Xinxing Wan相关文献与解析，生知库 | 链接生命科学的每一步

NAT10 promotes liver lipogenesis in mouse through N4-acetylcytidine modification of Srebf1 and Scap mRNA

NAT10通过N4-乙酰胞苷修饰Srebf1和Scap mRNA促进小鼠肝脏脂肪生成

期刊:Lipids in Health and Disease

影响因子:3.9

doi:10.1186/s12944-024-02360-1

Zhouqi Wang, Xinxing Wan, Md Asaduzzaman Khan, Lin Peng, Xiaoying Sun, Xuan Yi, Ke Chen

表观遗传

发表日期：2024

文献求助收藏

hPER3 promotes adipogenesis via hHSP90AA1-mediated inhibition of Notch1 pathway

hPER3 通过 hHSP90AA1 介导的 Notch1 通路抑制促进脂肪生成

期刊:Cell Death & Disease

影响因子:8.1

doi:10.1038/s41419-021-03584-0

Xinxing Wan #, Liyong Zhu #, Liling Zhao, Lin Peng, Jing Xiong, Wenjun Yang, Jingjing Yuan, Fang Liang, Keke Zhang, Ke Chen

c-Jun Overexpression Accelerates Wound Healing in Diabetic Rats by Human Umbilical Cord-Derived Mesenchymal Stem Cells

c-Jun 过度表达加速人脐带间充质干细胞促进糖尿病大鼠伤口愈合

期刊:Stem Cells International

影响因子:3.8

doi:10.1155/2020/7430968

Chun Yue, Zi Guo, Yufang Luo, Jingjing Yuan, Xinxing Wan, Zhaohui Mo

Cbl Proto-Oncogene B (CBLB) c.197A>T Mutation Induces Mild Metabolic Dysfunction in Partial Type I Multiple Symmetric Lipomatosis (MSL)

Cbl 原癌基因 B (CBLB) c.197A>T 突变导致部分 I 型多发性对称性脂肪瘤 (MSL) 出现轻度代谢功能障碍

期刊:Diabetes Metabolic Syndrome and Obesity-Targets and Therapy

影响因子:2.8

doi:10.2147/DMSO.S273780

Ke Chen, Xinxing Wan, Liling Zhao, Shaoli Zhao, Lin Peng, Wenjun Yang, Jingjing Yuan, Liyong Zhu, Zhaohui Mo

Mesenchymal Stem Cells Coated by the Extracellular Matrix Promote Wound Healing in Diabetic Rats

细胞外基质包裹的间充质干细胞促进糖尿病大鼠伤口愈合

期刊:Stem Cells International

影响因子:3.8

doi:10.1155/2019/9564869

Linhao Wang, Fang Wang, Liling Zhao, Wenjun Yang, Xinxing Wan, Chun Yue, Zhaohui Mo

Oxidative Stress Alters the Morphological Responses of Myoblasts to Single-Site Membrane Photoporation

氧化应激改变成肌细胞对单一位点膜光穿孔的形态学反应

期刊:Cellular and Molecular Bioengineering

影响因子:5

doi:10.1007/s12195-017-0488-5

Duan, Xinxing; Wan, Jennifer M F; Mak, Arthur F T

miR-204-5p promotes the adipogenic differentiation of human adipose-derived mesenchymal stem cells by modulating DVL3 expression and suppressing Wnt/β-catenin signaling

miR-204-5p 通过调节 DVL3 表达和抑制 Wnt/β-catenin 信号传导促进人类脂肪间充质干细胞的脂肪形成分化

期刊:International Journal of Molecular Medicine

影响因子:5.7

doi:10.3892/ijmm.2015.2160

Honghui He, Ke Chen, Fang Wang, Liling Zhao, Xinxing Wan, Linghao Wang, Zhaohui Mo

miR-125a-3p and miR-483-5p promote adipogenesis via suppressing the RhoA/ROCK1/ERK1/2 pathway in multiple symmetric lipomatosis

miR-125a-3p 和 miR-483-5p 通过抑制多发性对称性脂肪瘤中的 RhoA/ROCK1/ERK1/2 通路促进脂肪生成

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/srep11909

Ke Chen, Honghui He, Yanhong Xie, Liling Zhao, Shaoli Zhao, Xinxing Wan, Wenjun Yang, Zhaohui Mo

The molecular impact of sonoporation: A transcriptomic analysis of gene regulation profile.

声穿孔的分子影响：基因调控谱的转录组分析

Shift work promotes adipogenesis via cortisol-dependent downregulation of EGR3-HDAC6 pathway

轮班工作通过皮质醇依赖性 EGR3-HDAC6 通路下调促进脂肪生成

NAT10 promotes liver lipogenesis in mouse through N4-acetylcytidine modification of Srebf1 and Scap mRNA

NAT10通过N4-乙酰胞苷修饰Srebf1和Scap mRNA促进小鼠肝脏脂肪生成

hPER3 promotes adipogenesis via hHSP90AA1-mediated inhibition of Notch1 pathway

hPER3 通过 hHSP90AA1 介导的 Notch1 通路抑制促进脂肪生成

c-Jun Overexpression Accelerates Wound Healing in Diabetic Rats by Human Umbilical Cord-Derived Mesenchymal Stem Cells

c-Jun 过度表达加速人脐带间充质干细胞促进糖尿病大鼠伤口愈合

Cbl Proto-Oncogene B (CBLB) c.197A>T Mutation Induces Mild Metabolic Dysfunction in Partial Type I Multiple Symmetric Lipomatosis (MSL)

Cbl 原癌基因 B (CBLB) c.197A>T 突变导致部分 I 型多发性对称性脂肪瘤 (MSL) 出现轻度代谢功能障碍

Mesenchymal Stem Cells Coated by the Extracellular Matrix Promote Wound Healing in Diabetic Rats

细胞外基质包裹的间充质干细胞促进糖尿病大鼠伤口愈合

Oxidative Stress Alters the Morphological Responses of Myoblasts to Single-Site Membrane Photoporation

氧化应激改变成肌细胞对单一位点膜光穿孔的形态学反应

miR-204-5p promotes the adipogenic differentiation of human adipose-derived mesenchymal stem cells by modulating DVL3 expression and suppressing Wnt/β-catenin signaling

miR-204-5p 通过调节 DVL3 表达和抑制 Wnt/β-catenin 信号传导促进人类脂肪间充质干细胞的脂肪形成分化

miR-125a-3p and miR-483-5p promote adipogenesis via suppressing the RhoA/ROCK1/ERK1/2 pathway in multiple symmetric lipomatosis

miR-125a-3p 和 miR-483-5p 通过抑制多发性对称性脂肪瘤中的 RhoA/ROCK1/ERK1/2 通路促进脂肪生成