日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Cardiomyocyte Foxp1-Specific Deletion Promotes Post-injury Heart Regeneration via Targeting Usp20-HIF1ɑ-Hand1 Signaling Pathway

心肌细胞Foxp1特异性缺失通过靶向Usp20-HIF1α-Hand1信号通路促进损伤后心脏再生

Wang, Yanfang; Wang, Xiaoyu; Fang, Ji; Chen, Xiaoli; Xu, Teng; Zhuang, Tao; Peng, Sheng; Bao, Wenzhen; Wu, Wenrun; Lu, Yushi; Wang, Haikun; Tomlinson, Brian; Chan, Paul; Zhuang, Shougang; Zhang, Qi; Zhang, Lin; Liu, Zhongmin; Pi, Jingjiang; Zhang, Yuzhen; Liu, Jie

SLC1A5-dependent glutamine uptake in hepatocytes promotes liver regeneration

肝细胞中SLC1A5依赖的谷氨酰胺摄取促进肝脏再生

Duan, Yunhao; Peng, Sheng; Yue, Jinnan; Ding, Fan; Chen, Xiaoli; Pan, Wenqi; Gao, Xiaotong; Pang, Peilin; Zhang, Yuting; Wu, Wenrun; Schneider, Kai Markus; Cubero, Francisco Javier; Trautwein, Christian; Zhang, Lin; Zhang, Yuzhen; Liu, Jie; Zhao, Gang

Foxp1 Is Required for Renal Intercalated Cell Differentiation and Acid-Base Regulation.

Foxp1 是肾脏闰细胞分化和酸碱调节所必需的

Wu Shi-Ting, Feng Yu, Song Renhua, Qi Yanmiao, Li Lin, Lu Dongbo, Wang Yixuan, Wu Wenrun, Morgan Angela, Wang Xiaohong, Xia Yin, Liu Renjing, Alexander Stephen I, Wong Justin, Zhang Yuzhen, Zheng Xiangjian

Corrigendum to "Endothelial Gata6 deletion reduces monocyte recruitment and proinflammatory macrophage formation and attenuates atherosclerosis through Cmpk2-Nlrp3 pathways" [Redox Biol. 64 (2023) 102775]

对“内皮细胞 Gata6 缺失通过 Cmpk2-Nlrp3 通路减少单核细胞募集和促炎巨噬细胞形成并减弱动脉粥样硬化”的更正 [Redox Biol. 64 (2023) 102775]

Wu, Wenrun; Bao, Wenzhen; Chen, Xiaoli; Lu, Yushi; Fang, Ji; Liu, Jiwen; Peng, Sheng; Pi, Jingjiang; Tomlinson, Brian; Chan, Paul; Zhang, Qi; Zhang, Lin; Liu, Zhongmin; Liu, Jie; Zhang, Yuzhen; Zhuang, Tao

Endothelial Gata6 deletion reduces monocyte recruitment and proinflammatory macrophage formation and attenuates atherosclerosis through Cmpk2-Nlrp3 pathways

内皮细胞Gata6缺失通过Cmpk2-Nlrp3通路减少单核细胞募集和促炎巨噬细胞形成,并减轻动脉粥样硬化。

Wu, Wenrun; Bao, Wenzhen; Chen, Xiaoli; Lu, Yushi; Fang, Ji; Liu, Jiwen; Peng, Sheng; Pi, Jingjiang; Tomlinson, Brian; Chan, Paul; Zhang, Qi; Zhang, Lin; Liu, Zhongmin; Liu, Jie; Zhang, Yuzhen; Zhuang, Tao