Winoto Astar相关文献与解析，生知库 | 链接生命科学的每一步

B cell lymphoma 2 (Bcl-2) residues essential for Bcl-2's apoptosis-inducing interaction with Nur77/Nor-1 orphan steroid receptors

细胞淋巴瘤 2 (Bcl-2) 残基对于 Bcl-2 与 Nur77/Nor-1 孤儿类固醇受体的凋亡诱导相互作用至关重要

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.RA117.001101

Banta, Karl L; Wang, Xinyue; Das, Phani; Winoto, Astar

Premalignant PTEN-deficient thymocytes activate microRNAs miR-146a and miR-146b as a cellular defense against malignant transformation

癌前PTEN缺陷型胸腺细胞激活microRNA miR-146a和miR-146b，作为细胞抵御恶性转化的防御机制。

期刊:Blood

影响因子:23.1

doi:10.1182/blood-2013-11-539411

Burger, Megan L; Xue, Ling; Sun, Yuefang; Kang, Chulho; Winoto, Astar

Fas-associated death domain (FADD) and the E3 ubiquitin-protein ligase TRIM21 interact to negatively regulate virus-induced interferon production

Fas相关死亡结构域（FADD）与E3泛素连接酶TRIM21相互作用，负调控病毒诱导的干扰素产生。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M110.172288

Young, Jennifer A; Sermwittayawong, Decha; Kim, Hee-Jung; Nandu, Suruchi; An, Namsil; Erdjument-Bromage, Hediye; Tempst, Paul; Coscoy, Laurent; Winoto, Astar

Comparative study of hematopoietic differentiation between human embryonic stem cell lines

人类胚胎干细胞系造血分化的比较研究

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0019854

Melichar, Heather; Li, Ou; Ross, Jenny; Haber, Hilary; Cado, Dragana; Nolla, Hector; Robey, Ellen A; Winoto, Astar

Coupling of the cell cycle and apoptotic machineries in developing T cells.

发育中 T 细胞的细胞周期和凋亡机制的耦合

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M109.035535

Xue Ling, Sun Yuefang, Chiang Leslie, He Bo, Kang Chulho, Nolla Hector, Winoto Astar

During negative selection, Nur77 family proteins translocate to mitochondria where they associate with Bcl-2 and expose its proapoptotic BH3 domain

在负选择过程中，Nur77家族蛋白易位至线粒体，在那里它们与Bcl-2结合并暴露其促凋亡的BH3结构域。

期刊:Journal of Experimental Medicine

影响因子:10.6

doi:10.1084/jem.20080101

Thompson, Jennifer; Winoto, Astar

Non-redundant function of the MEK5-ERK5 pathway in thymocyte apoptosis

MEK5-ERK5通路在胸腺细胞凋亡中的非冗余功能

期刊:EMBO Journal

影响因子:8.3

doi:10.1038/emboj.2008.114

Sohn, Sue J; Lewis, Gavin M; Winoto, Astar

The role of the PI3K-AKT kinase pathway in T-cell development beyond the beta checkpoint

PI3K-AKT激酶通路在T细胞发育中超越β检查点的作用

期刊:European Journal of Immunology

影响因子:3.7

doi:10.1002/eji.200838614

Xue, Ling; Chiang, Leslie; Kang, Chulho; Winoto, Astar

Transcriptional regulation of tissue-specific genes by the ERK5 mitogen-activated protein kinase

ERK5丝裂原活化蛋白激酶对组织特异性基因的转录调控

期刊:Molecular and Cellular Biology

影响因子:2.7

doi:10.1128/MCB.25.19.8553-8566.2005

Sohn, Sue J; Li, Dongling; Lee, Linda K; Winoto, Astar

Necroptotic-susceptible dendritic cells exhibit enhanced antitumor activities in mice

易发生坏死性凋亡的树突状细胞在小鼠体内表现出增强的抗肿瘤活性。

B cell lymphoma 2 (Bcl-2) residues essential for Bcl-2's apoptosis-inducing interaction with Nur77/Nor-1 orphan steroid receptors

细胞淋巴瘤 2 (Bcl-2) 残基对于 Bcl-2 与 Nur77/Nor-1 孤儿类固醇受体的凋亡诱导相互作用至关重要

Premalignant PTEN-deficient thymocytes activate microRNAs miR-146a and miR-146b as a cellular defense against malignant transformation

癌前PTEN缺陷型胸腺细胞激活microRNA miR-146a和miR-146b，作为细胞抵御恶性转化的防御机制。

Fas-associated death domain (FADD) and the E3 ubiquitin-protein ligase TRIM21 interact to negatively regulate virus-induced interferon production

Fas相关死亡结构域（FADD）与E3泛素连接酶TRIM21相互作用，负调控病毒诱导的干扰素产生。

Comparative study of hematopoietic differentiation between human embryonic stem cell lines

人类胚胎干细胞系造血分化的比较研究

Coupling of the cell cycle and apoptotic machineries in developing T cells.

发育中 T 细胞的细胞周期和凋亡机制的耦合

During negative selection, Nur77 family proteins translocate to mitochondria where they associate with Bcl-2 and expose its proapoptotic BH3 domain

在负选择过程中，Nur77家族蛋白易位至线粒体，在那里它们与Bcl-2结合并暴露其促凋亡的BH3结构域。

Non-redundant function of the MEK5-ERK5 pathway in thymocyte apoptosis

MEK5-ERK5通路在胸腺细胞凋亡中的非冗余功能

The role of the PI3K-AKT kinase pathway in T-cell development beyond the beta checkpoint

PI3K-AKT激酶通路在T细胞发育中超越β检查点的作用

Transcriptional regulation of tissue-specific genes by the ERK5 mitogen-activated protein kinase

ERK5丝裂原活化蛋白激酶对组织特异性基因的转录调控