Wei Tzuping相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

Spinal disk injury induces germinal center formation, pronociceptive antibody production, and chronic nociceptive sensitization in a male mouse back pain model

脊髓椎间盘损伤可诱导雄性小鼠背痛模型中生发中心形成、促伤害性抗体产生和慢性伤害性感受敏化。

影响因子:5.5

doi:10.1097/j.pain.0000000000003706

Wei, Tzuping; Guo, Tian-Zhi; Shi, Xiaoyou; Li, Wen-Wu; Clark, J David; Kingery, Wade S

发表日期：2025

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Autoantibodies cause nociceptive sensitization in a mouse model of degenerative osteoarthritis.

自身抗体可导致退行性骨关节炎小鼠模型出现伤害性感受敏化

影响因子:5.5

doi:10.1097/j.pain.0000000000003500

Guo Tian-Zhi, Shi Xiaoyou, Li Xuanying, Li Wen-Wu, Wei Tzuping, Sahbaie Peyman, McAllister Tiffany N, Angst Martin S, Clark J David, Kingery Wade S

免疫/内分泌

发表日期：2024

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Pronociceptive autoantibodies in the spinal cord mediate nociceptive sensitization, loss of function, and spontaneous pain in the lumbar disk puncture model of chronic back pain

脊髓中的促伤害感受自身抗体介导腰椎间盘穿刺慢性背痛模型中的伤害感受敏化、功能丧失和自发性疼痛。

影响因子:5.5

doi:10.1097/j.pain.0000000000002725

Guo, Tian-Zhi; Shi, Xiaoyou; Li, Wen-Wu; Wei, Tzuping; Sahbaie, Peyman; Clark, J David; Kingery, Wade S

免疫/内分泌

发表日期：2023

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Mice lacking substance P have normal bone modeling but diminished bone formation, increased resorption, and accelerated osteopenia with aging

缺乏P物质的小鼠骨骼重塑功能正常，但骨形成减少，骨吸收增加，并且随着年龄增长，骨质疏松症加速发生。

doi:10.1016/j.bone.2020.115806

Wang, Liping; Hou, Saiyun; Sabsovich, Ilya; Guo, Tian-Zhi; Wei, Tzuping; Kingery, Wade S

发表日期：2021

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Passive transfer autoimmunity in a mouse model of complex regional pain syndrome

小鼠复杂区域疼痛综合征模型中的被动转移自身免疫

影响因子:5.5

doi:10.1097/j.pain.0000000000001046

Guo, Tian-Zhi; Shi, Xiaoyou; Li, Wen-Wu; Wei, Tzuping; Clark, John David; Kingery, Wade S

免疫/内分泌

发表日期：2017

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Acute versus chronic phase mechanisms in a rat model of CRPS

CRPS大鼠模型中急性期与慢性期机制的研究

期刊:Journal of Neuroinflammation

影响因子:10.1

doi:10.1186/s12974-015-0472-8

Wei, Tzuping; Guo, Tian-Zhi; Li, Wen-Wu; Kingery, Wade S; Clark, John David

发表日期：2016

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Bisphosphonates Inhibit Pain, Bone Loss, and Inflammation in a Rat Tibia Fracture Model of Complex Regional Pain Syndrome

双膦酸盐可抑制大鼠胫骨骨折复杂区域疼痛综合征模型中的疼痛、骨质流失和炎症

期刊:Anesthesia and Analgesia

影响因子:3.8

doi:10.1213/ANE.0000000000001518

Wang, Liping; Guo, Tian-Zhi; Hou, Saiyun; Wei, Tzuping; Li, Wen-Wu; Shi, Xiaoyou; Clark, J David; Kingery, Wade S

Immunology/Inflammation

发表日期：2016

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Facilitated spinal neuropeptide signaling and upregulated inflammatory mediator expression contribute to postfracture nociceptive sensitization

脊髓神经肽信号传导增强和炎症介质表达上调导致骨折后伤害性感受敏化。

影响因子:5.5

doi:10.1097/j.pain.0000000000000204

Shi, Xiaoyou; Guo, Tian-Zhi; Wei, Tzuping; Li, Wen-Wu; Clark, David J; Kingery, Wade S

发表日期：2015

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Immobilization contributes to exaggerated neuropeptide signaling, inflammatory changes, and nociceptive sensitization after fracture in rats

骨折后，大鼠固定会导致神经肽信号过度激活、炎症反应增强和伤害感受敏化。

期刊:Journal of Pain

doi:10.1016/j.jpain.2014.07.004

Guo, Tian-Zhi; Wei, Tzuping; Li, Wen-Wu; Li, Xiang-Qi; Clark, J David; Kingery, Wade S

发表日期：2014

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Neuropeptide deficient mice have attenuated nociceptive, vascular, and inflammatory changes in a tibia fracture model of complex regional pain syndrome.

神经肽缺乏的小鼠在胫骨骨折引起的复杂区域疼痛综合征模型中，伤害性感受、血管和炎症变化均有所减轻

期刊:Molecular Pain

影响因子:2.8

doi:10.1186/1744-8069-8-85

Guo Tian-Zhi, Wei Tzuping, Shi Xiaoyou, Li Wen-Wu, Hou Saiyun, Wang Liping, Tsujikawa Kazutake, Rice Kenner C, Cheng Kejun, Clark David J, Kingery Wade S

发表日期：2012

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