (RMS) and in progressive multiple sclerosis (PMS)

Ocrevus 可减少复发型多发性硬化症 (RMS) 和进展型多发性硬化症 (PMS) 中的 TH40 细胞(一种全身炎症的生物标志物)。

Curran, Christian; Vaitaitis, Gisela; Waid, Dan; Volmer, Timothy; Alverez, Enrique; Wagner, David H

CD40-targeted peptide proposed for type 1 diabetes therapy lacks relevant binding affinity to its cognate receptor. Reply to Pagni PP, Wolf A, Lo Conte M et al [letter]

用于治疗 1 型糖尿病的 CD40 靶向肽缺乏与其同源受体相关的结合亲和力。回复 Pagni PP、Wolf A、Lo Conte M 等人的[信函]

Vaitaitis, Gisela M; Olmstead, Michael H; Waid, Dan M; Carter, Jessica R; Wagner, David H Jr

Biomarker Discovery in Pre-Type 1 Diabetes; Th40 Cells as a Predictive Risk Factor

1型糖尿病前期生物标志物的发现;Th40细胞作为预测风险因素

Vaitaitis, Gisela M; Rihanek, Marynette; Alkanani, Aimon K; Waid, Dan M; Gottlieb, Peter A; Wagner, David H

A CD40-targeted peptide controls and reverses type 1 diabetes in NOD mice

一种靶向CD40的肽能够控制并逆转NOD小鼠的1型糖尿病

Vaitaitis, Gisela M; Olmstead, Michael H; Waid, Dan M; Carter, Jessica R; Wagner, David H Jr

Defining a new biomarker for the autoimmune component of Multiple Sclerosis: Th40 cells

确定多发性硬化症自身免疫成分的新生物标志物:Th40细胞

Waid, Dan M; Schreiner, Teri; Vaitaitis, Gisela; Carter, Jessica R; Corboy, John R; Wagner, David H Jr

An alternative role for Foxp3 as an effector T cell regulator controlled through CD40

Foxp3 的另一种作用是作为效应 T 细胞调节因子,其调控受 CD40 控制。

Vaitaitis, Gisela M; Carter, Jessica R; Waid, Dan M; Olmstead, Michael H; Wagner, David H Jr

Disruption of the homeostatic balance between autoaggressive (CD4+CD40+) and regulatory (CD4+CD25+FoxP3+) T cells promotes diabetes

自身反应性(CD4+CD40+)和调节性(CD4+CD25+FoxP3+)T细胞之间稳态平衡的破坏会促进糖尿病的发生。

Waid, Dan M; Vaitaitis, Gisela M; Pennock, Nathan D; Wagner, David H Jr