日期 影响因子
日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:
–

Metabolomic analysis of pressure-overloaded and infarcted mouse hearts

对压力超负荷和梗死小鼠心脏进行代谢组学分析

期刊:Circulation-Heart Failure
影响因子:8.4
doi:10.1161/CIRCHEARTFAILURE.114.001151
Sansbury, Brian E; DeMartino, Angelica M; Xie, Zhengzhi; Brooks, Alan C; Brainard, Robert E; Watson, Lewis J; DeFilippis, Andrew P; Cummins, Timothy D; Harbeson, Matthew A; Brittian, Kenneth R; Prabhu, Sumanth D; Bhatnagar, Aruni; Jones, Steven P; Hill, Bradford G
代谢
发表日期：2014
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Cardiomyocyte Ogt is essential for postnatal viability

心肌细胞Ogt对于出生后的存活至关重要

期刊:American Journal of Physiology-Heart and Circulatory Physiology
影响因子:4.1
doi:10.1152/ajpheart.00438.2013
Watson, Lewis J; Long, Bethany W; DeMartino, Angelica M; Brittian, Kenneth R; Readnower, Ryan D; Brainard, Robert E; Cummins, Timothy D; Annamalai, Lakshmanan; Hill, Bradford G; Jones, Steven P
细胞生物学
发表日期：2014
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MicroRNA-539 is up-regulated in failing heart, and suppresses O-GlcNAcase expression.

MicroRNA-539 在衰竭的心脏中上调，并抑制 O-GlcNAcase 的表达

期刊:Journal of Biological Chemistry
影响因子:3.9
doi:10.1074/jbc.M114.578682
Muthusamy Senthilkumar, DeMartino Angelica M, Watson Lewis J, Brittian Kenneth R, Zafir Ayesha, Dassanayaka Sujith, Hong Kyung U, Jones Steven P
心血管
发表日期：2014
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High fat feeding in mice is insufficient to induce cardiac dysfunction and does not exacerbate heart failure

高脂饮食不足以诱发小鼠心脏功能障碍，也不会加剧心力衰竭。

期刊:PLoS One
影响因子:2.6
doi:10.1371/journal.pone.0083174
Brainard, Robert E; Watson, Lewis J; Demartino, Angelica M; Brittian, Kenneth R; Readnower, Ryan D; Boakye, Adjoa Agyemang; Zhang, Deqing; Hoetker, Joseph David; Bhatnagar, Aruni; Baba, Shahid Pervez; Jones, Steven P
心力衰竭
心血管
发表日期：2013
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Reduced cardiac fructose 2,6 bisphosphate increases hypertrophy and decreases glycolysis following aortic constriction

主动脉缩窄后，心脏果糖2,6-二磷酸水平降低可增加心肌肥大并减少糖酵解。

期刊:PLoS One
影响因子:2.6
doi:10.1371/journal.pone.0053951
Wang, Jianxun; Xu, Jianxiang; Wang, Qianwen; Brainard, Robert E; Watson, Lewis J; Jones, Steven P; Epstein, Paul N
发表日期：2013
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O-GlcNAc signaling is essential for NFAT-mediated transcriptional reprogramming during cardiomyocyte hypertrophy.

O-GlcNAc 信号传导对于心肌细胞肥大过程中 NFAT 介导的转录重编程至关重要

期刊:American Journal of Physiology-Heart and Circulatory Physiology
影响因子:4.1
doi:10.1152/ajpheart.00775.2011
Facundo Heberty T, Brainard Robert E, Watson Lewis J, Ngoh Gladys A, Hamid Tariq, Prabhu Sumanth D, Jones Steven P
细胞生物学
发表日期：2012
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Cardiac overexpression of 8-oxoguanine DNA glycosylase 1 protects mitochondrial DNA and reduces cardiac fibrosis following transaortic constriction

心脏中8-氧鸟嘌呤DNA糖基化酶1的过度表达可保护线粒体DNA并减少主动脉缩窄术后的心肌纤维化

期刊:American Journal of Physiology-Heart and Circulatory Physiology
影响因子:4.1
doi:10.1152/ajpheart.00157.2011
Wang, Jianxun; Wang, Qianwen; Watson, Lewis J; Jones, Steven P; Epstein, Paul N
线粒体
发表日期：2011
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Non-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transition

非经典糖基转移酶调节缺氧后心肌细胞死亡和线粒体通透性转换

期刊:Journal of Molecular and Cellular Cardiology
影响因子:4.7
doi:10.1016/j.yjmcc.2008.04.009
Ngoh, Gladys A; Watson, Lewis J; Facundo, Heberty T; Dillmann, Wolfgang; Jones, Steven P
细胞生物学
线粒体
发表日期：2008
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Cardiac phosphatase-deficient 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase increases glycolysis, hypertrophy, and myocyte resistance to hypoxia

心脏磷酸酶缺乏症中6-磷酸果糖-2-激酶/果糖-2,6-二磷酸酶可增加糖酵解、肥大和心肌细胞对缺氧的抵抗力。

期刊:American Journal of Physiology-Heart and Circulatory Physiology
影响因子:4.1
doi:10.1152/ajpheart.91501.2007
Wang, Qianwen; Donthi, Rajakumar V; Wang, Jianxun; Lange, Alex J; Watson, Lewis J; Jones, Steven P; Epstein, Paul N
信号转导
细胞生物学
发表日期：2008
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