日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Revisiting the role of metabolic reprogramming as a contributor to prostate cancer disease progression

重新审视代谢重编程在促进前列腺癌疾病进展中的作用

Ding, Hekang; Meng, Qingwei; Zhou, Jun; Chen, Li; Huang, Jiaoti; Zhao, Bing; Xu, Lingfan

Docosahexaenoic acid mitigates experimental autoimmune prostatitis by inhibiting Th17 cell differentiation via the PPARγ/NF-κB/IL-17A pathway.

二十二碳六烯酸通过 PPARγ/NF-κB/IL-17A 通路抑制 Th17 细胞分化,从而减轻实验性自身免疫性前列腺炎

Wu Weikang, Yue Shaoyu, Wang Xu, Li WeiYi, Xu Lingfan, Guan Yu, Liang Chao-Zhao

Corrigendum: Long non-coding RNA SNHG1 regulates the Wnt/β-catenin and PI3K/AKT/mTOR signaling pathways via EZH2 to affect the proliferation, apoptosis, and autophagy of prostate cancer cell

更正:长链非编码RNA SNHG1通过EZH2调控Wnt/β-catenin和PI3K/AKT/mTOR信号通路,从而影响前列腺癌细胞的增殖、凋亡和自噬。

Chen, Junyi; Wang, Fubo; Xu, Huan; Xu, Lingfan; Chen, Dong; Wang, Jialiang; Huang, Sihuai; Wen, Yiqun; Fang, Longmin

Targeting glutamine dependence with DRP-104 inhibits proliferation and tumor growth of castration-resistant prostate cancer

利用 DRP-104 靶向谷氨酰胺依赖性可抑制去势抵抗性前列腺癌的增殖和肿瘤生长。

Moon, David; Hauck, J Spencer; Jiang, Xue; Quang, Holly; Xu, Lingfan; Zhang, Fan; Gao, Xia; Wild, Robert; Everitt, Jeffrey I; Macias, Everardo; He, Yiping; Huang, Jiaoti

Author Correction: Ectopic JAK-STAT activation enables the transition to a stem-like and multilineage state conferring AR-targeted therapy resistance

作者更正:异位JAK-STAT激活可使细胞向干细胞样多谱系状态转变,从而赋予其对雄激素受体靶向治疗的耐药性。

Deng, Su; Wang, Choushi; Wang, Yunguan; Xu, Yaru; Li, Xiaoling; Johnson, Nickolas A; Mukherji, Atreyi; Lo, U-Ging; Xu, Lingfan; Gonzalez, Julisa; Metang, Lauren A; Ye, Jianfeng; Tirado, Carla Rodriguez; Rodarte, Kathia; Zhou, Yinglu; Xie, Zhiqun; Arana, Carlos; Annamalai, Valli; Liu, Xihui; Vander Griend, Donald J; Strand, Douglas; Hsieh, Jer-Tsong; Li, Bo; Raj, Ganesh; Wang, Tao; Mu, Ping

Reprogramming hormone-sensitive prostate cancer to a lethal neuroendocrine cancer lineage by mitochondrial pyruvate carrier (MPC).

通过线粒体丙酮酸载体(MPC)将激素敏感型前列腺癌重编程为致命的神经内分泌癌谱系

Xu Huan, Liu Zhixiao, Gao Dajun, Li Peizhang, Shen Yanting, Sun Yi, Xu Lingfan, Song Nan, Wang Yue, Zhan Ming, Gao Xu, Wang Zhong

N-cadherin inhibitor creates a microenvironment that protect TILs from immune checkpoints and Treg cells

N-钙黏蛋白抑制剂可创造一种微环境,保护肿瘤浸润淋巴细胞免受免疫检查点和调节性T细胞的攻击。

Sun, Yi; Jing, Jun; Xu, Huan; Xu, Lingfan; Hu, Hailiang; Tang, Cai; Liu, Shengzhuo; Wei, Qiang; Duan, Ruiqi; Guo, Ju; Yang, Lu

Long Non-Coding RNA SNHG1 Regulates the Wnt/β-Catenin and PI3K/AKT/mTOR Signaling Pathways via EZH2 to Affect the Proliferation, Apoptosis, and Autophagy of Prostate Cancer Cell

长链非编码RNA SNHG1通过EZH2调控Wnt/β-Catenin和PI3K/AKT/mTOR信号通路影响前列腺癌细胞增殖、凋亡和自噬

Junyi Chen, Fubo Wang, Huan Xu, Lingfan Xu, Dong Chen, Jialiang Wang, Sihuai Huang, Yiqun Wen, Longmin Fang

Correction to: N-Myc promotes therapeutic resistance development of neuroendocrine prostate cancer by differentially regulating miR-421/ ATM pathway

更正:N-Myc 通过差异性调控 miR-421/ATM 通路促进神经内分泌前列腺癌的治疗耐药性发展

Yin, Yu; Xu, Lingfan; Chang, Yan; Zeng, Tao; Chen, Xufeng; Wang, Aifen; Groth, Jeff; Foo, Wen-Chi; Liang, Chaozhao; Hu, Hailiang; Huang, Jiaoti

Targeting cellular heterogeneity with CXCR2 blockade for the treatment of therapy-resistant prostate cancer

利用CXCR2阻断靶向细胞异质性治疗耐药性前列腺癌

Li, Yanjing; He, Yiping; Butler, William; Xu, Lingfan; Chang, Yan; Lei, Kefeng; Zhang, Hong; Zhou, Yinglu; Gao, Allen C; Zhang, Qingfu; Taylor, Daniel G; Cheng, Donghui; Farber-Katz, Suzette; Karam, Rachid; Landrith, Tyler; Li, Bing; Wu, Sitao; Hsuan, Vickie; Yang, Qing; Hu, Hailiang; Chen, Xufeng; Flowers, Melissa; McCall, Shannon J; Lee, John K; Smith, Bryan A; Park, Jung Wook; Goldstein, Andrew S; Witte, Owen N; Wang, Qianben; Rettig, Matthew B; Armstrong, Andrew J; Cheng, Qing; Huang, Jiaoti