日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

hUMSC-derived exosomes alleviate follicular interstitial cell autophagy by let-7a-5p/AMPK/mTOR axis in POI rats.

hUMSC 来源的外泌体通过 let-7a-5p/AMPK/mTOR 轴减轻 POI 大鼠的卵泡间质细胞自噬

Tang Yu, He Yu, Huo Xingyu, Chen Juntong, Qian Maojiao, Huang Haoyu, Meng Yixuan, Zhang Lianshuang, Xu Feibo, Zhang Yukun, Bao Hongchu, Xiong Yanlian

MSC-derived exosomes improve endometrial fibrosis via the lncRNA IGF2R/ miR-143-5p/AQP8 axis.

MSC衍生的外泌体通过lncRNA IGF2R/ miR-143-5p/AQP8轴改善子宫内膜纤维化

Huo Xingyu, Chen Juntong, Qian Maojiao, Xue Qian, Xu Pengzhan, Wang Yueming, Jiang Zhonglin, Luo Qianqian, Xiong Yanlian

Exosomes Derived From Human Mesenchymal Stem Cells Mitigate Follicular Interstitial Cell Ferroptosis via the miR-26a-5p/PTEN/GPX4 Axis in Rats with Chemotherapy-Induced Premature Ovarian Insufficiency.

源自人类间充质干细胞的外泌体通过 miR-26a-5p/PTEN/GPX4 轴减轻化疗诱导的卵巢早衰大鼠的卵泡间质细胞铁死亡

Chen Juntong, Huo Xingyu, Qian Maojiao, Xue Qian, He Yu, Xu Pengzhan, Wang Yueming, Tang Xiaoxuan, Luo Qianqian, Bao Hongchu, Xiong Yanlian

miR-9-5p alleviates the development of abdominal aortic aneurysm by regulating the differentiation of CD4(+)IL-10(+)T cells via targeting the crosstalk between Nrf2 and NF-κB signaling pathways.

miR-9-5p 通过靶向 Nrf2 和 NF-κB 信号通路之间的串扰来调节 CD4(+)IL-10(+)T 细胞的分化,从而减轻腹主动脉瘤的发展

Liu Hongfu, Zhang Jinyi, Li Lubin, Yu Benxiang, Zhang Chunlei, Niu Wenqiang, Cheng Yawen, Dong Hengyang, Zhang Yukun, Luo Xinlin, Xiong Yanlian, Wang Yueming

Corrigendum to "miR-9-5p alleviates the development of abdominal aortic aneurysm by regulating the differentiation of CD4(+)IL-10(+)T cells via targeting the crosstalk between Nrf2 and NF-κB signaling pathways" [Turkish Journal of Biology 49 (4) 2025 380-391]

对“miR-9-5p 通过靶向 Nrf2 和 NF-κB 信号通路之间的串扰来调节 CD4(+)IL-10(+)T 细胞的分化,从而缓解腹主动脉瘤的发展”的更正 [土耳其生物学杂志 49 (4) 2025 380-391]

Liu, Hongfu; Zhang, Jinyi; Li, Lubin; Yu, Benxiang; Zhang, Chunlei; Niu, Wenqiang; Cheng, Yawen; Dong, Hengyang; Zhang, Yukun; Luo, Xinlin; Xiong, Yanlian; Wang, Yueming

Correction: hPMSCs protects against D-galactose-induced oxidative damage of CD4(+) T cells through activating Akt-mediated Nrf2 antioxidant signaling

更正:hPMSCs通过激活Akt介导的Nrf2抗氧化信号通路来保护CD4(+) T细胞免受D-半乳糖诱导的氧化损伤。

Xiong, Yanlian; Wang, Yueming; Zhang, Jiashen; Zhao, Nannan; Zhang, Hengchao; Zhang, Aiping; Zhao, Dongmei; Yu, Zhenhai; Yin, Yancun; Song, Lele; Xiong, Yanlei; Luan, Xiying