Wilkinson Kevin A相关文献与解析，生知库

AntÃ³n-PlÃ¡garo Carlos, Chen Kai-En, Guo Qian, Liu Meihan, Evans Ashley J, Lewis Philip A, Heesom Kate J, Wilkinson Kevin A, Collins Brett M, Cullen Peter J

其它

发表日期：2025

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Erratum: Sustained postsynaptic kainate receptor activation downregulates AMPA receptor surface expression and induces hippocampal LTD

勘误：持续的突触后红藻氨酸受体激活会下调AMPA受体表面表达并诱导海马LTD。

期刊:iScience

影响因子:4.1

doi:10.1016/j.isci.2025.112152

Nair, Jithin D; Braksator, Ellen; Yucel, Busra P; Fletcher-Jones, Alexandra; Seager, Richard; Mellor, Jack R; Bashir, Zafar I; Wilkinson, Kevin A; Henley, Jeremy M

SGIP1 binding to the α-helical H9 domain of cannabinoid receptor 1 promotes axonal surface expression

SGIP1与大麻素受体1的α螺旋H9结构域结合，促进其轴突表面表达。

期刊:Journal of Cell Science

影响因子:3.6

doi:10.1242/jcs.261551

Fletcher-Jones, Alexandra; Spackman, Ellen; Craig, Tim J; Nakamura, Yasuko; Wilkinson, Kevin A; Henley, Jeremy M

A single coiled-coil domain mutation in hIKCa channel subunits disrupts preferential formation of heteromeric hSK1:hIKCa channels

hIKCa通道亚基中单个卷曲螺旋结构域的突变会破坏异源hSK1:hIKCa通道的优先形成。

期刊:European Journal of Neuroscience

影响因子:2.4

doi:10.1111/ejn.16189

Charlick, James N; Bozadzhieva, Daniella; Butler, Andrew S; Wilkinson, Kevin A; Marrion, Neil V

发表日期：2024

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SUMOylation of synaptic and synapse-associated proteins: An update

突触及突触相关蛋白的SUMO化：最新进展

期刊:Journal of Neurochemistry

影响因子:4

doi:10.1111/jnc.15103

Henley, Jeremy M; Seager, Richard; Nakamura, Yasuko; Talandyte, Karolina; Nair, Jithin; Wilkinson, Kevin A

Mechanisms and roles of mitochondrial localisation and dynamics in neuronal function

线粒体定位和动态变化在神经元功能中的机制和作用

期刊:Neuronal Signaling

影响因子:

doi:10.1042/NS20200008

Seager, Richard; Lee, Laura; Henley, Jeremy M; Wilkinson, Kevin A

Exciting Times: New Advances Towards Understanding the Regulation and Roles of Kainate Receptors

激动人心的时刻：理解红藻氨酸受体调控及其作用的新进展

期刊:Neurochemical Research

影响因子:3.8

doi:10.1007/s11064-017-2450-2

Evans, Ashley J; Gurung, Sonam; Henley, Jeremy M; Nakamura, Yasuko; Wilkinson, Kevin A

信号转导

发表日期：2019

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Commentary: Analysis of SUMO1-conjugation at synapses

评论：突触处SUMO1结合的分析

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2017.00345

Wilkinson, Kevin A; Martin, Stéphane; Tyagarajan, Shiva K; Arancio, Ottavio; Craig, Tim J; Guo, Chun; Fraser, Paul E; Goldstein, Steven A N; Henley, Jeremy M

Neuronal SUMOylation: mechanisms, physiology, and roles in neuronal dysfunction

神经元SUMO化：机制、生理学及其在神经元功能障碍中的作用

期刊:Physiological Reviews

影响因子:28.7

doi:10.1152/physrev.00008.2014

Henley, Jeremy M; Craig, Tim J; Wilkinson, Kevin A

AMPA receptor trafficking and the mechanisms underlying synaptic plasticity and cognitive aging

AMPA受体转运及其在突触可塑性和认知老化中的作用机制

期刊:Dialogues in Clinical Neuroscience

影响因子:8.9

doi:10.31887/DCNS.2013.15.1/jhenley

Henley, Jeremy M; Wilkinson, Kevin A

Mapping of endosomal proximity proteomes reveals Retromer as a hub for RAB GTPase regulation.

内体邻近蛋白质组的绘制揭示了 Retromer 是 RAB GTPase 调控的枢纽

Erratum: Sustained postsynaptic kainate receptor activation downregulates AMPA receptor surface expression and induces hippocampal LTD

勘误：持续的突触后红藻氨酸受体激活会下调AMPA受体表面表达并诱导海马LTD。

SGIP1 binding to the α-helical H9 domain of cannabinoid receptor 1 promotes axonal surface expression

SGIP1与大麻素受体1的α螺旋H9结构域结合，促进其轴突表面表达。

A single coiled-coil domain mutation in hIKCa channel subunits disrupts preferential formation of heteromeric hSK1:hIKCa channels

hIKCa通道亚基中单个卷曲螺旋结构域的突变会破坏异源hSK1:hIKCa通道的优先形成。

SUMOylation of synaptic and synapse-associated proteins: An update

突触及突触相关蛋白的SUMO化：最新进展

Mechanisms and roles of mitochondrial localisation and dynamics in neuronal function

线粒体定位和动态变化在神经元功能中的机制和作用

Exciting Times: New Advances Towards Understanding the Regulation and Roles of Kainate Receptors

激动人心的时刻：理解红藻氨酸受体调控及其作用的新进展

Commentary: Analysis of SUMO1-conjugation at synapses

评论：突触处SUMO1结合的分析

Neuronal SUMOylation: mechanisms, physiology, and roles in neuronal dysfunction

神经元SUMO化：机制、生理学及其在神经元功能障碍中的作用

AMPA receptor trafficking and the mechanisms underlying synaptic plasticity and cognitive aging

AMPA受体转运及其在突触可塑性和认知老化中的作用机制