日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Cell-type resolved transcriptomic approaches for dissecting selective vulnerability in neurodegeneration

利用细胞类型分辨的转录组学方法解析神经退行性疾病中的选择性脆弱性

Wood, Caleb A; Tran, Nicholas M

Chemogenetic neuronal silencing decouples c-Jun activation from cell death in the temporal cortex

化学遗传学神经元沉默使颞叶皮层中c-Jun激活与细胞死亡脱钩

Wood, Caleb A; Somasundaram, Preethi; Dundee, Jacob M; Rudy, Melissa A; Watkins, Trent A; Jankowsky, Joanna L

TMEM106B coding variant is protective and deletion detrimental in a mouse model of tauopathy

在tau蛋白病小鼠模型中,TMEM106B编码变异具有保护作用,而缺失则有害。

Edwards, George A; Wood, Caleb A; Nguyen, Quynh; Kim, Peter J; Gomez-Gutierrez, Ruben; Park, Kyung-Won; Zurhellen, Cody; Al-Ramahi, Ismael; Jankowsky, Joanna L

Combining multiomics and drug perturbation profiles to identify muscle-specific treatments for spinal muscular atrophy

结合多组学和药物扰动谱来确定脊髓性肌萎缩症的肌肉特异性治疗方法

Katharina E Meijboom, Viola Volpato, Jimena Monzón-Sandoval, Joseph M Hoolachan, Suzan M Hammond, Frank Abendroth, Olivier G de Jong, Gareth Hazell, Nina Ahlskog, Matthew Ja Wood, Caleb Webber, Melissa Bowerman

The nuts and bolts of omics for the clinical allergist

临床过敏科医生的组学基础知识

Virkud, Yamini V; Kelly, Rachel S; Wood, Caleb; Lasky-Su, Jessica A

VAChT overexpression increases acetylcholine at the synaptic cleft and accelerates aging of neuromuscular junctions.

VAChT 过度表达会增加突触间隙中的乙酰胆碱,加速神经肌肉接头的衰老

Sugita Satoshi, Fleming Leland L, Wood Caleb, Vaughan Sydney K, Gomes Matheus P S M, Camargo Wallace, Naves Ligia A, Prado Vania F, Prado Marco A M, Guatimosim Cristina, Valdez Gregorio

Validation of operant social motivation paradigms using BTBR T+tf/J and C57BL/6J inbred mouse strains

利用BTBR T+tf/J和C57BL/6J近交系小鼠验证操作性社会动机范式

Martin, Loren; Sample, Hannah; Gregg, Michael; Wood, Caleb