TRIM24-mediated K27-linked ubiquitination of ULK1 alleviates energy stress-induced autophagy and promote prostate cancer growth in the context of SPOP mutation
TRIM24介导的ULK1 K27连接的泛素化可减轻能量应激诱导的自噬，并在SPOP突变的情况下促进前列腺癌的生长。
期刊:Cell Death and Differentiation
影响因子:15.4
doi:10.1038/s41418-025-01582-9
Chen, Shimin; Lin, Jichun; Yang, Zhan; Wang, Yuanjing; Wang, Qiang; Wang, Dong; Qu, Yue; Lin, Qian; Liu, Jia; Yan, Shi; Wang, Zixin; Qian, Xueyu; Xiao, Yutian; Li, Xue; Chen, Yinuo; Fang, Wenshuo; Zhao, Jiaojiao; Lu, Zhimin; Ren, He; Zhu, Yasheng; Ma, Leina
肿瘤
表观遗传
肿瘤免疫
ULK1
泛素化
SPOP
前列腺癌
自噬
