日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Disparate leukemia mutations converge on nuclear phase-separated condensates.

不同的白血病突变最终都导致核内相分离凝聚体的形成。

Datar Gandhar K, Khabusheva Elmira, Anand Archish, Beale Joshua, Sadek Marwa, Chen Chun-Wei, Potolitsyna Evdokiia, Alcantara-Contessoto Nayara, Liu Guangyuan, De La Fuente Josephine, Dollinger Christina, Guzman Anna, Martell Alejandra, Wohlan Katharina, Maiti Abhishek, Short Nicholas J, Yi S Stephen, Andresen Vibeke, Gjertsen Bjørn Tore, Falini Brunangelo, Rau Rachel E, Brunetti Lorenzo, Sahni Nidhi, Goodell Margaret A, Riback Joshua A

Nuclear Phase Separation Drives NPM1-mutant Acute Myeloid Leukemia.

核相分离驱动NPM1突变型急性髓系白血病

Datar Gandhar K, Khabusheva Elmira, Anand Archish, Beale Joshua, Sadek Marwa, Chen Chun-Wei, Potolitsyna Evdokiia, Alcantara-Contessoto Nayara, Liu Guangyuan, De La Fuente Josephine, Dollinger Christina, Guzman Anna, Martell Alejandra, Wohlan Katharina, Maiti Abhishek, Short Nicholas J, Yi S Stephen, Andresen Vibeke, Gjertsen Bjørn Tore, Falini Brunangelo, Rau Rachel E, Brunetti Lorenzo, Sahni Nidhi, Goodell Margaret A, Riback Joshua A

SOD1 is a synthetic-lethal target in PPM1D-mutant leukemia cells

SOD1是PPM1D突变型白血病细胞中的合成致死靶点。

Linda Zhang ,Joanne I Hsu ,Etienne D Braekeleer ,Chun-Wei Chen ,Tajhal D Patel ,Alejandra G Martell ,Anna G Guzman ,Katharina Wohlan ,Sarah M Waldvogel ,Hidetaka Uryu ,Ayala Tovy ,Elsa Callen ,Rebecca L Murdaugh ,Rosemary Richard ,Sandra Jansen ,Lisenka Vissers ,Bert B A de Vries ,Andre Nussenzweig ,Shixia Huang ,Cristian Coarfa ,Jamie Anastas ,Koichi Takahashi ,George Vassiliou ,Margaret A Goodell

SOD1 is a synthetic lethal target in PPM1D-mutant leukemia cells

SOD1 是 PPM1D 突变白血病细胞中的合成致死靶点

Linda Zhang, Joanne I Hsu, Etienne D Braekeleer, Chun-Wei Chen, Tajhal D Patel, Alejandra G Martell, Anna G Guzman, Katharina Wohlan, Sarah M Waldvogel, Hidetaka Urya, Ayala Tovy, Elsa Callen, Rebecca Murdaugh, Rosemary Richard, Sandra Jansen, Lisenka Vissers, Bert B A de Vries, Andre Nussenzweig, S

TET2 modulates spatial relocalization of heterochromatin in aged hematopoietic stem and progenitor cells

TET2调节衰老造血干细胞和祖细胞中异染色质的空间重定位

Tingting Hong #,Jia Li #,Lei Guo,Maryn Cavalier,Tianlu Wang,Yaling Dou,Aaron DeLaFuente,Shaohai Fang,Anna Guzman,Katherina Wohlan,Chiraag Kapadia,Carina Rosas,Yaling Yang,C Cameron Yin,Shaoying Li,M James You,Xiaodong Cheng,Margaret A Goodell,Yubin Zhou,Yun Huang

SWI/SNF Blockade Disrupts PU.1-Directed Enhancer Programs in Normal Hematopoietic Cells and Acute Myeloid Leukemia

SWI/SNF 阻断会破坏正常造血细胞和急性髓系白血病中的 PU.1 定向增强子程序

Courtney Chambers, Katerina Cermakova, Yuen San Chan, Kristen Kurtz, Katharina Wohlan, Andrew Henry Lewis, Christiana Wang, Anh Pham, Milan Dejmek, Michal Sala, Mario Loeza Cabrera, Rogelio Aguilar, Radim Nencka, H Daniel Lacorazza, Rachel E Rau, H Courtney Hodges

Chemotherapy-Free Targeted Anti-BCR-ABL+ Acute Lymphoblastic Leukemia Therapy May Benefit the Heart

无需化疗的抗 BCR-ABL+ 急性淋巴细胞白血病靶向疗法可能有益于心脏

Hanna Kirchhoff, Melanie Ricke-Hoch, Katharina Wohlan, Stefan Pietzsch, Ümran Karsli, Sergej Erschow, Robert Zweigerdt, Arnold Ganser, Matthias Eder, Michaela Scherr, Denise Hilfiker-Kleiner

Anthracycline-free tumor elimination in mice leads to functional and molecular cardiac recovery from cancer-induced alterations in contrast to long-lasting doxorubicin treatment effects

与长期阿霉素治疗效果相比,小鼠无蒽环类药物肿瘤消除可使癌症引起的心脏功能和分子恢复

Stefan Pietzsch, Katharina Wohlan, James T Thackeray, Maren Heimerl, Sven Schuchardt, Michaela Scherr, Melanie Ricke-Hoch #, Denise Hilfiker-Kleiner #

Optimized induction of mitochondrial apoptosis for chemotherapy-free treatment of BCR-ABL+acute lymphoblastic leukemia

优化线粒体凋亡诱导以用于 BCR-ABL+急性淋巴细胞白血病的无化疗治疗

Scherr, Michaela; Kirchhoff, Hanna; Battmer, Karin; Wohlan, Katharina; Lee, Chun-Wei; Ricke-Hoch, Melanie; Erschow, Sergej; Law, Edward; Kloos, Arnold; Heuser, Michael; Ganser, Arnold; Hilfiker-Kleiner, Denise; Heidenreich, Olaf; Eder, Matthias

Stable depletion of RUNX1-ETO in Kasumi-1 cells induces expression and enhanced proteolytic activity of Cathepsin G and Neutrophil Elastase

Kasumi-1 细胞中 RUNX1-ETO 的稳定消耗会诱导组织蛋白酶 G 和中性粒细胞弹性蛋白酶的表达和蛋白水解活性增强

Caroline Schoenherr, Katharina Wohlan, Iris Dallmann, Andreas Pich, Jan Hegermann, Arnold Ganser, Denise Hilfiker-Kleiner, Olaf Heidenreich, Michaela Scherr, Matthias Eder