日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Short-Term DMOG treatment rejuvenates senescent mesenchymal stem cells by enhancing mitochondrial function and mitophagy through the HIF-1α/BNIP3 pathway.

短期 DMOG 治疗通过 HIF-1α/BNIP3 通路增强线粒体功能和线粒体自噬,从而使衰老间充质干细胞恢复活力

Wen Jiaxin, Yi Lingxian, Chen Lei, Xu Jinhan, Zhang Yanmin, Cheng Qiheng, Ping Hangyu, Wang Huanyu, Shuang Feng, Chai Wei, Weng Tujun

Overexpression of interleukin-10 in engineered macrophages protects endothelial cells against LPS-induced injury in vitro

在体外实验中,基因工程改造的巨噬细胞中白细胞介素-10的过表达可保护内皮细胞免受LPS诱导的损伤。

Yi, Lingxian; Weng, Tujun; Nie, Penghui; Zhu, Lin; Gao, Mingming; Jia, Hongxing; Yang, Shaohua; Li, Xiubin; Zhang, Luo; Xu, Ye; Ma, Penglin; Hu, Mei

Intraperitoneal injection of Desferal® alleviated the age-related bone loss and senescence of bone marrow stromal cells in rats

腹腔注射去铁胺®可减轻大鼠与年龄相关的骨质流失和骨髓基质细胞衰老。

Yi, Lingxian; Ju, Yue; He, Ying; Yin, Xiushan; Xu, Ye; Weng, Tujun

Blockade of Fgfr1 with PD166866 Protects Cartilage from the Catabolic Effects Induced by Interleukin-1β: A Genome-Wide Expression Profiles Analysis

利用PD166866阻断Fgfr1可保护软骨免受白细胞介素-1β诱导的分解代谢作用:全基因组表达谱分析

Yi, Lingxian; Lan, Guihua; Ju, Yue; Yin, Xiushan; Zhang, Peipei; Xu, Ye; Weng, Tujun

Synergistic Effects of FGF-18 and TGF-β3 on the Chondrogenesis of Human Adipose-Derived Mesenchymal Stem Cells in the Pellet Culture

FGF-18 和 TGF-β3 对人脂肪来源间充质干细胞在球状培养中软骨形成具有协同作用

Huang, Liangjie; Yi, Lingxian; Zhang, Chunli; He, Ying; Zhou, Liangliang; Liu, Yan; Qian, Long; Hou, Shuxun; Weng, Tujun

Fibroblast Growth Factor Receptor 3 Deficiency Does Not Impair the Osteoanabolic Action of Parathyroid Hormone on Mice

成纤维细胞生长因子受体3缺乏症并不损害甲状旁腺激素对小鼠的骨合成代谢作用

Xie, Yangli; Yi, Lingxian; Weng, Tujun; Huang, Junlan; Luo, Fengtao; Jiang, Wanling; Xian, Cory J; Du, Xiaolan; Chen, Lin

Inactivation of Vhl in osteochondral progenitor cells causes high bone mass phenotype and protects against age-related bone loss in adult mice

骨软骨祖细胞中Vhl基因的失活会导致高骨量表型,并能保护成年小鼠免受与年龄相关的骨质流失。

Weng, Tujun; Xie, Yangli; Huang, Junlan; Luo, Fengtao; Yi, Lingxian; He, Qifen; Chen, Di; Chen, Lin

A Ser252Trp mutation in fibroblast growth factor receptor 2 (FGFR2) mimicking human Apert syndrome reveals an essential role for FGF signaling in the regulation of endochondral bone formation

成纤维细胞生长因子受体2 (FGFR2) 中的Ser252Trp突变模拟人类Apert综合征,揭示了FGF信号在软骨内成骨调控中的关键作用

Chen, Peng; Zhang, Li; Weng, Tujun; Zhang, Shichang; Sun, Shijin; Chang, Mingtao; Li, Yang; Zhang, Bo; Zhang, Lianyang

Dynamic morphological changes in the skulls of mice mimicking human Apert syndrome resulting from gain-of-function mutation of FGFR2 (P253R)

小鼠颅骨的动态形态变化模拟了由FGFR2(P253R)功能获得性突变引起的人类Apert综合征

Du, Xiaolan; Weng, Tujun; Sun, Qidi; Su, Nan; Chen, Zhi; Qi, Huabing; Jin, Ming; Yin, Liangjun; He, Qifen; Chen, Lin

Smad4 is required for maintaining normal murine postnatal bone homeostasis

Smad4是维持小鼠出生后正常骨骼稳态所必需的。

Tan, Xiaohong; Weng, Tujun; Zhang, Jishuai; Wang, Jian; Li, Wenlong; Wan, Haifeng; Lan, Yu; Cheng, Xuan; Hou, Ning; Liu, Haihong; Ding, Jun; Lin, Fuyu; Yang, Ruifu; Gao, Xiang; Chen, Di; Yang, Xiao